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282 related items for PubMed ID: 8348149

  • 1. Motheaten and viable motheaten mice have mutations in the haematopoietic cell phosphatase gene.
    Tsui HW, Siminovitch KA, de Souza L, Tsui FW.
    Nat Genet; 1993 Jun; 4(2):124-9. PubMed ID: 8348149
    [Abstract] [Full Text] [Related]

  • 2. Increased G-CSF responsiveness of bone marrow cells from hematopoietic cell phosphatase deficient viable motheaten mice.
    Tapley P, Shevde NK, Schweitzer PA, Gallina M, Christianson SW, Lin IL, Stein RB, Shultz LD, Rosen J, Lamb P.
    Exp Hematol; 1997 Feb; 25(2):122-31. PubMed ID: 9015212
    [Abstract] [Full Text] [Related]

  • 3. Aberrant regulation of bone trace elements in motheaten and osteopetrosis mutant mice.
    Yamada G, Nakamura S, Haraguchi R, Sakai M, Suzuki K, Miyado K, Hasuwa H, Ogino Y, Minami T, Tohno Y, Blum M, Shultz LD.
    Cell Mol Biol (Noisy-le-grand); 1998 Mar; 44(2):315-9. PubMed ID: 9593582
    [Abstract] [Full Text] [Related]

  • 4. Aberrant expression of the NF-kappaB and IkappaB proteins in B cells from viable motheaten mice.
    Khaled AR, Butfiloski EJ, Villas B, Sobel ES, Schiffenbauer J.
    Autoimmunity; 1999 Mar; 30(2):115-28. PubMed ID: 10435725
    [Abstract] [Full Text] [Related]

  • 5. DNA-binding antibodies from viable motheaten mutant mice: implications for B cell tolerance.
    Westhoff CM, Whittier A, Kathol S, McHugh J, Zajicek C, Shultz LD, Wylie DE.
    J Immunol; 1997 Sep 15; 159(6):3024-33. PubMed ID: 9300728
    [Abstract] [Full Text] [Related]

  • 6. Absence of CD5 dramatically reduces progression of pulmonary inflammatory lesions in SHP-1 protein-tyrosine phosphatase-deficient 'viable motheaten' mice.
    Joliat MJ, Lang PA, Lyons BL, Burzenski L, Lynes MA, Yi T, Sundberg JP, Shultz LD.
    J Autoimmun; 2002 Mar 15; 18(2):105-17. PubMed ID: 11908943
    [Abstract] [Full Text] [Related]

  • 7. Molecular basis of the motheaten phenotype.
    Tsui FW, Tsui HW.
    Immunol Rev; 1994 Apr 15; 138():185-206. PubMed ID: 8070815
    [Abstract] [Full Text] [Related]

  • 8. T cell developmental defects in 'viable motheaten' mice deficient in SHP-1 protein-tyrosine phosphatase. Developmental defects are corrected in vitro in the presence of normal hematopoietic-origin stromal cells and in vivo by exogenous IL-7.
    Christianson SW, Greiner DL, Deluca D, Leif J, Phillips NE, Hayes SM, Hayashi S, Joliat MJ, Lyons BL, Shultz LD.
    J Autoimmun; 2002 Mar 15; 18(2):119-30. PubMed ID: 11908944
    [Abstract] [Full Text] [Related]

  • 9. Defective in vitro migratory capacity of bone marrow cells from viable motheaten mice in response to normal thymus culture supernatants.
    Haar JL, Popp JD, Shultz LD.
    Exp Hematol; 1989 Jan 15; 17(1):21-4. PubMed ID: 2562796
    [Abstract] [Full Text] [Related]

  • 10. Functional consequences of the SHP-1 defect in motheaten viable mice: role of NF-kappa B.
    Khaled AR, Butfiloski EJ, Sobel ES, Schiffenbauer J.
    Cell Immunol; 1998 Apr 10; 185(1):49-58. PubMed ID: 9636682
    [Abstract] [Full Text] [Related]

  • 11. Signalling by the W/Kit receptor tyrosine kinase is negatively regulated in vivo by the protein tyrosine phosphatase Shp1.
    Paulson RF, Vesely S, Siminovitch KA, Bernstein A.
    Nat Genet; 1996 Jul 10; 13(3):309-15. PubMed ID: 8673130
    [Abstract] [Full Text] [Related]

  • 12. B cell-specific deletion of protein-tyrosine phosphatase Shp1 promotes B-1a cell development and causes systemic autoimmunity.
    Pao LI, Lam KP, Henderson JM, Kutok JL, Alimzhanov M, Nitschke L, Thomas ML, Neel BG, Rajewsky K.
    Immunity; 2007 Jul 10; 27(1):35-48. PubMed ID: 17600736
    [Abstract] [Full Text] [Related]

  • 13. Identification of PTP1C mutation as the genetic defect in motheaten and viable motheaten mice: a step toward defining the roles of protein tyrosine phosphatases in the regulation of hemopoietic cell differentiation and function.
    Bignon JS, Siminovitch KA.
    Clin Immunol Immunopathol; 1994 Nov 10; 73(2):168-79. PubMed ID: 7923924
    [Abstract] [Full Text] [Related]

  • 14. Hematopoietic cell protein-tyrosine phosphatase-deficient motheaten mice exhibit T cell apoptosis defect.
    Su X, Zhou T, Yang PA, Wang Z, Mountz JD.
    J Immunol; 1996 Jun 01; 156(11):4198-208. PubMed ID: 8666788
    [Abstract] [Full Text] [Related]

  • 15. Deficiency of SHP-1 protein-tyrosine phosphatase in "viable motheaten" mice results in retinal degeneration.
    Lyons BL, Smith RS, Hurd RE, Hawes NL, Burzenski LM, Nusinowitz S, Hasham MG, Chang B, Shultz LD.
    Invest Ophthalmol Vis Sci; 2006 Mar 01; 47(3):1201-9. PubMed ID: 16505059
    [Abstract] [Full Text] [Related]

  • 16. The Src-homology domain 2-bearing protein tyrosine phosphatase-1 inhibits antigen receptor-induced apoptosis of activated peripheral T cells.
    Zhang J, Somani AK, Watt S, Mills GB, Siminovitch KA.
    J Immunol; 1999 Jun 01; 162(11):6359-67. PubMed ID: 10352248
    [Abstract] [Full Text] [Related]

  • 17. Severe defects in immunity and hematopoiesis caused by SHP-1 protein-tyrosine-phosphatase deficiency.
    Shultz LD, Rajan TV, Greiner DL.
    Trends Biotechnol; 1997 Aug 01; 15(8):302-7. PubMed ID: 9263478
    [Abstract] [Full Text] [Related]

  • 18. B and T cells are not required for the viable motheaten phenotype.
    Yu CC, Tsui HW, Ngan BY, Shulman MJ, Wu GE, Tsui FW.
    J Exp Med; 1996 Feb 01; 183(2):371-80. PubMed ID: 8627150
    [Abstract] [Full Text] [Related]

  • 19. Expression and function of the protein tyrosine phosphatase SHP-1 in oligodendrocytes.
    Massa PT, Saha S, Wu C, Jarosinski KW.
    Glia; 2000 Feb 15; 29(4):376-85. PubMed ID: 10652447
    [Abstract] [Full Text] [Related]

  • 20. Lack of requirement for SHP-1 in both Fas-mediated and perforin-mediated cell death induced by CTL.
    Takayama H, Lee MH, Shirota-Someya Y.
    J Immunol; 1996 Nov 01; 157(9):3943-8. PubMed ID: 8892626
    [Abstract] [Full Text] [Related]

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