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452 related items for PubMed ID: 8548533

  • 1. Pattern of cytokines and pharmacomodulation in sepsis induced by cecal ligation and puncture compared with that induced by endotoxin.
    Villa P, Sartor G, Angelini M, Sironi M, Conni M, Gnocchi P, Isetta AM, Grau G, Buurman W, van Tits LJ.
    Clin Diagn Lab Immunol; 1995 Sep; 2(5):549-53. PubMed ID: 8548533
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  • 2. [The effect of some drugs on the levels of selected cytokines in experimental septic shock].
    Ladny JR, Dzienis H, Dadan J, Polaków J, Venskutonis D, Puchalski Z.
    Wiad Lek; 1997 Sep; 50 Suppl 1 Pt 2():247-51. PubMed ID: 9424882
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  • 3. Pulmonary and hepatic gene expression following cecal ligation and puncture: monophosphoryl lipid A prophylaxis attenuates sepsis-induced cytokine and chemokine expression and neutrophil infiltration.
    Salkowski CA, Detore G, Franks A, Falk MC, Vogel SN.
    Infect Immun; 1998 Aug; 66(8):3569-78. PubMed ID: 9673235
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  • 4. Lipopolysaccharide-tumor necrosis factor-glucocorticoid interactions during cecal ligation and puncture-induced sepsis in mature versus senescent mice.
    Hyde SR, McCallum RE.
    Infect Immun; 1992 Mar; 60(3):976-82. PubMed ID: 1541572
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  • 5. Comparison of the mortality and inflammatory response of two models of sepsis: lipopolysaccharide vs. cecal ligation and puncture.
    Remick DG, Newcomb DE, Bolgos GL, Call DR.
    Shock; 2000 Feb; 13(2):110-6. PubMed ID: 10670840
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  • 6. Intrahepatic nuclear factor-kappa B activity and alpha 1-acid glycoprotein transcription do not predict outcome after cecal ligation and puncture in the rat.
    Chen J, Raj N, Kim P, Andrejko KM, Deutschman CS.
    Crit Care Med; 2001 Mar; 29(3):589-96. PubMed ID: 11373425
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  • 8. Heme-oxygenase-1 induction and carbon monoxide-releasing molecule inhibit lipopolysaccharide (LPS)-induced high-mobility group box 1 release in vitro and improve survival of mice in LPS- and cecal ligation and puncture-induced sepsis model in vivo.
    Tsoyi K, Lee TY, Lee YS, Kim HJ, Seo HG, Lee JH, Chang KC.
    Mol Pharmacol; 2009 Jul; 76(1):173-82. PubMed ID: 19366789
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  • 9. Expression and contribution of endogenous IL-13 in an experimental model of sepsis.
    Matsukawa A, Hogaboam CM, Lukacs NW, Lincoln PM, Evanoff HL, Strieter RM, Kunkel SL.
    J Immunol; 2000 Mar 01; 164(5):2738-44. PubMed ID: 10679115
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  • 10. Liu-Shen-Wan, a traditional Chinese medicine, improves survival in sepsis induced by cecal ligation and puncture via reducing TNF-alpha levels, MDA content and enhancing macrophage phagocytosis.
    Ma H, Kou J, Zhu D, Yan Y, Yu B.
    Int Immunopharmacol; 2006 Aug 01; 6(8):1355-62. PubMed ID: 16782549
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  • 12. [Protective effect and mechanism of Ribociclib on sepsis induced-acute kidney injury].
    Wang Q, Gong X, Jia J, Li G.
    Zhonghua Wei Zhong Bing Ji Jiu Yi Xue; 2020 Feb 01; 32(2):204-209. PubMed ID: 32275007
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  • 13. [Protective effects of recombinant trichinella spiralis-53 000 protein combining with imipenem on polymicrobial septic mice].
    Li F, Chen Z, Tang H, Liang Y, Li Z, Wu J, Zeng L, Yang W, Hu X, Ma Z.
    Zhonghua Wei Zhong Bing Ji Jiu Yi Xue; 2016 May 01; 28(5):433-8. PubMed ID: 29920040
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  • 14. Deficiency of alpha-calcitonin gene-related peptide induces inflammatory responses and lethality in sepsis.
    Lee JK, Jung JS, Park SH, Sim YB, Suh HW.
    Cytokine; 2013 Nov 01; 64(2):548-54. PubMed ID: 24021706
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  • 17. Requirement of TNF and TNF receptor type 2 for LPS-induced protection from lethal septic peritonitis.
    Echtenacher B, Männel DN.
    J Endotoxin Res; 2002 Nov 01; 8(5):365-9. PubMed ID: 12537695
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