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162 related items for PubMed ID: 8557661

  • 1. Tumor necrosis factor promotes phosphorylation and binding of insulin receptor substrate 1 to phosphatidylinositol 3-kinase in 3T3-L1 adipocytes.
    Guo D, Donner DB.
    J Biol Chem; 1996 Jan 12; 271(2):615-8. PubMed ID: 8557661
    [Abstract] [Full Text] [Related]

  • 2. Cross-talk between the platelet-derived growth factor and the insulin signaling pathways in 3T3-L1 adipocytes.
    Ricort JM, Tanti JF, Van Obberghen E, Le Marchand-Brustel Y.
    J Biol Chem; 1997 Aug 08; 272(32):19814-8. PubMed ID: 9242642
    [Abstract] [Full Text] [Related]

  • 3. A phosphatidylinositol 3-kinase/Akt/mTOR pathway mediates and PTEN antagonizes tumor necrosis factor inhibition of insulin signaling through insulin receptor substrate-1.
    Ozes ON, Akca H, Mayo LD, Gustin JA, Maehama T, Dixon JE, Donner DB.
    Proc Natl Acad Sci U S A; 2001 Apr 10; 98(8):4640-5. PubMed ID: 11287630
    [Abstract] [Full Text] [Related]

  • 4. Reciprocal feedback regulation of insulin receptor and insulin receptor substrate tyrosine phosphorylation by phosphoinositide 3-kinase in primary adipocytes.
    Hers I, Bell CJ, Poole AW, Jiang D, Denton RM, Schaefer E, Tavaré JM.
    Biochem J; 2002 Dec 15; 368(Pt 3):875-84. PubMed ID: 12220227
    [Abstract] [Full Text] [Related]

  • 5. Insulin regulation of membrane-associated insulin receptor substrate 1.
    Heller-Harrison RA, Morin M, Czech MP.
    J Biol Chem; 1995 Oct 13; 270(41):24442-50. PubMed ID: 7592659
    [Abstract] [Full Text] [Related]

  • 6. Growth hormone induces cellular insulin resistance by uncoupling phosphatidylinositol 3-kinase and its downstream signals in 3T3-L1 adipocytes.
    Takano A, Haruta T, Iwata M, Usui I, Uno T, Kawahara J, Ueno E, Sasaoka T, Kobayashi M.
    Diabetes; 2001 Aug 13; 50(8):1891-900. PubMed ID: 11473053
    [Abstract] [Full Text] [Related]

  • 7. Tumor necrosis factor-alpha-induced insulin resistance in 3T3-L1 adipocytes is accompanied by a loss of insulin receptor substrate-1 and GLUT4 expression without a loss of insulin receptor-mediated signal transduction.
    Stephens JM, Lee J, Pilch PF.
    J Biol Chem; 1997 Jan 10; 272(2):971-6. PubMed ID: 8995390
    [Abstract] [Full Text] [Related]

  • 8. Activation of PI 3-kinase in 3T3-L1 adipocytes by association with insulin receptor substrate-1.
    Lamphere L, Carpenter CL, Sheng ZF, Kallen RG, Lienhard GE.
    Am J Physiol; 1994 Mar 10; 266(3 Pt 1):E486-94. PubMed ID: 7513124
    [Abstract] [Full Text] [Related]

  • 9. Serine/threonine phosphorylation of insulin receptor substrate 1 modulates insulin receptor signaling.
    Tanti JF, Grémeaux T, van Obberghen E, Le Marchand-Brustel Y.
    J Biol Chem; 1994 Feb 25; 269(8):6051-7. PubMed ID: 8119950
    [Abstract] [Full Text] [Related]

  • 10. Chronic tumor necrosis factor-alpha treatment causes insulin resistance via insulin receptor substrate-1 serine phosphorylation and suppressor of cytokine signaling-3 induction in 3T3-L1 adipocytes.
    Ishizuka K, Usui I, Kanatani Y, Bukhari A, He J, Fujisaka S, Yamazaki Y, Suzuki H, Hiratani K, Ishiki M, Iwata M, Urakaze M, Haruta T, Kobayashi M.
    Endocrinology; 2007 Jun 25; 148(6):2994-3003. PubMed ID: 17379643
    [Abstract] [Full Text] [Related]

