These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

179 related items for PubMed ID: 8626855

  • 1. Constitutive activation of the Gs alpha protein-adenylate cyclase pathway may not be sufficient to generate toxic thyroid adenomas.
    Derwahl M, Hamacher C, Russo D, Broecker M, Manole D, Schatz H, Kopp P, Filetti S.
    J Clin Endocrinol Metab; 1996 May; 81(5):1898-904. PubMed ID: 8626855
    [Abstract] [Full Text] [Related]

  • 2. Expression of functional stimulatory guanine nucleotide binding protein in nonfunctioning thyroid adenomas is not correlated to adenylate cyclase activity and growth of these tumors.
    Hamacher C, Studer H, Zbaeren J, Schatz H, Derwahl M.
    J Clin Endocrinol Metab; 1995 May; 80(5):1724-32. PubMed ID: 7745026
    [Abstract] [Full Text] [Related]

  • 3.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 4. Pathogenesis of toxic thyroid adenomas and nodules: relevance of activating mutations in the TSH-receptor and Gs-alpha gene, the possible role of iodine deficiency and secondary and TSH-independent molecular mechanisms.
    Derwahl M, Manole D, Sobke A, Broecker M.
    Exp Clin Endocrinol Diabetes; 1998 May; 106 Suppl 4():S6-9. PubMed ID: 9867188
    [Abstract] [Full Text] [Related]

  • 5.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 6.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 7. The thyrotropin receptor (TSH-R) is not an oncogene for thyroid tumors: structural studies of the TSH-R and the alpha-subunit of Gs in human thyroid neoplasms.
    Matsuo K, Friedman E, Gejman PV, Fagin JA.
    J Clin Endocrinol Metab; 1993 Jun; 76(6):1446-51. PubMed ID: 8501149
    [Abstract] [Full Text] [Related]

  • 8. Diversity and prevalence of somatic mutations in the thyrotropin receptor and Gs alpha genes as a cause of toxic thyroid adenomas.
    Parma J, Duprez L, Van Sande J, Hermans J, Rocmans P, Van Vliet G, Costagliola S, Rodien P, Dumont JE, Vassart G.
    J Clin Endocrinol Metab; 1997 Aug; 82(8):2695-701. PubMed ID: 9253356
    [Abstract] [Full Text] [Related]

  • 9. TSH receptor and Gs-alpha gene mutations in the pathogenesis of toxic thyroid adenomas--a note of caution.
    Derwahl M.
    J Clin Endocrinol Metab; 1996 Aug; 81(8):2783-5. PubMed ID: 8768829
    [Abstract] [Full Text] [Related]

  • 10. [Mutations in the thyrotropin receptor gene in the pathogenesis of toxic thyroid adenomas, toxic goiter nodules and autosomal dominant hyperthyroidism].
    Derwahl M.
    Z Arztl Fortbild Qualitatssich; 1999 Apr; 93 Suppl 1():25-8. PubMed ID: 10355046
    [Abstract] [Full Text] [Related]

  • 11. Structural studies of the thyrotropin receptor and Gs alpha in human thyroid cancers: low prevalence of mutations predicts infrequent involvement in malignant transformation.
    Spambalg D, Sharifi N, Elisei R, Gross JL, Medeiros-Neto G, Fagin JA.
    J Clin Endocrinol Metab; 1996 Nov; 81(11):3898-901. PubMed ID: 8923835
    [Abstract] [Full Text] [Related]

  • 12.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 13. Absence of activating mutations of the genes encoding the alpha-subunits of G11 and Gq in thyroid neoplasia.
    Ringel MD, Saji M, Schwindinger WF, Segev D, Zeiger MA, Levine MA.
    J Clin Endocrinol Metab; 1998 Feb; 83(2):554-9. PubMed ID: 9467574
    [Abstract] [Full Text] [Related]

  • 14.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 15.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 16. Identification of constitutively activating somatic thyrotropin receptor mutations in a subset of toxic multinodular goiters.
    Holzapfel HP, Führer D, Wonerow P, Weinland G, Scherbaum WA, Paschke R.
    J Clin Endocrinol Metab; 1997 Dec; 82(12):4229-33. PubMed ID: 9398745
    [Abstract] [Full Text] [Related]

  • 17. Thyrotropin receptor mutations in hyperfunctioning thyroid adenomas from Brazil.
    Nogueira CR, Kopp P, Arseven OK, Santos CL, Jameson JL, Medeiros-Neto G.
    Thyroid; 1999 Nov; 9(11):1063-8. PubMed ID: 10595453
    [Abstract] [Full Text] [Related]

  • 18. Low incidence of the stimulatory G protein alpha-subunit mutations in autonomously functioning thyroid adenomas in Japan.
    Tanaka K, Nagayama Y, Takeshita A, Namba H, Yamashita S, Niwa M, Nagataki S.
    Thyroid; 1996 Jun; 6(3):195-9. PubMed ID: 8837326
    [Abstract] [Full Text] [Related]

  • 19. Molecular aspects of the pathogenesis of nodular goiters, thyroid nodules and adenomas.
    Derwahl M.
    Exp Clin Endocrinol Diabetes; 1996 Jun; 104 Suppl 4():32-5. PubMed ID: 8980997
    [Abstract] [Full Text] [Related]

  • 20. Somatic mutations causing constitutive activity of the thyrotropin receptor are the major cause of hyperfunctioning thyroid adenomas: identification of additional mutations activating both the cyclic adenosine 3',5'-monophosphate and inositol phosphate-Ca2+ cascades.
    Parma J, Van Sande J, Swillens S, Tonacchera M, Dumont J, Vassart G.
    Mol Endocrinol; 1995 Jun; 9(6):725-33. PubMed ID: 8592518
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 9.