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Journal Abstract Search

107 related items for PubMed ID: 8302600

  • 21. Viral mutations enhance the Max binding properties of the vMyc b-HLH-LZ domain.
    Crouch DH, Fisher F, La Rocca SA, Goding CR, Gillespie DA.
    Nucleic Acids Res; 2005; 33(16):5235-42. PubMed ID: 16166655
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  • 22. Mad1 expression in the absence of differentiation: effect of cAMP on the B-lymphoid cell line Reh.
    Naderi S, Blomhoff HK.
    J Cell Physiol; 1999 Jan; 178(1):76-84. PubMed ID: 9886493
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  • 25. max encodes a sequence-specific DNA-binding protein and is not regulated by serum growth factors.
    Berberich S, Hyde-DeRuyscher N, Espenshade P, Cole M.
    Oncogene; 1992 Apr; 7(4):775-9. PubMed ID: 1565473
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  • 27. Acquisition of essential somatic cell cycle regulatory protein expression and implied activity occurs at the second to third cell division in mouse preimplantation embryos.
    Xie Y, Sun T, Wang QT, Wang Y, Wang F, Puscheck E, Rappolee DA.
    FEBS Lett; 2005 Jan 17; 579(2):398-408. PubMed ID: 15642350
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  • 28. DNA binding of Myc/Max/Mad network complexes to oligonucleotides containing two E box elements: c-Myc/Max heterodimers do not bind DNA cooperatively.
    Vervoorts J, Lüscher B.
    Biol Chem; 1999 Sep 17; 380(9):1121-6. PubMed ID: 10543451
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  • 30. Variant Max protein, derived by alternative splicing, associates with c-Myc in vivo and inhibits transactivation.
    Arsura M, Deshpande A, Hann SR, Sonenshein GE.
    Mol Cell Biol; 1995 Dec 17; 15(12):6702-9. PubMed ID: 8524235
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  • 31. Functional analysis of the carboxy-terminal transforming region of v-Myc: binding to Max is necessary, but not sufficient, for cellular transformation.
    Min S, Mascarenhas NT, Taparowsky EJ.
    Oncogene; 1993 Oct 17; 8(10):2691-701. PubMed ID: 8378081
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  • 32. Sequential expression of the MAD family of transcriptional repressors during differentiation and development.
    Quéva C, Hurlin PJ, Foley KP, Eisenman RN.
    Oncogene; 1998 Feb 26; 16(8):967-77. PubMed ID: 9519870
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  • 34. c-Myc and Max interactions in quiescent and mitogen-stimulated primary hepatocytes.
    Skouteris GG, Schröder CH.
    Exp Cell Res; 1996 Jun 15; 225(2):237-44. PubMed ID: 8660911
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  • 37. Kinetics of myc-max-mad gene expression during hepatocyte proliferation in vivo: Differential regulation of mad family and stress-mediated induction of c-myc.
    Mauleon I, Lombard MN, Muñoz-Alonso MJ, Cañelles M, Leon J.
    Mol Carcinog; 2004 Feb 15; 39(2):85-90. PubMed ID: 14750213
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  • 38. Analysis of the DNA-binding activities of Myc/Max/Mad network complexes during induced differentiation of U-937 monoblasts and F9 teratocarcinoma cells.
    Larsson LG, Bahram F, Burkhardt H, Lüscher B.
    Oncogene; 1997 Aug 07; 15(6):737-48. PubMed ID: 9264414
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  • 39. The C. elegans MDL-1 and MXL-1 proteins can functionally substitute for vertebrate MAD and MAX.
    Yuan J, Tirabassi RS, Bush AB, Cole MD.
    Oncogene; 1998 Sep 03; 17(9):1109-18. PubMed ID: 9764821
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  • 40. Transfected wild-type and mutant max regulate cell growth and differentiation of murine erythroleukemia cells.
    Cogliati T, Dunn BK, Bar-Ner M, Cultraro CM, Segal S.
    Oncogene; 1993 May 03; 8(5):1263-8. PubMed ID: 8479748
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