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Journal Abstract Search

405 related items for PubMed ID: 8650157

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  • 3. Release of copper ions from the familial amyotrophic lateral sclerosis-associated Cu,Zn-superoxide dismutase mutants.
    Eum WS, Kang JH.
    Mol Cells; 1999 Feb 28; 9(1):110-4. PubMed ID: 10102580
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  • 4. Enhanced oxidative damage by the familial amyotrophic lateral sclerosis-associated Cu,Zn-superoxide dismutase mutants.
    Kang JH, Eum WS.
    Biochim Biophys Acta; 2000 Dec 15; 1524(2-3):162-70. PubMed ID: 11113563
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  • 7. The free radical-generating function of a familial amyotrophic lateral sclerosis-associated D90A Cu,Zn-superoxide dismutase mutant.
    Kim SM, Eum WS, Kwon OB, Kang JH.
    Biochem Mol Biol Int; 1998 Dec 15; 46(6):1191-200. PubMed ID: 9891852
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  • 8. Glycation proceeds faster in mutated Cu, Zn-superoxide dismutases related to familial amyotrophic lateral sclerosis.
    Takamiya R, Takahashi M, Myint T, Park YS, Miyazawa N, Endo T, Fujiwara N, Sakiyama H, Misonou Y, Miyamoto Y, Fujii J, Taniguchi N.
    FASEB J; 2003 May 15; 17(8):938-40. PubMed ID: 12626432
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  • 9. Stabilization of mutant Cu/Zn superoxide dismutase (SOD1) protein by coexpressed wild SOD1 protein accelerates the disease progression in familial amyotrophic lateral sclerosis mice.
    Fukada K, Nagano S, Satoh M, Tohyama C, Nakanishi T, Shimizu A, Yanagihara T, Sakoda S.
    Eur J Neurosci; 2001 Dec 15; 14(12):2032-6. PubMed ID: 11860498
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  • 10. Reexamination of the mechanism of hydroxyl radical adducts formed from the reaction between familial amyotrophic lateral sclerosis-associated Cu,Zn superoxide dismutase mutants and H2O2.
    Singh RJ, Karoui H, Gunther MR, Beckman JS, Mason RP, Kalyanaraman B.
    Proc Natl Acad Sci U S A; 1998 Jun 09; 95(12):6675-80. PubMed ID: 9618471
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  • 11. Characterization of wild-type and amyotrophic lateral sclerosis-related mutant Cu,Zn-superoxide dismutases overproduced in baculovirus-infected insect cells.
    Fujii J, Myint T, Seo HG, Kayanoki Y, Ikeda Y, Taniguchi N.
    J Neurochem; 1995 Apr 09; 64(4):1456-61. PubMed ID: 7891072
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  • 12. Gain in functions of mutant Cu,Zn-superoxide dismutases as a causative factor in familial amyotrophic lateral sclerosis: less reactive oxidant formation but high spontaneous aggregation and precipitation.
    Okado-Matsumoto A, Myint T, Fujii J, Taniguchi N.
    Free Radic Res; 2000 Jul 09; 33(1):65-73. PubMed ID: 10826922
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  • 13. Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.
    Beqollari D, Romberg CF, Dobrowolny G, Martini M, Voss AA, Musarò A, Bannister RA.
    Skelet Muscle; 2016 Jul 09; 6():24. PubMed ID: 27340545
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  • 14. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 09; 7(6 Pt B):623-43. PubMed ID: 11114261
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  • 16. The familial amyotrophic lateral sclerosis-associated amino acid substitutions E100G, G93A, and G93R do not influence the rate of inactivation of copper- and zinc-containing superoxide dismutase by H2O2.
    Liochev SI, Chen LL, Hallewell RA, Fridovich I.
    Arch Biochem Biophys; 1998 Apr 15; 352(2):237-9. PubMed ID: 9587411
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  • 17. Reactions of hydrogen peroxide with familial amyotrophic lateral sclerosis mutant human copper-zinc superoxide dismutases studied by pulse radiolysis.
    Goto JJ, Gralla EB, Valentine JS, Cabelli DE.
    J Biol Chem; 1998 Nov 13; 273(46):30104-9. PubMed ID: 9804764
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  • 18. Protein oxidative damage in a transgenic mouse model of familial amyotrophic lateral sclerosis.
    Andrus PK, Fleck TJ, Gurney ME, Hall ED.
    J Neurochem; 1998 Nov 13; 71(5):2041-8. PubMed ID: 9798929
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