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Journal Abstract Search


160 related items for PubMed ID: 8475501

  • 1. Reduction of hepatic phosphoenolpyruvate carboxykinase (PEPCK) activity by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) is due to decreased mRNA levels.
    Stahl BU, Beer DG, Weber LW, Rozman K.
    Toxicology; 1993 Mar 30; 79(1):81-95. PubMed ID: 8475501
    [Abstract] [Full Text] [Related]

  • 2. Decreased hepatic phosphoenolpyruvate carboxykinase gene expression after 2,3,7,8-tetrachlorodibenzo-p-dioxin treatment: implications for the acute toxicity of chlorinated dibenzo-p-dioxins in the rat.
    Stahl BU, Beer DG, Weber LW, Lebofsky M, Rozman K.
    Arch Toxicol Suppl; 1992 Mar 30; 15():151-5. PubMed ID: 1510582
    [Abstract] [Full Text] [Related]

  • 3. 2,3,7,8-Tetrachlorodibenzo-p-dioxin blocks the physiological regulation of hepatic phosphoenolpyruvate carboxykinase activity in primary rat hepatocytes.
    Stahl BU.
    Toxicology; 1995 Nov 20; 103(1):45-52. PubMed ID: 8525489
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  • 4. Subchronic effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and their reversibility in male Sprague-Dawley rats.
    Li X, Rozman KK.
    Toxicology; 1995 Mar 31; 97(1-3):133-40. PubMed ID: 7716779
    [Abstract] [Full Text] [Related]

  • 5. Effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on liver phosphoenolpyruvate carboxykinase (PEPCK) activity, glucose homeostasis and plasma amino acid concentrations in the most TCDD-susceptible and the most TCDD-resistant rat strains.
    Viluksela M, Unkila M, Pohjanvirta R, Tuomisto JT, Stahl BU, Rozman KK, Tuomisto J.
    Arch Toxicol; 1999 Aug 31; 73(6):323-36. PubMed ID: 10447560
    [Abstract] [Full Text] [Related]

  • 6. Correlation between toxicity and effects on intermediary metabolism in 2,3,7,8-tetrachlorodibenzo-p-dioxin-treated male C57BL/6J and DBA/2J mice.
    Weber LW, Lebofsky M, Stahl BU, Smith S, Rozman KK.
    Toxicol Appl Pharmacol; 1995 Mar 31; 131(1):155-62. PubMed ID: 7878671
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  • 8. Short- and long-term biochemical effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin in female Long-Evans rats.
    Fan F, Rozman KK.
    Toxicol Lett; 1995 Jan 31; 75(1-3):209-16. PubMed ID: 7863529
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  • 9. Reduced activities of key enzymes of gluconeogenesis as possible cause of acute toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in rats.
    Weber LW, Lebofsky M, Stahl BU, Gorski JR, Muzi G, Rozman K.
    Toxicology; 1991 Feb 31; 66(2):133-44. PubMed ID: 1849669
    [Abstract] [Full Text] [Related]

  • 10. Relationship between acute toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and disturbance of intermediary metabolism in the Long-Evans rat.
    Fan F, Rozman KK.
    Arch Toxicol; 1994 Feb 31; 69(2):73-8. PubMed ID: 7717864
    [Abstract] [Full Text] [Related]

  • 11. 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) and 1,2,3,4,7,8-hexachlorodibenzo-p-dioxin (HxCDD) alter body weight by decreasing insulin-like growth factor I (IGF-I) signaling.
    Croutch CR, Lebofsky M, Schramm KW, Terranova PF, Rozman KK.
    Toxicol Sci; 2005 May 31; 85(1):560-71. PubMed ID: 15703265
    [Abstract] [Full Text] [Related]

  • 12. Induction of ethoxyresorufin-O-deethylase and inhibition of glucocorticoid receptor binding in skin and liver of haired and hairless HRS/J mice by topically applied 2,3,7,8-tetrachlorodibenzo-p-dioxin.
    Stohs SJ, Abbott BD, Lin FH, Birnbaum LS.
    Toxicology; 1990 Dec 17; 65(1-2):123-36. PubMed ID: 2274963
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  • 14. Subchronic/chronic toxicity of a mixture of four chlorinated dibenzo-p-dioxins in rats. II. Biochemical effects.
    Viluksela M, Stahl BU, Birnbaum LS, Rozman KK.
    Toxicol Appl Pharmacol; 1998 Jul 17; 151(1):70-8. PubMed ID: 9705888
    [Abstract] [Full Text] [Related]

  • 15. Dioxin-responsive genes: examination of dose-response relationships using quantitative reverse transcriptase-polymerase chain reaction.
    Vanden Heuvel JP, Clark GC, Kohn MC, Tritscher AM, Greenlee WF, Lucier GW, Bell DA.
    Cancer Res; 1994 Jan 01; 54(1):62-8. PubMed ID: 8261464
    [Abstract] [Full Text] [Related]

  • 16. Relationship between CYP1A enzyme activities and protein levels in rats treated with 2,3,7,8-tetrachlorodibenzo-p-dioxin.
    DeVito MJ, Beebe LE, Menache M, Birnbaum LS.
    J Toxicol Environ Health; 1996 Mar 01; 47(4):379-94. PubMed ID: 8600290
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  • 17. Key enzymes of gluconeogenesis are dose-dependently reduced in 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-treated rats.
    Weber LW, Lebofsky M, Greim H, Rozman K.
    Arch Toxicol; 1991 Mar 01; 65(2):119-23. PubMed ID: 2059151
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  • 18. Subchronic/chronic toxicity of 1,2,3,4,6,7,8-heptachlorodibenzo-p-dioxin (HpCDD) in rats. Part II. Biochemical effects.
    Viluksela M, Stahl BU, Birnbaum LS, Rozman KK.
    Toxicol Appl Pharmacol; 1997 Oct 01; 146(2):217-26. PubMed ID: 9344889
    [Abstract] [Full Text] [Related]

  • 19. Biphasic response for hepatic microsomal enzyme induction by 2,3,7,8-tetrachlorodibenzo-p-dioxin in C57BL/6J and DBA/2J mice.
    Shen ES, Guengerich FP, Olson JR.
    Biochem Pharmacol; 1989 Nov 15; 38(22):4075-84. PubMed ID: 2512931
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  • 20. Sepsis-induced alterations in phosphoenolpyruvate carboxykinase expression: the role of insulin and glucagon.
    Deutschman CS, De Maio A, Buchman TG, Clemens MG.
    Circ Shock; 1993 Aug 15; 40(4):295-302. PubMed ID: 8375031
    [Abstract] [Full Text] [Related]


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