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252 related items for PubMed ID: 8098843

  • 1. A conserved C-terminal sequence that is deleted in v-ErbA is essential for the biological activities of c-ErbA (the thyroid hormone receptor).
    Saatcioglu F, Bartunek P, Deng T, Zenke M, Karin M.
    Mol Cell Biol; 1993 Jun; 13(6):3675-85. PubMed ID: 8098843
    [Abstract] [Full Text] [Related]

  • 2. Role of the conserved C-terminal region of thyroid hormone receptor-alpha in ligand-dependent transcriptional activation.
    Selmi-Ruby S, Casanova J, Malhotra S, Roussett B, Raaka BM, Samuels HH.
    Mol Cell Endocrinol; 1998 Mar 16; 138(1-2):105-14. PubMed ID: 9685219
    [Abstract] [Full Text] [Related]

  • 3. Mutations in the conserved C-terminal sequence in thyroid hormone receptor dissociate hormone-dependent activation from interference with AP-1 activity.
    Saatcioglu F, Lopez G, West BL, Zandi E, Feng W, Lu H, Esmaili A, Apriletti JW, Kushner PJ, Baxter JD, Karin M.
    Mol Cell Biol; 1997 Aug 16; 17(8):4687-95. PubMed ID: 9234725
    [Abstract] [Full Text] [Related]

  • 4. Myb-Ets fusion oncoprotein inhibits thyroid hormone receptor/c-ErbA and retinoic acid receptor functions: a novel mechanism of action for leukemogenic transformation by E26 avian retrovirus.
    Rascle A, Ferrand N, Gandrillon O, Samarut J.
    Mol Cell Biol; 1996 Nov 16; 16(11):6338-51. PubMed ID: 8887663
    [Abstract] [Full Text] [Related]

  • 5. Thyroid hormone receptor/and v-erbA. A single amino acid difference in the C-terminal region influences dominant negative activity and receptor dimer formation.
    Selmi S, Samuels HH.
    J Biol Chem; 1991 Jun 25; 266(18):11589-93. PubMed ID: 1675637
    [Abstract] [Full Text] [Related]

  • 6. Modulation of normal erythroid differentiation by the endogenous thyroid hormone and retinoic acid receptors: a possible target for v-erbA oncogene action.
    Schroeder C, Gibson L, Zenke M, Beug H.
    Oncogene; 1992 Feb 25; 7(2):217-27. PubMed ID: 1347914
    [Abstract] [Full Text] [Related]

  • 7. v-erbA oncogene activation entails the loss of hormone-dependent regulator activity of c-erbA.
    Zenke M, Muñoz A, Sap J, Vennström B, Beug H.
    Cell; 1990 Jun 15; 61(6):1035-49. PubMed ID: 1972036
    [Abstract] [Full Text] [Related]

  • 8. Thyroid hormone receptor transcriptional activity is potentially autoregulated by truncated forms of the receptor.
    Bigler J, Hokanson W, Eisenman RN.
    Mol Cell Biol; 1992 May 15; 12(5):2406-17. PubMed ID: 1314955
    [Abstract] [Full Text] [Related]

  • 9. v-erbA overexpression is required to extinguish c-erbA function in erythroid cell differentiation and regulation of the erbA target gene CAII.
    Disela C, Glineur C, Bugge T, Sap J, Stengl G, Dodgson J, Stunnenberg H, Beug H, Zenke M.
    Genes Dev; 1991 Nov 15; 5(11):2033-47. PubMed ID: 1682217
    [Abstract] [Full Text] [Related]

  • 10. A transferable silencing domain is present in the thyroid hormone receptor, in the v-erbA oncogene product and in the retinoic acid receptor.
    Baniahmad A, Köhne AC, Renkawitz R.
    EMBO J; 1992 Mar 15; 11(3):1015-23. PubMed ID: 1347744
    [Abstract] [Full Text] [Related]

  • 11. c-ErbA, but not v-ErbA, competes with a putative erythroid repressor for binding to the carbonic anhydrase II promoter.
    Rascle A, Ghysdael J, Samarut J.
    Oncogene; 1994 Oct 15; 9(10):2853-67. PubMed ID: 7916146
    [Abstract] [Full Text] [Related]

  • 12. Isolation of a cDNA encoding human Rev-ErbA alpha: transcription from the noncoding DNA strand of a thyroid hormone receptor gene results in a related protein that does not bind thyroid hormone.
    Lazar MA, Jones KE, Chin WW.
    DNA Cell Biol; 1990 Mar 15; 9(2):77-83. PubMed ID: 1971514
    [Abstract] [Full Text] [Related]

  • 13. The lack of transcriptional activation of the v-erbA oncogene is in part due to a mutation present in the DNA binding domain of the protein.
    de Verneuil H, Metzger D.
    Nucleic Acids Res; 1990 Aug 11; 18(15):4489-97. PubMed ID: 1975094
    [Abstract] [Full Text] [Related]

  • 14. Unliganded T3R, but not its oncogenic variant, v-erbA, suppresses RAR-dependent transactivation by titrating out RXR.
    Barettino D, Bugge TH, Bartunek P, Vivanco Ruiz MD, Sonntag-Buck V, Beug H, Zenke M, Stunnenberg HG.
    EMBO J; 1993 Apr 11; 12(4):1343-54. PubMed ID: 8096810
    [Abstract] [Full Text] [Related]

  • 15. Requirement for the C-terminal domain of the v-erbA oncogene protein for biological function and transcriptional repression.
    Forrest D, Muñoz A, Raynoschek C, Vennström B, Beug H.
    Oncogene; 1990 Mar 11; 5(3):309-16. PubMed ID: 1969136
    [Abstract] [Full Text] [Related]

  • 16. A novel cis element mediating ligand-independent activation by c-ErbA: implications for hormonal regulation.
    Saatcioglu F, Deng T, Karin M.
    Cell; 1993 Dec 17; 75(6):1095-105. PubMed ID: 7903219
    [Abstract] [Full Text] [Related]

  • 17. The v-erbA oncogene requires cooperation with tyrosine kinases to arrest erythroid differentiation induced by ligand-activated endogenous c-erbA and retinoic acid receptor.
    Schroeder C, Gibson L, Beug H.
    Oncogene; 1992 Feb 17; 7(2):203-16. PubMed ID: 1347913
    [Abstract] [Full Text] [Related]

  • 18. The thyroid hormone receptor interferes with transcriptional activation via the AP-1 complex.
    Schmidt ED, Cramer SJ, Offringa R.
    Biochem Biophys Res Commun; 1993 Apr 15; 192(1):151-60. PubMed ID: 8386506
    [Abstract] [Full Text] [Related]

  • 19. Roles of v-erbA homodimers and heterodimers in mediating dominant negative activity by v-erbA.
    Yen PM, Ikeda M, Brubaker JH, Forgione M, Sugawara A, Chin WW.
    J Biol Chem; 1994 Jan 14; 269(2):903-9. PubMed ID: 7904604
    [Abstract] [Full Text] [Related]

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