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Journal Abstract Search

127 related items for PubMed ID: 8147881

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  • 9. Partial reconstitution of the respiratory burst oxidase in lymphoblastoid B cell lines lacking p67-phox after transfection with an expression vector containing wild-type and mutant p67 -phox cDNAs: Deletions of the carboxy and amino terminal residues of p67-phox are not required for activity.
    Chanock SJ, Faust LR, Barrett D, Christensen B, Newburger PE, Babior BM.
    Exp Hematol; 1996 Mar; 24(4):531-6. PubMed ID: 8608803
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  • 10. Statistical and mutational analysis of chronic granulomatous disease in Japan with special reference to gp91-phox and p22-phox deficiency.
    Ishibashi F, Nunoi H, Endo F, Matsuda I, Kanegasaki S.
    Hum Genet; 2000 May; 106(5):473-81. PubMed ID: 10914676
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  • 11. Mutational analysis of patients with p47-phox-deficient chronic granulomatous disease: The significance of recombination events between the p47-phox gene (NCF1) and its highly homologous pseudogenes.
    Vázquez N, Lehrnbecher T, Chen R, Christensen BL, Gallin JI, Malech H, Holland S, Zhu S, Chanock SJ.
    Exp Hematol; 2001 Feb; 29(2):234-43. PubMed ID: 11166463
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  • 12. Chronic granulomatous disease caused by mutations other than the common GT deletion in NCF1, the gene encoding the p47phox component of the phagocyte NADPH oxidase.
    Roos D, de Boer M, Köker MY, Dekker J, Singh-Gupta V, Ahlin A, Palmblad J, Sanal O, Kurenko-Deptuch M, Jolles S, Wolach B.
    Hum Mutat; 2006 Dec; 27(12):1218-29. PubMed ID: 16972229
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  • 17. Molecular characterization of autosomal recessive chronic granulomatous disease caused by a defect of the nicotinamide adenine dinucleotide phosphate (reduced form) oxidase component p67-phox.
    Patiño PJ, Rae J, Noack D, Erickson R, Ding J, de Olarte DG, Curnutte JT.
    Blood; 1999 Oct 01; 94(7):2505-14. PubMed ID: 10498624
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  • 19. A novel mutation in NCF1 in an adult CGD patient with a liver abscess as first presentation.
    van de Vosse E, van Wengen A, van Geelen JA, de Boer M, Roos D, van Dissel JT.
    J Hum Genet; 2009 Jun 01; 54(6):313-6. PubMed ID: 19329991
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  • 20. A donor splice site mutation in intron 1 of CYBA, leading to chronic granulomatous disease.
    de Boer M, Hartl D, Wintergerst U, Belohradsky BH, Roos D.
    Blood Cells Mol Dis; 2005 Jun 01; 35(3):365-9. PubMed ID: 16157492
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