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15. Restricted expression of G86R Cu/Zn superoxide dismutase in astrocytes results in astrocytosis but does not cause motoneuron degeneration. Gong YH, Parsadanian AS, Andreeva A, Snider WD, Elliott JL. J Neurosci; 2000 Jan 15; 20(2):660-5. PubMed ID: 10632595 [Abstract] [Full Text] [Related]
17. Mice lacking cytosolic copper/zinc superoxide dismutase display a distinctive motor axonopathy. Shefner JM, Reaume AG, Flood DG, Scott RW, Kowall NW, Ferrante RJ, Siwek DF, Upton-Rice M, Brown RH. Neurology; 1999 Oct 12; 53(6):1239-46. PubMed ID: 10522879 [Abstract] [Full Text] [Related]
18. Distal axonopathy in an alsin-deficient mouse model. Deng HX, Zhai H, Fu R, Shi Y, Gorrie GH, Yang Y, Liu E, Dal Canto MC, Mugnaini E, Siddique T. Hum Mol Genet; 2007 Dec 01; 16(23):2911-20. PubMed ID: 17855450 [Abstract] [Full Text] [Related]
19. Superoxide dismutase is an abundant component in cell bodies, dendrites, and axons of motor neurons and in a subset of other neurons. Pardo CA, Xu Z, Borchelt DR, Price DL, Sisodia SS, Cleveland DW. Proc Natl Acad Sci U S A; 1995 Feb 14; 92(4):954-8. PubMed ID: 7862672 [Abstract] [Full Text] [Related]
20. Hindlimb motor neurons require Cu/Zn superoxide dismutase for maintenance of neuromuscular junctions. Flood DG, Reaume AG, Gruner JA, Hoffman EK, Hirsch JD, Lin YG, Dorfman KS, Scott RW. Am J Pathol; 1999 Aug 14; 155(2):663-72. PubMed ID: 10433959 [Abstract] [Full Text] [Related] Page: [Next] [New Search]