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65 related items for PubMed ID: 8649778
1. Synergism between two growth regulatory pathways: cooperative transformation of NIH3T3 cells by G alpha 12 and c-raf-1. Zhang Y, Saez R, Leal MA, Chan AM. Oncogene; 1996 Jun 06; 12(11):2377-83. PubMed ID: 8649778 [Abstract] [Full Text] [Related]
2. Ras-interacting domain of Ral GDP dissociation stimulator like (RGL) reverses v-Ras-induced transformation and Raf-1 activation in NIH3T3 cells. Okazaki M, Kishida S, Murai H, Hinoi T, Kikuchi A. Cancer Res; 1996 May 15; 56(10):2387-92. PubMed ID: 8625316 [Abstract] [Full Text] [Related]
3. Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways. Rusyn EV, Reynolds ER, Shao H, Grana TM, Chan TO, Andres DA, Cox AD. Oncogene; 2000 Sep 28; 19(41):4685-94. PubMed ID: 11032018 [Abstract] [Full Text] [Related]
4. smg/rap1/Krev-1 p21s inhibit the signal pathway to the c-fos promoter/enhancer from c-Ki-ras p21 but not from c-raf-1 kinase in NIH3T3 cells. Sakoda T, Kaibuchi K, Kishi K, Kishida S, Doi K, Hoshino M, Hattori S, Takai Y. Oncogene; 1992 Sep 28; 7(9):1705-11. PubMed ID: 1323817 [Abstract] [Full Text] [Related]
5. G2A is an oncogenic G protein-coupled receptor. Zohn IE, Klinger M, Karp X, Kirk H, Symons M, Chrzanowska-Wodnicka M, Der CJ, Kay RJ. Oncogene; 2000 Aug 10; 19(34):3866-77. PubMed ID: 10951580 [Abstract] [Full Text] [Related]
6. An essential role for Rac in Ras transformation. Qiu RG, Chen J, Kirn D, McCormick F, Symons M. Nature; 1995 Mar 30; 374(6521):457-9. PubMed ID: 7700355 [Abstract] [Full Text] [Related]
7. Rac-1 dependent stimulation of the JNK/SAPK signaling pathway by Vav. Crespo P, Bustelo XR, Aaronson DS, Coso OA, Lopez-Barahona M, Barbacid M, Gutkind JS. Oncogene; 1996 Aug 01; 13(3):455-60. PubMed ID: 8760286 [Abstract] [Full Text] [Related]
8. H-ras and raf-1 cooperate in transformation of NIH3T3 fibroblasts. Cuadrado A, Bruder JT, Heidaran MA, App H, Rapp UR, Aaronson SA. Oncogene; 1993 Sep 01; 8(9):2443-8. PubMed ID: 8361757 [Abstract] [Full Text] [Related]
9. R-Ras is regulated by activators and effectors distinct from those that control Ras function. Huff SY, Quilliam LA, Cox AD, Der CJ. Oncogene; 1997 Jan 16; 14(2):133-43. PubMed ID: 9010215 [Abstract] [Full Text] [Related]
11. Transformation suppressor activity of C3G is independent of its CDC25-homology domain. Guerrero C, Fernandez-Medarde A, Rojas JM, Font de Mora J, Esteban LM, Santos E. Oncogene; 1998 Feb 05; 16(5):613-24. PubMed ID: 9482107 [Abstract] [Full Text] [Related]
14. Cooperative transformation of NIH3T3 cells by G alpha12 and Rac1. Tolkacheva T, Feuer B, Lorenzi MV, Saez R, Chan AM. Oncogene; 1997 Aug 07; 15(6):727-35. PubMed ID: 9264413 [Abstract] [Full Text] [Related]
15. Signal transduction elements of TC21, an oncogenic member of the R-Ras subfamily of GTP-binding proteins. Movilla N, Crespo P, Bustelo XR. Oncogene; 1999 Oct 21; 18(43):5860-9. PubMed ID: 10557073 [Abstract] [Full Text] [Related]
16. Ornithine decarboxylase induction in transformation by H-Ras and RhoA. Shantz LM, Pegg AE. Cancer Res; 1998 Jul 01; 58(13):2748-53. PubMed ID: 9661886 [Abstract] [Full Text] [Related]
17. PKC epsilon functions as an oncogene by enhancing activation of the Raf kinase. Cacace AM, Ueffing M, Philipp A, Han EK, Kolch W, Weinstein IB. Oncogene; 1996 Dec 19; 13(12):2517-26. PubMed ID: 9000126 [Abstract] [Full Text] [Related]
18. Raf and RhoA cooperate to transform intestinal epithelial cells and induce growth resistance to transforming growth factor beta. Du J, Jiang B, Coffey RJ, Barnard J. Mol Cancer Res; 2004 Apr 19; 2(4):233-41. PubMed ID: 15140945 [Abstract] [Full Text] [Related]