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Journal Abstract Search

243 related items for PubMed ID: 8776736

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  • 3. Intracellular retention of mutant gonadotropin receptors results in loss of hormone binding activity of the follitropin receptor but not of the lutropin/choriogonadotropin receptor.
    Rozzell TG, Wang H, Liu X, Segaloff DL.
    Mol Endocrinol; 1995 Dec; 9(12):1727-36. PubMed ID: 8614409
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  • 4. Hormone-binding properties and glycosylation pattern of a recombinant form of the extracellular domain of the luteinizing hormone/chorionic gonadotropin receptor expressed in mammalian cells.
    Thomas DM, Segaloff DL.
    Endocrinology; 1994 Nov; 135(5):1902-12. PubMed ID: 7956911
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  • 5. The differential binding affinities of the luteinizing hormone (LH)/choriogonadotropin receptor for LH and choriogonadotropin are dictated by different extracellular domain residues.
    Galet C, Ascoli M.
    Mol Endocrinol; 2005 May; 19(5):1263-76. PubMed ID: 15677709
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  • 6. Determination of residues important in hormone binding to the extracellular domain of the luteinizing hormone/chorionic gonadotropin receptor by site-directed mutagenesis and modeling.
    Bhowmick N, Huang J, Puett D, Isaacs NW, Lapthorn AJ.
    Mol Endocrinol; 1996 Sep; 10(9):1147-59. PubMed ID: 8885249
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  • 9. Transfected cells express mostly the intracellular precursor of the lutropin/choriogonadotropin receptor but this precursor binds choriogonadotropin with high affinity.
    Fabritz J, Ryan S, Ascoli M.
    Biochemistry; 1998 Jan 13; 37(2):664-72. PubMed ID: 9425089
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  • 10. Progressive cytoplasmic tail truncations of the lutropin-choriogonadotropin receptor prevent agonist- or phorbol ester-induced phosphorylation, impair agonist- or phorbol ester-induced desensitization, and enhance agonist-induced receptor down-regulation.
    Wang Z, Hipkin RW, Ascoli M.
    Mol Endocrinol; 1996 Jun 13; 10(6):748-59. PubMed ID: 8776735
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  • 11. Phosphorylation of the lutropin/choriogonadotropin receptor facilitates uncoupling of the receptor from adenylyl cyclase and endocytosis of the bound hormone.
    Wang Z, Liu X, Ascoli M.
    Mol Endocrinol; 1997 Feb 13; 11(2):183-92. PubMed ID: 9013765
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  • 12. Two mutations of the lutropin/choriogonadotropin receptor that impair signal transduction also interfere with receptor-mediated endocytosis.
    Dhanwada KR, Vijapurkar U, Ascoli M.
    Mol Endocrinol; 1996 May 13; 10(5):544-54. PubMed ID: 8732685
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  • 14. A dileucine-based motif in the C-terminal tail of the lutropin/choriogonadotropin receptor inhibits endocytosis of the agonist-receptor complex.
    Nakamura K, Ascoli M.
    Mol Pharmacol; 1999 Oct 13; 56(4):728-36. PubMed ID: 10496955
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  • 15. Lutropins appear to contact two independent sites in the extracellular domain of their receptors.
    Bernard MP, Myers RV, Moyle WR.
    Biochem J; 1998 Nov 01; 335 ( Pt 3)(Pt 3):611-7. PubMed ID: 9794802
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  • 16. Mutations that induce constitutive activation and mutations that impair signal transduction modulate the basal and/or agonist-stimulated internalization of the Lutropin/Choriogonadotropin receptor.
    Min KS, Liu X, Fabritz J, Jaquette J, Abell AN, Ascoli M.
    J Biol Chem; 1998 Dec 25; 273(52):34911-9. PubMed ID: 9857020
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  • 18. The regulation of the binding affinity of the luteinizing hormone/choriogonadotropin receptor by sodium ions is mediated by a highly conserved aspartate located in the second transmembrane domain of G protein-coupled receptors.
    Quintana J, Wang H, Ascoli M.
    Mol Endocrinol; 1993 Jun 25; 7(6):767-75. PubMed ID: 8395653
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