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Journal Abstract Search

239 related items for PubMed ID: 8577058

  • 1. [Batten disease (Neuronal ceroid lipofuscinoses)--accumulation of ATP synthase subunit c caused by the delay of lysosomal degradation].
    Ezaki J, Kominami E.
    Nihon Rinsho; 1995 Dec; 53(12):3055-61. PubMed ID: 8577058
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  • 3. Abnormal degradative pathway of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid-lipofuscinosis (Batten disease).
    Ezaki J, Wolfe LS, Ishidoh K, Kominami E.
    Am J Med Genet; 1995 Jun 05; 57(2):254-9. PubMed ID: 7668341
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  • 4. Disease-specific pathology in neurons cultured from sheep affected with ceroid lipofuscinosis.
    Hughes SM, Kay GW, Jordan TW, Rickards GK, Palmer DN.
    Mol Genet Metab; 1999 Apr 05; 66(4):381-6. PubMed ID: 10191133
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  • 7. Specific delay of degradation of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid lipofuscinosis (Batten disease).
    Ezaki J, Wolfe LS, Higuti T, Ishidoh K, Kominami E.
    J Neurochem; 1995 Feb 05; 64(2):733-41. PubMed ID: 7830067
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  • 11. Diagnoses of neuronal ceroid-lipofuscinosis by immunochemical methods.
    Hosain S, Kaufmann WE, Negrin G, Watkins PA, Siakotos AN, Palmer DN, Naidu S.
    Am J Med Genet; 1995 Jun 05; 57(2):239-45. PubMed ID: 7668338
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  • 13. You say lipofuscin, we say ceroid: defining autofluorescent storage material.
    Seehafer SS, Pearce DA.
    Neurobiol Aging; 2006 Apr 05; 27(4):576-88. PubMed ID: 16455164
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  • 20. Decreased lysosomal subunit c-degrading activity in fibroblasts from patients with late infantile neuronal ceroid lipofuscinosis.
    Ezaki J, Wolfe LS, Kominami E.
    Neuropediatrics; 1997 Feb 05; 28(1):53-5. PubMed ID: 9151323
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