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2. Superoxide responses to formyl-methionyl-leucyl-phenylalanine in primed neutrophils. Role of intracellular and extracellular calcium. Walker BA, Hagenlocker BE, Ward PA. J Immunol; 1991 May 01; 146(9):3124-31. PubMed ID: 1849943 [Abstract] [Full Text] [Related]
3. Enhancement of chemotactic peptide-induced activation of phosphoinositide 3-kinase by granulocyte-macrophage colony-stimulating factor and its relation to the cytokine-mediated priming of neutrophil superoxide-anion production. Kodama T, Hazeki K, Hazeki O, Okada T, Ui M. Biochem J; 1999 Jan 15; 337 ( Pt 2)(Pt 2):201-9. PubMed ID: 9882616 [Abstract] [Full Text] [Related]
4. Differential inhibition of human neutrophil activation by cyclosporins A, D, and H. Cyclosporin H is a potent and effective inhibitor of formyl peptide-induced superoxide formation. Wenzel-Seifert K, Grünbaum L, Seifert R. J Immunol; 1991 Sep 15; 147(6):1940-6. PubMed ID: 1653806 [Abstract] [Full Text] [Related]
5. Dual effects of formylpeptides on the adhesion of endotoxin-primed human neutrophils. Bellavite P, Chirumbolo S, Lippi G, Andrioli G, Bonazzi L, Ferro I. Cell Biochem Funct; 1993 Dec 15; 11(4):231-9. PubMed ID: 7903912 [Abstract] [Full Text] [Related]
6. Superoxide anion production and phospholipase D-mediated generation of diacylglycerol are subnormal after N-formyl-methionyl-leucyl-phenylalanine stimulation of polymorphonuclear granulocytes in polycythemia vera. Samuelsson J, Hansson A, Rosendahl K, Palmblad J. J Lab Clin Med; 1993 Feb 15; 121(2):310-9. PubMed ID: 8381848 [Abstract] [Full Text] [Related]
7. Temporal adaptation of neutrophil oxidative responsiveness to n-formyl-methionyl-leucyl-phenylalanine. Acceleration by granulocyte-macrophage colony stimulating factor. English D, Broxmeyer HE, Gabig TG, Akard LP, Williams DE, Hoffman R. J Immunol; 1988 Oct 01; 141(7):2400-6. PubMed ID: 3049807 [Abstract] [Full Text] [Related]
8. Activation and priming of human monocytes by monocyte chemotactic activating factor: cooperation with other inflammatory cytokines and close association between an increase in cytoplasmic free Ca2+ and intracellular acidification. Azuma EK, Yuo A, Matsushima T, Kasahara T, Mizoguchi H, Saito M, Takaku F, Kitagawa S. Exp Hematol; 1996 Feb 01; 24(2):169-75. PubMed ID: 8641338 [Abstract] [Full Text] [Related]
9. Temporal adaptation of human neutrophil metabolic responsiveness to the peptide formylmethionyl-leucyl phenylalanine: a comparison between human neutrophils and granule-depleted neutrophil cytoplasts. Dahlgren C. Cell Biochem Funct; 1990 Jan 01; 8(1):57-64. PubMed ID: 2160338 [Abstract] [Full Text] [Related]
10. (2R,3R)-2-(3',4'-dihydroxybenzyl)-3-(3'',4''-dimethoxybenzyl)butyrolactone suppresses fMLP-induced superoxide production by inhibiting fMLP-receptor binding in human neutrophils. Huang YJ, Chen IS, Tseng CP, Day YJ, Lin YC, Liao CH. Biochem Pharmacol; 2008 Feb 01; 75(3):688-97. PubMed ID: 17988652 [Abstract] [Full Text] [Related]
11. Interleukin-8 primes human neutrophils for enhanced superoxide anion production. Wozniak A, Betts WH, Murphy GA, Rokicinski M. Immunology; 1993 Aug 01; 79(4):608-15. PubMed ID: 8406585 [Abstract] [Full Text] [Related]
12. Desensitization of formyl peptide receptors is abolished in calcium ionophore-primed neutrophils: an association of the ligand-receptor complex to the cytoskeleton is not required for a rapid termination of the NADPH-oxidase response. Liu L, Harbecke O, Elwing H, Follin P, Karlsson A, Dahlgren C. J Immunol; 1998 Mar 01; 160(5):2463-8. PubMed ID: 9498791 [Abstract] [Full Text] [Related]
13. Activation of human neutrophils by substance P: effect on FMLP-stimulated oxidative and arachidonic acid metabolism and on antibody-dependent cell-mediated cytotoxicity. Wozniak A, McLennan G, Betts WH, Murphy GA, Scicchitano R. Immunology; 1989 Nov 01; 68(3):359-64. PubMed ID: 2480329 [Abstract] [Full Text] [Related]
14. ATP induces transient elevations of [Ca2+]i in human neutrophils and primes these cells for enhanced O2- generation. Kuhns DB, Wright DG, Nath J, Kaplan SS, Basford RE. Lab Invest; 1988 Apr 01; 58(4):448-53. PubMed ID: 2833660 [Abstract] [Full Text] [Related]
15. Intracellular pH modulates the generation of superoxide radicals by human neutrophils. Simchowitz L. J Clin Invest; 1985 Sep 01; 76(3):1079-89. PubMed ID: 2995444 [Abstract] [Full Text] [Related]
16. Prostaglandin E1 inhibits N-formyl-methionyl-leucyl-phenylalanine-mediated depolarization responses by decreasing the proportion of responsive cells without affecting chemotaxin-induced forward light scatter changes. Fletcher MP. J Immunol; 1987 Dec 15; 139(12):4167-73. PubMed ID: 3693900 [Abstract] [Full Text] [Related]
17. Deactivation of guinea pig pulmonary alveolar macrophage responses to N-formyl-methionyl-leucyl-phenylalanine: chemotaxis, superoxide generation, and binding. Daughaday CC, Mehta J, Spilberg I, Atkinson JP. J Immunol; 1985 Mar 15; 134(3):1823-6. PubMed ID: 2981922 [Abstract] [Full Text] [Related]
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19. Characterization of N-formyl-methionyl-leucyl-phenylalanine receptors on human neutrophils. Effects of isolation and temperature on receptor expression and functional activity. Tennenberg SD, Zemlan FP, Solomkin JS. J Immunol; 1988 Dec 01; 141(11):3937-44. PubMed ID: 2846695 [Abstract] [Full Text] [Related]
20. Rebamipide suppresses formyl-methionyl-leucyl-phenylalanine (fMLP)-induced superoxide production by inhibiting fMLP-receptor binding in human neutrophils. Nagano C, Azuma A, Ishiyama H, Sekiguchi K, Imagawa K, Kikuchi M. J Pharmacol Exp Ther; 2001 Apr 01; 297(1):388-94. PubMed ID: 11259567 [Abstract] [Full Text] [Related] Page: [Next] [New Search]