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Journal Abstract Search

268 related items for PubMed ID: 8253716

  • 1. Tumor necrosis factor-alpha suppresses insulin-induced tyrosine phosphorylation of insulin receptor and its substrates.
    Feinstein R, Kanety H, Papa MZ, Lunenfeld B, Karasik A.
    J Biol Chem; 1993 Dec 15; 268(35):26055-8. PubMed ID: 8253716
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  • 2. Sphingomyelinase and ceramide suppress insulin-induced tyrosine phosphorylation of the insulin receptor substrate-1.
    Kanety H, Hemi R, Papa MZ, Karasik A.
    J Biol Chem; 1996 Apr 26; 271(17):9895-7. PubMed ID: 8626623
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  • 3. Tumor necrosis factor alpha-induced phosphorylation of insulin receptor substrate-1 (IRS-1). Possible mechanism for suppression of insulin-stimulated tyrosine phosphorylation of IRS-1.
    Kanety H, Feinstein R, Papa MZ, Hemi R, Karasik A.
    J Biol Chem; 1995 Oct 06; 270(40):23780-4. PubMed ID: 7559552
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  • 4. Tumor necrosis factor (TNF)-alpha inhibits insulin signaling through stimulation of the p55 TNF receptor and activation of sphingomyelinase.
    Peraldi P, Hotamisligil GS, Buurman WA, White MF, Spiegelman BM.
    J Biol Chem; 1996 May 31; 271(22):13018-22. PubMed ID: 8662983
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  • 7. Phorbol ester-induced serine phosphorylation of the insulin receptor decreases its tyrosine kinase activity.
    Takayama S, White MF, Kahn CR.
    J Biol Chem; 1988 Mar 05; 263(7):3440-7. PubMed ID: 3125181
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  • 11. The effect of tumor necrosis factor-alpha on tissue specificity and selectivity to insulin signaling.
    Yamaguchi K, Higashiura K, Ura N, Murakami H, Hyakukoku M, Furuhashi M, Shimamoto K.
    Hypertens Res; 2003 May 05; 26(5):389-96. PubMed ID: 12887130
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  • 13. Differential effects of tumor necrosis factor-alpha on protein kinase C isoforms alpha and delta mediate inhibition of insulin receptor signaling.
    Rosenzweig T, Braiman L, Bak A, Alt A, Kuroki T, Sampson SR.
    Diabetes; 2002 Jun 05; 51(6):1921-30. PubMed ID: 12031982
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  • 15. Insulin stimulates tyrosine phosphorylation of multiple high molecular weight substrates in Fao hepatoma cells.
    Miralpeix M, Sun XJ, Backer JM, Myers MG, Araki E, White MF.
    Biochemistry; 1992 Sep 22; 31(37):9031-9. PubMed ID: 1382584
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  • 16. Reduced tyrosine kinase activity of the insulin receptor in obesity-diabetes. Central role of tumor necrosis factor-alpha.
    Hotamisligil GS, Budavari A, Murray D, Spiegelman BM.
    J Clin Invest; 1994 Oct 22; 94(4):1543-9. PubMed ID: 7523453
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  • 17. Short-term exposure to tumor necrosis factor-alpha does not affect insulin-stimulated glucose uptake in skeletal muscle.
    Nolte LA, Hansen PA, Chen MM, Schluter JM, Gulve EA, Holloszy JO.
    Diabetes; 1998 May 22; 47(5):721-6. PubMed ID: 9588442
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  • 18. Insulin stimulates PKCzeta -mediated phosphorylation of insulin receptor substrate-1 (IRS-1). A self-attenuated mechanism to negatively regulate the function of IRS proteins.
    Liu YF, Paz K, Herschkovitz A, Alt A, Tennenbaum T, Sampson SR, Ohba M, Kuroki T, LeRoith D, Zick Y.
    J Biol Chem; 2001 Apr 27; 276(17):14459-65. PubMed ID: 11278339
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  • 19. Tumor necrosis factor-alpha- and hyperglycemia-induced insulin resistance. Evidence for different mechanisms and different effects on insulin signaling.
    Kroder G, Bossenmaier B, Kellerer M, Capp E, Stoyanov B, Mühlhöfer A, Berti L, Horikoshi H, Ullrich A, Häring H.
    J Clin Invest; 1996 Mar 15; 97(6):1471-7. PubMed ID: 8617880
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  • 20. IRS-1-mediated inhibition of insulin receptor tyrosine kinase activity in TNF-alpha- and obesity-induced insulin resistance.
    Hotamisligil GS, Peraldi P, Budavari A, Ellis R, White MF, Spiegelman BM.
    Science; 1996 Feb 02; 271(5249):665-8. PubMed ID: 8571133
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