These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


PUBMED FOR HANDHELDS

Journal Abstract Search


566 related items for PubMed ID: 8760286

  • 1. Rac-1 dependent stimulation of the JNK/SAPK signaling pathway by Vav.
    Crespo P, Bustelo XR, Aaronson DS, Coso OA, Lopez-Barahona M, Barbacid M, Gutkind JS.
    Oncogene; 1996 Aug 01; 13(3):455-60. PubMed ID: 8760286
    [Abstract] [Full Text] [Related]

  • 2. Tyrosine phosphorylation of Vav stimulates IL-6 production in mast cells by a Rac/c-Jun N-terminal kinase-dependent pathway.
    Song JS, Haleem-Smith H, Arudchandran R, Gomez J, Scott PM, Mill JF, Tan TH, Rivera J.
    J Immunol; 1999 Jul 15; 163(2):802-10. PubMed ID: 10395673
    [Abstract] [Full Text] [Related]

  • 3. Vav transformation requires activation of multiple GTPases and regulation of gene expression.
    Palmby TR, Abe K, Karnoub AE, Der CJ.
    Mol Cancer Res; 2004 Dec 15; 2(12):702-11. PubMed ID: 15634759
    [Abstract] [Full Text] [Related]

  • 4. Cellular functions of TC10, a Rho family GTPase: regulation of morphology, signal transduction and cell growth.
    Murphy GA, Solski PA, Jillian SA, Pérez de la Ossa P, D'Eustachio P, Der CJ, Rush MG.
    Oncogene; 1999 Jul 01; 18(26):3831-45. PubMed ID: 10445846
    [Abstract] [Full Text] [Related]

  • 5. Vav cooperates with Ras to transform rodent fibroblasts but is not a Ras GDP/GTP exchange factor.
    Bustelo XR, Suen KL, Leftheris K, Meyers CA, Barbacid M.
    Oncogene; 1994 Aug 01; 9(8):2405-13. PubMed ID: 8036025
    [Abstract] [Full Text] [Related]

  • 6. Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways.
    Rusyn EV, Reynolds ER, Shao H, Grana TM, Chan TO, Andres DA, Cox AD.
    Oncogene; 2000 Sep 28; 19(41):4685-94. PubMed ID: 11032018
    [Abstract] [Full Text] [Related]

  • 7. TCR and CD28 are coupled via ZAP-70 to the activation of the Vav/Rac-1-/PAK-1/p38 MAPK signaling pathway.
    Salojin KV, Zhang J, Delovitch TL.
    J Immunol; 1999 Jul 15; 163(2):844-53. PubMed ID: 10395678
    [Abstract] [Full Text] [Related]

  • 8. Rac1 and RhoG promote cell survival by the activation of PI3K and Akt, independently of their ability to stimulate JNK and NF-kappaB.
    Murga C, Zohar M, Teramoto H, Gutkind JS.
    Oncogene; 2002 Jan 10; 21(2):207-16. PubMed ID: 11803464
    [Abstract] [Full Text] [Related]

  • 9. A role for the small GTPase Rac in polyomavirus middle-T antigen-mediated activation of the serum response element and in cell transformation.
    Urich M, Senften M, Shaw PE, Ballmer-Hofer K.
    Oncogene; 1997 Mar 13; 14(10):1235-41. PubMed ID: 9121774
    [Abstract] [Full Text] [Related]

  • 10. Transformation suppressor activity of C3G is independent of its CDC25-homology domain.
    Guerrero C, Fernandez-Medarde A, Rojas JM, Font de Mora J, Esteban LM, Santos E.
    Oncogene; 1998 Feb 05; 16(5):613-24. PubMed ID: 9482107
    [Abstract] [Full Text] [Related]

  • 11. Cooperative transformation of NIH3T3 cells by G alpha12 and Rac1.
    Tolkacheva T, Feuer B, Lorenzi MV, Saez R, Chan AM.
    Oncogene; 1997 Aug 07; 15(6):727-35. PubMed ID: 9264413
    [Abstract] [Full Text] [Related]

  • 12. The activity of the Ets transcription factor PEA3 is regulated by two distinct MAPK cascades.
    O'Hagan RC, Tozer RG, Symons M, McCormick F, Hassell JA.
    Oncogene; 1996 Sep 19; 13(6):1323-33. PubMed ID: 8808707
    [Abstract] [Full Text] [Related]

  • 13. Phosphotyrosine-dependent activation of Rac-1 GDP/GTP exchange by the vav proto-oncogene product.
    Crespo P, Schuebel KE, Ostrom AA, Gutkind JS, Bustelo XR.
    Nature; 1997 Jan 09; 385(6612):169-72. PubMed ID: 8990121
    [Abstract] [Full Text] [Related]

  • 14. Vav: function and regulation in hematopoietic cell signaling.
    Bonnefoy-Bérard N, Munshi A, Yron I, Wu S, Collins TL, Deckert M, Shalom-Barak T, Giampa L, Herbert E, Hernandez J, Meller N, Couture C, Altman A.
    Stem Cells; 1996 May 09; 14(3):250-68. PubMed ID: 8724692
    [Abstract] [Full Text] [Related]

  • 15. Stress-activated protein kinases are negatively regulated by cell density.
    Lallemand D, Ham J, Garbay S, Bakiri L, Traincard F, Jeannequin O, Pfarr CM, Yaniv M.
    EMBO J; 1998 Oct 01; 17(19):5615-26. PubMed ID: 9755162
    [Abstract] [Full Text] [Related]

  • 16. The two guanine nucleotide exchange factor domains of Trio link the Rac1 and the RhoA pathways in vivo.
    Bellanger JM, Lazaro JB, Diriong S, Fernandez A, Lamb N, Debant A.
    Oncogene; 1998 Jan 15; 16(2):147-52. PubMed ID: 9464532
    [Abstract] [Full Text] [Related]

  • 17. The PEA3 Ets transcription factor is a downstream target of the HER2/Neu receptor tyrosine kinase.
    O'Hagan RC, Hassell JA.
    Oncogene; 1998 Jan 22; 16(3):301-10. PubMed ID: 9467955
    [Abstract] [Full Text] [Related]

  • 18. Single point mutations in the SH2 domain impair the transforming potential of vav and fail to activate proto-vav.
    Katzav S.
    Oncogene; 1993 Jul 22; 8(7):1757-63. PubMed ID: 8510922
    [Abstract] [Full Text] [Related]

  • 19. Faciogenital dysplasia protein (FGD1) and Vav, two related proteins required for normal embryonic development, are upstream regulators of Rho GTPases.
    Olson MF, Pasteris NG, Gorski JL, Hall A.
    Curr Biol; 1996 Dec 01; 6(12):1628-33. PubMed ID: 8994827
    [Abstract] [Full Text] [Related]

  • 20. Distinct expression patterns and transforming properties of multiple isoforms of Ost, an exchange factor for RhoA and Cdc42.
    Lorenzi MV, Castagnino P, Chen Q, Hori Y, Miki T.
    Oncogene; 1999 Aug 19; 18(33):4742-55. PubMed ID: 10467422
    [Abstract] [Full Text] [Related]


    Page: [Next] [New Search]
    of 29.