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Journal Abstract Search

152 related items for PubMed ID: 8570222

  • 1. Detection and subcellular localization of an AML1 chimeric protein in the t(8;21) positive acute myeloid leukemia.
    Sacchi N, Schiaffonati L, Magnani I, Pappalardo C, Hughes AJ, Darfler M, Hoogeveen AT.
    Oncogene; 1996 Jan 18; 12(2):437-44. PubMed ID: 8570222
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  • 2. The AML1 gene: a transcription factor involved in the pathogenesis of myeloid and lymphoid leukemias.
    Lo Coco F, Pisegna S, Diverio D.
    Haematologica; 1997 Jan 18; 82(3):364-70. PubMed ID: 9234595
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  • 3. Ribozymes cleave the AML1/MTG8 fusion transcript and inhibit proliferation of leukemic cells with t(8;21).
    Matsushita H, Kobayashi H, Mori S, Kizaki M, Ikeda Y.
    Biochem Biophys Res Commun; 1995 Oct 13; 215(2):431-7. PubMed ID: 7487974
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  • 5. Induction of apoptosis in myeloid leukaemic cells by ribozymes targeted against AML1/MTG8.
    Matsushita H, Kizaki M, Kobayashi H, Muto A, Ikeda Y.
    Br J Cancer; 1999 Mar 13; 79(9-10):1325-31. PubMed ID: 10188872
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  • 8. Discrimination of target by siRNA: designing of AML1-MTG8 fusion mRNA-specific siRNA sequences.
    Kasashima K, Sakota E, Kozu T.
    Biochimie; 2004 Mar 13; 86(9-10):713-21. PubMed ID: 15556282
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  • 10. Functional domains of the t(8;21) fusion protein, AML-1/ETO.
    Lenny N, Meyers S, Hiebert SW.
    Oncogene; 1995 Nov 02; 11(9):1761-9. PubMed ID: 7478604
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  • 12. The fusion protein AML1-ETO in acute myeloid leukemia with translocation t(8;21) induces c-jun protein expression via the proximal AP-1 site of the c-jun promoter in an indirect, JNK-dependent manner.
    Elsässer A, Franzen M, Kohlmann A, Weisser M, Schnittger S, Schoch C, Reddy VA, Burel S, Zhang DE, Ueffing M, Tenen DG, Hiddemann W, Behre G.
    Oncogene; 2003 Aug 28; 22(36):5646-57. PubMed ID: 12944913
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  • 13. MYND-less splice variants of AML1-MTG8 (RUNX1-CBFA2T1) are expressed in leukemia with t(8;21).
    Kozu T, Fukuyama T, Yamami T, Akagi K, Kaneko Y.
    Genes Chromosomes Cancer; 2005 May 28; 43(1):45-53. PubMed ID: 15723339
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  • 14. AML1 and Pebp2: the gene for a transcription factor is involved in recurring translocations in human acute myeloid leukemia.
    Rowley JD.
    Jpn J Cancer Res; 1994 Dec 28; 85(12):inside front cover. PubMed ID: 7852178
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  • 16. AML1 and the AML1-ETO fusion protein in the pathogenesis of t(8;21) AML.
    Licht JD.
    Oncogene; 2001 Sep 10; 20(40):5660-79. PubMed ID: 11607817
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  • 17. The t(8;21) fusion protein, AML1 ETO, specifically represses the transcription of the p14(ARF) tumor suppressor in acute myeloid leukemia.
    Linggi B, Müller-Tidow C, van de Locht L, Hu M, Nip J, Serve H, Berdel WE, van der Reijden B, Quelle DE, Rowley JD, Cleveland J, Jansen JH, Pandolfi PP, Hiebert SW.
    Nat Med; 2002 Jul 10; 8(7):743-50. PubMed ID: 12091906
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