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Journal Abstract Search

146 related items for PubMed ID: 8456975

  • 1. Lysophosphatidylcholine modifies G protein-dependent signaling in porcine endothelial cells.
    Flavahan NA.
    Am J Physiol; 1993 Mar; 264(3 Pt 2):H722-7. PubMed ID: 8456975
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  • 2. Natural course of the impairment of endothelium-dependent relaxations after balloon endothelium removal in porcine coronary arteries. Possible dysfunction of a pertussis toxin-sensitive G protein.
    Shimokawa H, Flavahan NA, Vanhoutte PM.
    Circ Res; 1989 Sep; 65(3):740-53. PubMed ID: 2504508
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  • 3. Pertussis toxin inhibits endothelium-dependent relaxations to certain agonists in porcine coronary arteries.
    Flavahan NA, Shimokawa H, Vanhoutte PM.
    J Physiol; 1989 Jan; 408():549-60. PubMed ID: 2778738
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  • 5. Guanosine 5'-O-(3-thiotriphosphate) causes endothelium-dependent, pertussis toxin-sensitive relaxations in porcine coronary arteries.
    Shibano T, Codina J, Birnbaumer L, Vanhoutte PM.
    Biochem Biophys Res Commun; 1992 Nov 30; 189(1):324-9. PubMed ID: 1449486
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  • 6. Impairment of G-protein-mediated signal transduction in the porcine coronary endothelium during rejection after heart transplantation.
    Perrault LP, Bidouard JP, Janiak P, Villeneuve N, Bruneval P, Vilaine JP, Vanhoutte PM.
    Cardiovasc Res; 1999 Aug 01; 43(2):457-70. PubMed ID: 10536676
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  • 7. Pertussis toxin reduces endothelium-dependent and independent responses to alpha-2- adrenergic stimulation in systemic canine arteries and veins.
    Miller VM, Flavahan NA, Vanhoutte PM.
    J Pharmacol Exp Ther; 1991 Apr 01; 257(1):290-3. PubMed ID: 1850467
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  • 8. Endothelium-derived hyperpolarizing factor mediated relaxations in pig coronary arteries do not involve Gi/o proteins.
    Ng KF, Leung SW, Man RY, Vanhoutte PM.
    Acta Pharmacol Sin; 2008 Dec 01; 29(12):1419-24. PubMed ID: 19026160
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  • 9. Oxidized low density lipoproteins inhibit relaxations of porcine coronary arteries. Role of scavenger receptor and endothelium-derived nitric oxide.
    Tanner FC, Noll G, Boulanger CM, Lüscher TF.
    Circulation; 1991 Jun 01; 83(6):2012-20. PubMed ID: 2040054
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  • 12. Different activation of L-arginine pathway by bradykinin, serotonin, and clonidine in coronary arteries.
    Richard V, Tanner FC, Tschudi M, Lüscher TF.
    Am J Physiol; 1990 Nov 01; 259(5 Pt 2):H1433-9. PubMed ID: 2122744
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  • 13. Analysis of lysophophatidylcholine-induced endothelial dysfunction.
    Freeman JE, Kuo WY, Drenger B, Barnett TN, Levine MA, Flavahan NA.
    J Cardiovasc Pharmacol; 1996 Sep 01; 28(3):345-52. PubMed ID: 8877579
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  • 15. Importance of endothelium-derived nitric oxide in porcine coronary resistance arteries.
    Tschudi M, Richard V, Bühler FR, Lüscher TF.
    Am J Physiol; 1991 Jan 01; 260(1 Pt 2):H13-20. PubMed ID: 1992790
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  • 16. Effects of atrial and brain natriuretic peptides on lysophosphatidylcholine-mediated endothelial dysfunction.
    Murohara T, Kugiyama K, Ota Y, Doi H, Ogata N, Ohgushi M, Yasue H.
    J Cardiovasc Pharmacol; 1999 Dec 01; 34(6):870-8. PubMed ID: 10598132
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  • 18. LPC in oxidized LDL elicits vasocontraction and inhibits endothelium- dependent relaxation.
    Murohara T, Kugiyama K, Ohgushi M, Sugiyama S, Ohta Y, Yasue H.
    Am J Physiol; 1994 Dec 01; 267(6 Pt 2):H2441-9. PubMed ID: 7810742
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  • 20. Lysophosphatidylcholine inhibits endothelium-dependent hyperpolarization and N omega-nitro-L-arginine/indomethacin-resistant endothelium-dependent relaxation in the porcine coronary artery.
    Eizawa H, Yui Y, Inoue R, Kosuga K, Hattori R, Aoyama T, Sasayama S.
    Circulation; 1995 Dec 15; 92(12):3520-6. PubMed ID: 8521575
    [Abstract] [Full Text] [Related]

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