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139 related items for PubMed ID: 8425273

  • 1. Effects of five phorbol esters on gap junctional intercellular communication, morphological transformation and epidermal growth factor binding in Syrian hamster embryo cells.
    Husøy T, Mikalsen SO, Sanner T.
    Carcinogenesis; 1993 Jan; 14(1):73-7. PubMed ID: 8425273
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  • 2. 12-O-tetradecanoylphorbol-13-acetate-induced inhibition of gap junctional communication is differentially regulated in a transformation-sensitive Syrian hamster embryo cell line compared to early passage SHE cells.
    Roseng LE, Rivedal E, Sanner T.
    Carcinogenesis; 1993 Sep; 14(9):1851-5. PubMed ID: 8403209
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  • 3. Both tumour-promoting and non-promoting phorbol esters inhibit [125I]EGF binding and stimulate the phosphorylation of an 80 kd protein kinase C substrate protein in intact quiescent swiss 3T3 cells.
    Brooks G, Brooks SF.
    Carcinogenesis; 1990 Apr; 11(4):667-72. PubMed ID: 2323005
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  • 4. Regulation of gap junctional communication in Syrian hamster embryo cells by retinoic acid and 12-O-tetradecanoylphorbol-13-acetate.
    Rivedal E, Sanner T.
    Carcinogenesis; 1992 Feb; 13(2):199-203. PubMed ID: 1310904
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  • 7. Intercellular communication in colonies of Syrian hamster embryo cells and the susceptibility for morphological transformation.
    Mikalsen SO, Sanner T.
    Carcinogenesis; 1993 Feb; 14(2):251-7. PubMed ID: 8435866
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  • 9. The non-phorbol ester tumor promoter okadaic acid does not promote morphological transformation or inhibit junctional communication in hamster embryo cells.
    Rivedal E, Mikalsen SO, Sanner T.
    Biochem Biophys Res Commun; 1990 Mar 30; 167(3):1302-8. PubMed ID: 2322276
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  • 10. Increased gap junctional intercellular communication in Syrian hamster embryo cells treated with oxidative agents.
    Mikalsen SO, Sanner T.
    Carcinogenesis; 1994 Feb 30; 15(2):381-7. PubMed ID: 8313532
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  • 11. Effect of 1,1'-(2,2,2-trichloroethylidene)-bis(4-chlorobenzene) (DDT) on gap junctional intercellular communication and morphological transformation of Syrian hamster embryo cells.
    Roseng LE, Rivedal E, Skaare JU, Sanner T.
    Chem Biol Interact; 1994 Jan 30; 90(1):73-85. PubMed ID: 8131221
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  • 12. Modification of gap junctional intercellular communication by changes in extracellular pH in Syrian hamster embryo cells.
    Ruch RJ, Klaunig JE, Kerckaert GA, LeBoeuf RA.
    Carcinogenesis; 1990 Jun 30; 11(6):909-13. PubMed ID: 2347066
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  • 13. Effect of glucocorticoids on TPA-induced inhibition of gap-junctional communication and morphological transformation in Syrian hamster embryo cells.
    Roseng LE, Rivedal E.
    Cancer Lett; 1993 Aug 16; 72(1-2):25-30. PubMed ID: 8402570
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  • 14. HL-60 cell differentiation induced by phorbol- and 12-deoxyphorbol-esters.
    Ryves WJ, Dimitrijevic S, Gordge PC, Evans FJ.
    Carcinogenesis; 1994 Nov 16; 15(11):2501-6. PubMed ID: 7955099
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  • 15. The use of initiated cells as a test system for the detection of inhibitors of gap junctional intercellular communication.
    Jansen LA, Jongen WM.
    Carcinogenesis; 1996 Feb 16; 17(2):333-9. PubMed ID: 8625460
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  • 18. Inhibition of intercellular communication and enhancement of morphological transformation of Syrian hamster embryo cells by TPA. Use of TPA-sensitive and TPA-resistant cell lines.
    Rivedal E, Sanner T, Enomoto T, Yamasaki H.
    Carcinogenesis; 1985 Jun 16; 6(6):899-902. PubMed ID: 4006076
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  • 19. Recovery of gap junctional intercellular communication after phorbol ester treatment requires proteasomal degradation of protein kinase C.
    Leithe E, Cruciani V, Sanner T, Mikalsen SO, Rivedal E.
    Carcinogenesis; 2003 Jul 16; 24(7):1239-45. PubMed ID: 12807762
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  • 20. Phorbol ester-mediated inhibition of intercellular communication in BALB/c 3T3 cells: relationship to enhancement of cell transformation.
    Enomoto T, Yamasaki H.
    Cancer Res; 1985 Jun 16; 45(6):2681-8. PubMed ID: 3986803
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