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PUBMED FOR HANDHELDS

Journal Abstract Search


238 related items for PubMed ID: 8077359

  • 1. Abnormality of aldosterone and cortisol late pathways in glucocorticoid-remediable aldosteronism.
    Fallo F, Kuhnle U, Boscaro M, Sonino N.
    J Clin Endocrinol Metab; 1994 Sep; 79(3):772-4. PubMed ID: 8077359
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  • 2. Altered 11 beta-hydroxylase activity in glucocorticoid-suppressible hyperaldosteronism.
    Jamieson A, Ingram MC, Inglis GC, Davies E, Fraser R, Connell JM.
    J Clin Endocrinol Metab; 1996 Jun; 81(6):2298-302. PubMed ID: 8964867
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  • 3. 11 beta-Hydroxylase activity in glucocorticoid suppressible hyperaldosteronism: lessons for essential hypertension?
    Connell JM, Jamieson AJ, Davies E, Ingram M, Soro A, Fraser R.
    Endocr Res; 1996 Nov; 22(4):691-700. PubMed ID: 8969929
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  • 6. Assessment of 11beta-hydroxylase activity with plasma corticosterone, deoxycorticosterone, cortisol, and deoxycortisol: role of ACTH and angiotensin.
    Ganguly A, Meikle AW, Tyler FH, West CD.
    J Clin Endocrinol Metab; 1977 Mar; 44(3):560-8. PubMed ID: 190262
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  • 7. Aldosterone synthase gene variation and adrenocortical response to sodium status, angiotensin II and ACTH in normal male subjects.
    Kennon B, Ingram MC, Friel EC, Anderson NH, MacKenzie SM, Davies E, Shakerdi L, Wallace AM, Fraser R, Connell JM.
    Clin Endocrinol (Oxf); 2004 Aug; 61(2):174-81. PubMed ID: 15272911
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  • 11. Simultaneous radioimmunoassay of plasma aldosterone, corticosterone, 11-deoxycorticosterone, progesterone, 17-hydroxyprogesterone, 11-deoxycortisol, cortisol and cortisone.
    Sippell WG, Bidlingmaier F, Becker H, Brünig T, Dörr H, Hahn H, Golder W, Hollmann G, Knorr D.
    J Steroid Biochem; 1978 Jan; 9(1):63-74. PubMed ID: 203774
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  • 12. Longitudinal study of progestins, mineralocorticoids, and glucocorticoids throughout human pregnancy.
    Dörr HG, Heller A, Versmold HT, Sippell WG, Herrmann M, Bidlingmaier F, Knorr D.
    J Clin Endocrinol Metab; 1989 May; 68(5):863-8. PubMed ID: 2715289
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  • 13. Clinical and gene mutation studies on a Chinese pedigree with glucocorticoid-remediable aldosteronism.
    Ding W, Liu L, Hu R, Xu M, Chen J.
    Chin Med J (Engl); 2002 Jul; 115(7):979-82. PubMed ID: 12150724
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  • 14. Response of several adrenal steroids to ACTH stimulation in essential hypertension.
    Honda M, Nowaczynski W, Guthrie GP, Messerli FH, Tolis G, Kuchel O, Genest J.
    J Clin Endocrinol Metab; 1977 Feb; 44(2):264-72. PubMed ID: 190252
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  • 16. A German family with glucocorticoid-remediable aldosteronism.
    Vonend O, Altenhenne C, Büchner NJ, Dekomien G, Maser-Gluth C, Weiner SM, Sellin L, Hofebauer S, Epplen JT, Rump LC.
    Nephrol Dial Transplant; 2007 Apr; 22(4):1123-30. PubMed ID: 17277347
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  • 17. Aldosterone-producing adenomas do not contain glucocorticoid-remediable aldosteronism chimeric gene duplications.
    Carroll J, Dluhy R, Fallo F, Pistorello M, Bradwin G, Gomez-Sanchez CE, Mortensen R.
    J Clin Endocrinol Metab; 1996 Dec; 81(12):4310-2. PubMed ID: 8954032
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  • 18. Partial deficiency of adrenal 11-hydroxylase. A possible cause of primary hypertension.
    de Simone G, Tommaselli AP, Rossi R, Valentino R, Lauria R, Scopacasa F, Lombardi G.
    Hypertension; 1985 Dec; 7(2):204-10. PubMed ID: 2984117
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  • 19. Continuous adrenocorticotropin administration in hypopituitarism produces asynchronous increases of deoxycorticosterone and 11-deoxycortisol relative to other reduced zona fasciculata steroids.
    Kater CE, Irony I, Biglieri EG, Faiçal S.
    J Clin Endocrinol Metab; 1990 Aug; 71(2):305-10. PubMed ID: 2166067
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  • 20. Aldosterone, corticosterone, cortisol, 11-deoxycortisol and 11-deoxycorticosterone concentrations in the blood of chronically cannulated ovine foetuses: effect of ACTH.
    Brown EH, Coghlan JP, Hardy KJ, Wintour EM.
    Acta Endocrinol (Copenh); 1978 Jun; 88(2):364-74. PubMed ID: 208342
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