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434 related items for PubMed ID: 8509711
1. Evidence that cholesteryl ester and triglyceride accumulation in J774 macrophages induced by very low density lipoprotein subfractions occurs by different mechanisms. Evans AJ, Sawyez CG, Wolfe BM, Connelly PW, Maguire GF, Huff MW. J Lipid Res; 1993 May; 34(5):703-17. PubMed ID: 8509711 [Abstract] [Full Text] [Related]
2. Lipolysis is a prerequisite for lipid accumulation in HepG2 cells induced by large hypertriglyceridemic very low density lipoproteins. Evans AJ, Sawyez CG, Wolfe BM, Huff MW. J Biol Chem; 1992 May 25; 267(15):10743-51. PubMed ID: 1587849 [Abstract] [Full Text] [Related]
3. Inhibition of lipoprotein lipase induced cholesterol ester accumulation in human hepatoma HepG2 cells. Cianflone K, Avramoglu RK, Sawyez C, Huff MW. Atherosclerosis; 1996 Feb 25; 120(1-2):101-14. PubMed ID: 8645351 [Abstract] [Full Text] [Related]
4. Cholesterol accumulation in J774 macrophages induced by triglyceride-rich lipoproteins. Comparison of very low density lipoprotein from subjects with type III, IV, and V hyperlipoproteinemias. Huff MW, Evans AJ, Sawyez CG, Wolfe BM, Nestel PJ. Arterioscler Thromb; 1991 Feb 25; 11(2):221-33. PubMed ID: 1998641 [Abstract] [Full Text] [Related]
5. Characterization of very low density lipoproteins and intermediate density lipoproteins of normo- and hyperlipidemic apolipoprotein E-2 homozygotes. Schmitz G, Assmann G, Augustin J, Dirkes-Kersting A, Brennhaüsen B, Karoff C. J Lipid Res; 1985 Mar 25; 26(3):316-26. PubMed ID: 3989390 [Abstract] [Full Text] [Related]
7. Uptake of type IV hypertriglyceridemic VLDL by cultured macrophages is enhanced by interferon-gamma. Whitman SC, Argmann CA, Sawyez CG, Miller DB, Hegele RA, Huff MW. J Lipid Res; 1999 Jun 25; 40(6):1017-28. PubMed ID: 10357833 [Abstract] [Full Text] [Related]
8. Uptake by J774 macrophages of very-low-density lipoproteins isolated from apoE-deficient mice is mediated by a distinct receptor and stimulated by lipoprotein lipase. Hendriks WL, van der Sman-de Beer F, van Vlijmen BJ, van Vark LC, Hofker MH, Havekes LM. Arterioscler Thromb Vasc Biol; 1997 Mar 25; 17(3):498-504. PubMed ID: 9102168 [Abstract] [Full Text] [Related]
9. Oxidized type IV hypertriglyceridemic VLDL-remnants cause greater macrophage cholesteryl ester accumulation than oxidized LDL. Whitman SC, Sawyez CG, Miller DB, Wolfe BM, Huff MW. J Lipid Res; 1998 May 25; 39(5):1008-20. PubMed ID: 9610767 [Abstract] [Full Text] [Related]
10. Modification of type III VLDL, their remnants, and VLDL from ApoE-knockout mice by p-hydroxyphenylacetaldehyde, a product of myeloperoxidase activity, causes marked cholesteryl ester accumulation in macrophages. Whitman SC, Hazen SL, Miller DB, Hegele RA, Heinecke JW, Huff MW. Arterioscler Thromb Vasc Biol; 1999 May 25; 19(5):1238-49. PubMed ID: 10323775 [Abstract] [Full Text] [Related]
11. Influence of lipoprotein lipase and hepatic lipase on the transformation of VLDL and HDL during lipolysis of VLDL. Murdoch SJ, Breckenridge WC. Atherosclerosis; 1995 Dec 25; 118(2):193-212. PubMed ID: 8770314 [Abstract] [Full Text] [Related]
12. Hypertriglyceridemic very low density lipoproteins induce triglyceride synthesis and accumulation in mouse peritoneal macrophages. Gianturco SH, Bradley WA, Gotto AM, Morrisett JD, Peavy DL. J Clin Invest; 1982 Jul 25; 70(1):168-78. PubMed ID: 6282937 [Abstract] [Full Text] [Related]
13. The role of lipoprotein lipase in the metabolism of triglyceride-rich lipoproteins by macrophages. Lindqvist P, Ostlund-Lindqvist AM, Witztum JL, Steinberg D, Little JA. J Biol Chem; 1983 Aug 10; 258(15):9086-92. PubMed ID: 6874679 [Abstract] [Full Text] [Related]
14. Structural peculiarities of the binding of very low density lipoproteins and low density lipoproteins to the LDL receptor in hypertriglyceridemia: role of apolipoprotein E. Dergunov AD, Smirnova EA, Merched A, Visvikis S, Siest G, Yakushkin VV, Tsibulsky V. Biochim Biophys Acta; 2000 Feb 24; 1484(1):29-40. PubMed ID: 10685028 [Abstract] [Full Text] [Related]
15. Cholesteryl ester accumulation in mouse peritoneal macrophages induced by beta-migrating very low density lipoproteins from patients with atypical dysbetalipoproteinemia. Bersot TP, Innerarity TL, Mahley RW, Havel RJ. J Clin Invest; 1983 Sep 24; 72(3):1024-33. PubMed ID: 6309903 [Abstract] [Full Text] [Related]
16. Lipoprotein lipase- and hepatic triglyceride lipase- promoted very low density lipoprotein degradation proceeds via an apolipoprotein E-dependent mechanism. Medh JD, Fry GL, Bowen SL, Ruben S, Wong H, Chappell DA. J Lipid Res; 2000 Nov 24; 41(11):1858-71. PubMed ID: 11060356 [Abstract] [Full Text] [Related]
17. Very low and intermediate density lipoprotein fractions from apolipoprotein E gene-knockout mice induce cholesteryl ester accumulation in J774 macrophages. Zhang C, Akira M, Hideki H, Hisashi S, Seikoh H. Chin Med J (Engl); 1999 Jun 24; 112(6):543-5. PubMed ID: 11601335 [Abstract] [Full Text] [Related]
18. VLDL-induced triglyceride accumulation in human macrophages is mediated by modulation of LPL lipolytic activity in the absence of change in LPL mass. Milosavljevic D, Kontush A, Griglio S, Le Naour G, Thillet J, Chapman MJ. Biochim Biophys Acta; 2003 Feb 20; 1631(1):51-60. PubMed ID: 12573449 [Abstract] [Full Text] [Related]
19. Apolipoprotein E is the determinant that mediates the receptor uptake of beta-very low density lipoproteins by mouse macrophages. Innerarity TL, Arnold KS, Weisgraber KH, Mahley RW. Arteriosclerosis; 1986 Feb 20; 6(1):114-22. PubMed ID: 3942555 [Abstract] [Full Text] [Related]
20. Resistance of chylomicron and VLDL remnants to post-heparin lipolysis in ApoE-deficient mice: the role of apoE in lipoprotein lipase-mediated lipolysis in vivo and in vitro. Zsigmond E, Fuke Y, Li L, Kobayashi K, Chan L. J Lipid Res; 1998 Sep 20; 39(9):1852-61. PubMed ID: 9741698 [Abstract] [Full Text] [Related] Page: [Next] [New Search]