  • 11. Effects of cell-permeable ceramides and tumor necrosis factor-alpha on insulin signaling and glucose uptake in 3T3-L1 adipocytes.
    Wang CN, O'Brien L, Brindley DN.
    Diabetes; 1998 Jan 25; 47(1):24-31. PubMed ID: 9421370
    [Abstract] [Full Text] [Related]

  • 12. Regulation of insulin receptor, insulin receptor substrate-1 and phosphatidylinositol 3-kinase in 3T3-F442A adipocytes. Effects of differentiation, insulin, and dexamethasone.
    Saad MJ, Folli F, Araki E, Hashimoto N, Csermely P, Kahn CR.
    Mol Endocrinol; 1994 May 25; 8(5):545-57. PubMed ID: 7520127
    [Abstract] [Full Text] [Related]

  • 13. Extracellular matrix induced by TGFbeta impairs insulin signal transduction in 3T3-L1 preadipose cells.
    Gagnon AM, Chabot J, Pardasani D, Sorisky A.
    J Cell Physiol; 1998 Jun 25; 175(3):370-8. PubMed ID: 9572482
    [Abstract] [Full Text] [Related]

  • 14. Gastrin stimulates tyrosine phosphorylation of insulin receptor substrate 1 and its association with Grb2 and the phosphatidylinositol 3-kinase.
    Kowalski-Chauvel A, Pradayrol L, Vaysse N, Seva C.
    J Biol Chem; 1996 Oct 18; 271(42):26356-61. PubMed ID: 8824290
    [Abstract] [Full Text] [Related]

  • 15. The protein-tyrosine kinase fer associates with signaling complexes containing insulin receptor substrate-1 and phosphatidylinositol 3-kinase.
    Iwanishi M, Czech MP, Cherniack AD.
    J Biol Chem; 2000 Dec 15; 275(50):38995-9000. PubMed ID: 11006284
    [Abstract] [Full Text] [Related]

  • 16. Tumor necrosis factor (TNF)-α-induced repression of GKAP42 protein levels through cGMP-dependent kinase (cGK)-Iα causes insulin resistance in 3T3-L1 adipocytes.
    Ando Y, Shinozawa Y, Iijima Y, Yu BC, Sone M, Ooi Y, Watanaka Y, Chida K, Hakuno F, Takahashi S.
    J Biol Chem; 2015 Feb 27; 290(9):5881-92. PubMed ID: 25586176
    [Abstract] [Full Text] [Related]

  • 17. Interleukin-6 (IL-6) induces insulin resistance in 3T3-L1 adipocytes and is, like IL-8 and tumor necrosis factor-alpha, overexpressed in human fat cells from insulin-resistant subjects.
    Rotter V, Nagaev I, Smith U.
    J Biol Chem; 2003 Nov 14; 278(46):45777-84. PubMed ID: 12952969
    [Abstract] [Full Text] [Related]

  • 18. Tumor necrosis factor-alpha acutely inhibits insulin signaling in human adipocytes: implication of the p80 tumor necrosis factor receptor.
    Liu LS, Spelleken M, Röhrig K, Hauner H, Eckel J.
    Diabetes; 1998 Apr 14; 47(4):515-22. PubMed ID: 9568681
    [Abstract] [Full Text] [Related]

  • 19. IRS-1 tyrosine phosphorylation reflects insulin-induced metabolic and mitogenic responses in 3T3-L1 pre-adipocytes.
    Telting D, van der Zon GC, Dorrestijn J, Maassen JA.
    Arch Physiol Biochem; 2001 Feb 14; 109(1):52-62. PubMed ID: 11471071
    [Abstract] [Full Text] [Related]

  • 20. Tumor necrosis factor alpha-induced phosphorylation of insulin receptor substrate-1 (IRS-1). Possible mechanism for suppression of insulin-stimulated tyrosine phosphorylation of IRS-1.
    Kanety H, Feinstein R, Papa MZ, Hemi R, Karasik A.
    J Biol Chem; 1995 Oct 06; 270(40):23780-4. PubMed ID: 7559552
    [Abstract] [Full Text] [Related]


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