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Journal Abstract Search

301 related items for PubMed ID: 8721156

  • 1. Time course of striatal changes induced by 6-hydroxydopamine lesion of the nigrostriatal pathway, as studied by combined evaluation of rotational behaviour and striatal Fos expression.
    Labandeira-Garcia JL, Rozas G, Lopez-Martin E, Liste I, Guerra MJ.
    Exp Brain Res; 1996 Feb; 108(1):69-84. PubMed ID: 8721156
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  • 7. Comparison between normal developing striatum and developing striatal grafts using drug-induced Fos expression and neuron-specific enolase immunohistochemistry.
    Labandeira-Garcia JL, Tobio JP, Guerra MJ.
    Neuroscience; 1994 May; 60(2):399-415. PubMed ID: 7915411
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  • 9. Priming of a D1 dopamine receptor behavioural response is dissociated from striatal immediate-early gene activity.
    Paul ML, Currie RW, Robertson HA.
    Neuroscience; 1995 May; 66(2):347-59. PubMed ID: 7477877
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  • 10. Rotational behaviour and neurochemical changes in unilateral N-methyl-norsalsolinol and 6-hydroxydopamine lesioned rats.
    Moser A, Siebecker F, Nobbe F, Böhme V.
    Exp Brain Res; 1996 Nov; 112(1):89-95. PubMed ID: 8951410
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  • 11. Transection of corticostriatal afferents abolishes the hyperexpression of Fos and counteracts the development of rotational overcompensation induced by intrastriatal dopamine-rich grafts when challenged with amphetamine.
    Cenci MA, Björklund A.
    Brain Res; 1994 Nov 28; 665(1):167-74. PubMed ID: 7882012
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  • 12. Apomorphine priming alters the response of striatal outflow pathways to D2 agonist stimulation in 6-hydroxydopamine-lesioned rats.
    Pollack AE, Turgeon SM, Fink JS.
    Neuroscience; 1997 Jul 28; 79(1):79-93. PubMed ID: 9178866
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  • 13. D1-dopamine receptor agonists selectively activate striatal c-fos independent of rotational behaviour.
    Robertson HA, Peterson MR, Murphy K, Robertson GS.
    Brain Res; 1989 Dec 04; 503(2):346-9. PubMed ID: 2532558
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  • 14. Prior D1 dopamine receptor stimulation is required to prime D2-mediated striatal Fos expression in 6-hydroxydopamine-lesioned rats.
    Pollack AE, Yates TM.
    Neuroscience; 1999 Dec 04; 94(2):505-14. PubMed ID: 10579212
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  • 15. Evidence for a role of endogenous opioids in the nigrostriatal system: influence of naloxone and morphine on nigrostriatal dopaminergic supersensitivity.
    Hirschhorn ID, Hittner D, Gardner EL, Cubells J, Makman MH.
    Brain Res; 1983 Jun 27; 270(1):109-17. PubMed ID: 6409355
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  • 16. Dopaminergic grafts implanted into the neonatal or adult striatum: comparative effects on rotation and paw reaching deficits induced by subsequent unilateral nigrostriatal lesions in adulthood.
    Abrous DN, Torres EM, Dunnett SB.
    Neuroscience; 1993 Jun 27; 54(3):657-68. PubMed ID: 8332254
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  • 17. Drug-induced rotation intensity in unilateral dopamine-depleted rats is not correlated with end point or qualitative measures of forelimb or hindlimb motor performance.
    Metz GA, Whishaw IQ.
    Neuroscience; 2002 Jun 27; 111(2):325-36. PubMed ID: 11983318
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