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Journal Abstract Search

225 related items for PubMed ID: 8392595

  • 1. Fusion-defective mutants of mouse hepatitis virus A59 contain a mutation in the spike protein cleavage signal.
    Gombold JL, Hingley ST, Weiss SR.
    J Virol; 1993 Aug; 67(8):4504-12. PubMed ID: 8392595
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  • 2. Identification of peplomer cleavage site mutations arising during persistence of MHV-A59.
    Gombold JL, Hingley ST, Weiss SR.
    Adv Exp Med Biol; 1993 Aug; 342():157-63. PubMed ID: 8209723
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  • 5. MHV-A59 fusion mutants are attenuated and display altered hepatotropism.
    Hingley ST, Gombold JL, Lavi E, Weiss SR.
    Virology; 1994 Apr; 200(1):1-10. PubMed ID: 8128613
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  • 8. The spike protein of murine coronavirus mouse hepatitis virus strain A59 is not cleaved in primary glial cells and primary hepatocytes.
    Hingley ST, Leparc-Goffart I, Weiss SR.
    J Virol; 1998 Feb; 72(2):1606-9. PubMed ID: 9445064
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  • 11. Coronavirus-induced membrane fusion requires the cysteine-rich domain in the spike protein.
    Chang KW, Sheng Y, Gombold JL.
    Virology; 2000 Mar 30; 269(1):212-24. PubMed ID: 10725213
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  • 14. The virulence of mouse hepatitis virus strain A59 is not dependent on efficient spike protein cleavage and cell-to-cell fusion.
    Hingley ST, Leparc-Goffart I, Seo SH, Tsai JC, Weiss SR.
    J Neurovirol; 2002 Oct 30; 8(5):400-10. PubMed ID: 12402166
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  • 15. The C12 mutant of MHV-A59 is very weakly demyelinating and has five amino acid substitutions restricted to the spike and replicase genes.
    Leparc-Goffart I, Hingley ST, Jiang X, Chua MM, Lavi E, Weiss SR.
    Adv Exp Med Biol; 1998 Oct 30; 440():627-33. PubMed ID: 9782338
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  • 16. Murine coronavirus evolution in vivo: functional compensation of a detrimental amino acid substitution in the receptor binding domain of the spike glycoprotein.
    Navas-Martin S, Hingley ST, Weiss SR.
    J Virol; 2005 Jun 30; 79(12):7629-40. PubMed ID: 15919915
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  • 17. Acquired fusion activity of a murine coronavirus MHV-2 variant with mutations in the proteolytic cleavage site and the signal sequence of the S protein.
    Yamada YK, Takimoto K, Yabe M, Taguchi F.
    Virology; 1997 Jan 06; 227(1):215-9. PubMed ID: 9007076
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  • 18. Neutralization-resistant variants of a neurotropic coronavirus are generated by deletions within the amino-terminal half of the spike glycoprotein.
    Gallagher TM, Parker SE, Buchmeier MJ.
    J Virol; 1990 Feb 06; 64(2):731-41. PubMed ID: 1688627
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  • 19. Murine coronavirus spike protein determines the ability of the virus to replicate in the liver and cause hepatitis.
    Navas S, Seo SH, Chua MM, Das Sarma J, Lavi E, Hingley ST, Weiss SR.
    J Virol; 2001 Mar 06; 75(5):2452-7. PubMed ID: 11160748
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  • 20. Function of a 5'-end genomic RNA mutation that evolves during persistent mouse hepatitis virus infection in vitro.
    Chen W, Baric RS.
    J Virol; 1995 Dec 06; 69(12):7529-40. PubMed ID: 7494259
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