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9. N- and P-type Ca2+ channels are involved in acetylcholine release at a neuroneuronal synapse: only the N-type channel is the target of neuromodulators. Fossier P, Baux G, Tauc L. Proc Natl Acad Sci U S A; 1994 May 24; 91(11):4771-5. PubMed ID: 7910963 [Abstract] [Full Text] [Related]
10. Cyclic ADP-ribose and calcium-induced calcium release regulate neurotransmitter release at a cholinergic synapse of Aplysia. Mothet JP, Fossier P, Meunier FM, Stinnakre J, Tauc L, Baux G. J Physiol; 1998 Mar 01; 507 ( Pt 2)(Pt 2):405-14. PubMed ID: 9518701 [Abstract] [Full Text] [Related]
11. NO decreases evoked quantal ACh release at a synapse of Aplysia by a mechanism independent of Ca2+ influx and protein kinase G. Mothet JP, Fossier P, Tauc L, Baux G. J Physiol; 1996 Jun 15; 493 ( Pt 3)(Pt 3):769-84. PubMed ID: 8799898 [Abstract] [Full Text] [Related]
12. Presynaptic inhibition produced by an identified presynaptic inhibitory neuron. II. Presynaptic conductance changes caused by histamine. Kretz R, Shapiro E, Bailey CH, Chen M, Kandel ER. J Neurophysiol; 1986 Jan 15; 55(1):131-46. PubMed ID: 2419525 [Abstract] [Full Text] [Related]
13. G proteins are involved in the regulation of transmitter release at an Aplysia cholinergic synapse. Baux G, Fossier P, Tauc L. Brain Res; 1992 Mar 13; 575(1):155-8. PubMed ID: 1354552 [Abstract] [Full Text] [Related]
18. Acetylcholine modulation of high-voltage-activated calcium channels in the neurones acutely dissociated from rat paratracheal ganglia. Murai Y, Ishibashi H, Akaike N, Ito Y. Br J Pharmacol; 1998 Apr 13; 123(7):1441-9. PubMed ID: 9579741 [Abstract] [Full Text] [Related]