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Journal Abstract Search

458 related items for PubMed ID: 8658529

  • 1. Distribution and behavior of the Ah receptor in murine T lymphocytes.
    Lawrence BP, Leid M, Kerkvliet NI.
    Toxicol Appl Pharmacol; 1996 Jun; 138(2):275-84. PubMed ID: 8658529
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  • 2. Potentiation of CYP1A1 gene expression in MCF-7 human breast cancer cells cotreated with 2,3,7,8-tetrachlorodibenzo-p-dioxin and 12-O-tetradecanoylphorbol-13-acetate.
    Moore M, Narasimhan TR, Steinberg MA, Wang X, Safe S.
    Arch Biochem Biophys; 1993 Sep; 305(2):483-8. PubMed ID: 8396892
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  • 5. Responsiveness of the adult male rat reproductive tract to 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure: Ah receptor and ARNT expression, CYP1A1 induction, and Ah receptor down-regulation.
    Roman BL, Pollenz RS, Peterson RE.
    Toxicol Appl Pharmacol; 1998 Jun; 150(2):228-39. PubMed ID: 9653054
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  • 6. 2,3,7,8-Tetrachlorodibenzo-p-dioxin as growth modulator in mouse hepatocytes with high and low affinity Ah receptor.
    Schrenk D, Schäfer S, Bock KW.
    Carcinogenesis; 1994 Jan; 15(1):27-31. PubMed ID: 8293544
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  • 10. Mechanism of action of aryl hydrocarbon receptor antagonists: inhibition of 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced CYP1A1 gene expression.
    Merchant M, Morrison V, Santostefano M, Safe S.
    Arch Biochem Biophys; 1992 Nov 01; 298(2):389-94. PubMed ID: 1329656
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  • 11. Identification of 3'-methoxy-4'-nitroflavone as a pure aryl hydrocarbon (Ah) receptor antagonist and evidence for more than one form of the nuclear Ah receptor in MCF-7 human breast cancer cells.
    Lu YF, Santostefano M, Cunningham BD, Threadgill MD, Safe S.
    Arch Biochem Biophys; 1995 Jan 10; 316(1):470-7. PubMed ID: 7840652
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  • 12. Physicochemical differences in the AH receptors of the most TCDD-susceptible and the most TCDD-resistant rat strains.
    Pohjanvirta R, Viluksela M, Tuomisto JT, Unkila M, Karasinska J, Franc MA, Holowenko M, Giannone JV, Harper PA, Tuomisto J, Okey AB.
    Toxicol Appl Pharmacol; 1999 Feb 15; 155(1):82-95. PubMed ID: 10036221
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  • 13. Treatment of mice with 2,3,7,8-tetrachlorodibenzo-p-dioxin leads to aryl hydrocarbon receptor-dependent nuclear translocation of NF-kappaB and expression of Fas ligand in thymic stromal cells and consequent apoptosis in T cells.
    Camacho IA, Singh N, Hegde VL, Nagarkatti M, Nagarkatti PS.
    J Immunol; 2005 Jul 01; 175(1):90-103. PubMed ID: 15972635
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  • 14. Expression of functional aromatic hydrocarbon receptor and aromatic hydrocarbon nuclear translocator proteins in murine bone marrow stromal cells.
    Lavin AL, Hahn DJ, Gasiewicz TA.
    Arch Biochem Biophys; 1998 Apr 01; 352(1):9-18. PubMed ID: 9521805
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  • 18. T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4+ and CD8+ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDD.
    Kerkvliet NI, Shepherd DM, Baecher-Steppan L.
    Toxicol Appl Pharmacol; 2002 Dec 01; 185(2):146-52. PubMed ID: 12490139
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  • 19. Methoxychlor suppresses the 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-inducible CYP1A1 expression in murine Hepa-1c1c7 cells.
    Han EH, Jeong TC, Jeong HG.
    J Toxicol Environ Health A; 2007 Aug 01; 70(15-16):1304-9. PubMed ID: 17654248
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  • 20. 2,3,7,8-Tetrachlorodibenzo-p-dioxin affects the number and function of murine splenic dendritic cells and their expression of accessory molecules.
    Vorderstrasse BA, Kerkvliet NI.
    Toxicol Appl Pharmacol; 2001 Mar 01; 171(2):117-25. PubMed ID: 11222087
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