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190 related items for PubMed ID: 8632911

  • 1. Overexpression of the AML1 proto-oncoprotein in NIH3T3 cells leads to neoplastic transformation depending on the DNA-binding and transactivational potencies.
    Kurokawa M, Tanaka T, Tanaka K, Ogawa S, Mitani K, Yazaki Y, Hirai H.
    Oncogene; 1996 Feb 15; 12(4):883-92. PubMed ID: 8632911
    [Abstract] [Full Text] [Related]

  • 2. The t(8;21) fusion protein, AML1/ETO, transforms NIH3T3 cells and activates AP-1.
    Frank RC, Sun X, Berguido FJ, Jakubowiak A, Nimer SD.
    Oncogene; 1999 Mar 04; 18(9):1701-10. PubMed ID: 10208431
    [Abstract] [Full Text] [Related]

  • 3. An acute myeloid leukemia gene, AML1, regulates transcriptional activation and hemopoietic myeloid cell differentiation antagonistically by two alternative spliced forms.
    Tanaka T, Tanaka K, Ogawa S, Kurokawa M, Mitani K, Yazaki Y, Shibata Y, Hirai H.
    Leukemia; 1997 Apr 04; 11 Suppl 3():299-302. PubMed ID: 9209372
    [Abstract] [Full Text] [Related]

  • 4. Rearrangements of the AML1/CBFA2 gene in myeloid leukemia with the 3;21 translocation: in vitro and in vivo studies.
    Zent C, Rowley JD, Nucifora G.
    Leukemia; 1997 Apr 04; 11 Suppl 3():273-8. PubMed ID: 9209363
    [Abstract] [Full Text] [Related]

  • 5. The AML1 gene: a transcription factor involved in the pathogenesis of myeloid and lymphoid leukemias.
    Lo Coco F, Pisegna S, Diverio D.
    Haematologica; 1997 Apr 04; 82(3):364-70. PubMed ID: 9234595
    [Abstract] [Full Text] [Related]

  • 6. The AML1/Evi-1 fusion protein in the t(3;21) translocation exhibits transforming activity on Rat1 fibroblasts with dependence on the Evi-1 sequence.
    Kurokawa M, Ogawa S, Tanaka T, Mitani K, Yazaki Y, Witte ON, Hirai H.
    Oncogene; 1995 Sep 07; 11(5):833-40. PubMed ID: 7675444
    [Abstract] [Full Text] [Related]

  • 7. Leukemogenesis by the chromosomal translocations.
    Mitani K.
    Leukemia; 1997 Apr 07; 11 Suppl 3():294-6. PubMed ID: 9209370
    [Abstract] [Full Text] [Related]

  • 8. Rearrangement of the AML1/CBFA2 gene in myeloid leukemia with the 3;21 translocation: expression of co-existing multiple chimeric genes with similar functions as transcriptional repressors, but with opposite tumorigenic properties.
    Zent C, Kim N, Hiebert S, Zhang DE, Tenen DG, Rowley JD, Nucifora G.
    Curr Top Microbiol Immunol; 1996 Apr 07; 211():243-52. PubMed ID: 8585955
    [Abstract] [Full Text] [Related]

  • 9. The AML1/ETO fusion protein blocks transactivation of the GM-CSF promoter by AML1B.
    Frank R, Zhang J, Uchida H, Meyers S, Hiebert SW, Nimer SD.
    Oncogene; 1995 Dec 21; 11(12):2667-74. PubMed ID: 8545124
    [Abstract] [Full Text] [Related]

  • 10. Functional domains of the t(8;21) fusion protein, AML-1/ETO.
    Lenny N, Meyers S, Hiebert SW.
    Oncogene; 1995 Nov 02; 11(9):1761-9. PubMed ID: 7478604
    [Abstract] [Full Text] [Related]

  • 11. AML-2 is a potential target for transcriptional regulation by the t(8;21) and t(12;21) fusion proteins in acute leukemia.
    Meyers S, Lenny N, Sun W, Hiebert SW.
    Oncogene; 1996 Jul 18; 13(2):303-12. PubMed ID: 8710369
    [Abstract] [Full Text] [Related]

  • 12. Increased expression of AML1 during retinoic-acid-induced differentiation of U937 cells.
    Tanaka K, Tanaka T, Ogawa S, Kurokawa M, Mitani K, Yazaki Y, Hirai H.
    Biochem Biophys Res Commun; 1995 Jun 26; 211(3):1023-30. PubMed ID: 7598688
    [Abstract] [Full Text] [Related]

  • 13. DNA-binding domain of AML1, expressed in t(8;21) and t(3;21) myeloid leukemias, inhibits PEBP2/CBF DNA-binding but is not sufficient to transform 32D cl3 myeloid cells.
    Britos-Bray M, Sacchi N, Friedman AD.
    Leukemia; 1996 Jun 26; 10(6):984-90. PubMed ID: 8667656
    [Abstract] [Full Text] [Related]

  • 14. Molecular mechanism of blastic crisis in chronic myelocytic leukemia.
    Mitani K.
    Leukemia; 1997 Apr 26; 11 Suppl 3():503-5. PubMed ID: 9209439
    [Abstract] [Full Text] [Related]

  • 15. Alternative splicing and genomic structure of the AML1 gene involved in acute myeloid leukemia.
    Miyoshi H, Ohira M, Shimizu K, Mitani K, Hirai H, Imai T, Yokoyama K, Soeda E, Ohki M.
    Nucleic Acids Res; 1995 Jul 25; 23(14):2762-9. PubMed ID: 7651838
    [Abstract] [Full Text] [Related]

  • 16. Molecular cloning and characterization of PEBP2 beta, the heterodimeric partner of a novel Drosophila runt-related DNA binding protein PEBP2 alpha.
    Ogawa E, Inuzuka M, Maruyama M, Satake M, Naito-Fujimoto M, Ito Y, Shigesada K.
    Virology; 1993 May 25; 194(1):314-31. PubMed ID: 8386878
    [Abstract] [Full Text] [Related]

  • 17. The t(3;21) fusion product, AML1/Evi-1 blocks AML1-induced transactivation by recruiting CtBP.
    Izutsu K, Kurokawa M, Imai Y, Ichikawa M, Asai T, Maki K, Mitani K, Hirai H.
    Oncogene; 2002 Apr 18; 21(17):2695-703. PubMed ID: 11965542
    [Abstract] [Full Text] [Related]

  • 18. AML1A and AML1B can transactivate the human IL-3 promoter.
    Uchida H, Zhang J, Nimer SD.
    J Immunol; 1997 Mar 01; 158(5):2251-8. PubMed ID: 9036972
    [Abstract] [Full Text] [Related]

  • 19. AML1 and Pebp2: the gene for a transcription factor is involved in recurring translocations in human acute myeloid leukemia.
    Rowley JD.
    Jpn J Cancer Res; 1994 Dec 01; 85(12):inside front cover. PubMed ID: 7852178
    [No Abstract] [Full Text] [Related]

  • 20. Expression of AML1-d, a short human AML1 isoform, in embryonic stem cells suppresses in vivo tumor growth and differentiation.
    Aziz-Aloya RB, Levanon D, Karn H, Kidron D, Goldenberg D, Lotem J, Polak-Chaklon S, Groner Y.
    Cell Death Differ; 1998 Sep 01; 5(9):765-73. PubMed ID: 10200536
    [Abstract] [Full Text] [Related]

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