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Journal Abstract Search

137 related items for PubMed ID: 8781423

  • 1. Human platelet signaling defect characterized by impaired production of inositol-1,4,5-triphosphate and phosphatidic acid and diminished Pleckstrin phosphorylation: evidence for defective phospholipase C activation.
    Yang X, Sun L, Ghosh S, Rao AK.
    Blood; 1996 Sep 01; 88(5):1676-83. PubMed ID: 8781423
    [Abstract] [Full Text] [Related]

  • 2. Platelet activation with combination of ionophore A23187 and a direct protein kinase C activator induces normal secretion in patients with impaired receptor mediated secretion and abnormal signal transduction.
    Yang X, Sun L, Gabbeta J, Rao AK.
    Thromb Res; 1997 Nov 01; 88(3):317-28. PubMed ID: 9526952
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  • 3. Pathogenetic analysis of three cases with a bleeding disorder characterized by defective platelet aggregation induced by Ca2+ ionophores.
    Fuse I, Higuchi W, Uesugi Y, Aizawa Y.
    Br J Haematol; 2001 Mar 01; 112(3):603-8. PubMed ID: 11260060
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  • 4. Abnormal inside-out signal transduction-dependent activation of glycoprotein IIb-IIIa in a patient with impaired pleckstrin phosphorylation.
    Gabbeta J, Yang X, Sun L, McLane MA, Niewiarowski S, Rao AK.
    Blood; 1996 Feb 15; 87(4):1368-76. PubMed ID: 8608226
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  • 5. Interaction of nitric oxide and cGMP with signal transduction in activated platelets.
    Nguyen BL, Saitoh M, Ware JA.
    Am J Physiol; 1991 Oct 15; 261(4 Pt 2):H1043-52. PubMed ID: 1656783
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  • 6. Activation of phospholipase C and protein kinase C has little involvement in ADP-induced primary aggregation of human platelets: effects of diacylglycerols, the diacylglycerols, the diacylglycerol kinase inhibitor R59022, staurosporine and okadaic acid.
    Packham MA, Livne AA, Ruben DH, Rand ML.
    Biochem J; 1993 Mar 15; 290 ( Pt 3)(Pt 3):849-56. PubMed ID: 8384448
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  • 7. Protein kinase C-dependent and Ca2+-dependent mechanisms of secretion from streptolysin O-permeabilized platelets: effects of leakage of cytosolic proteins.
    Sloan DC, Haslam RJ.
    Biochem J; 1997 Nov 15; 328 ( Pt 1)(Pt 1):13-21. PubMed ID: 9359828
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  • 8. Factors affecting dense and alpha-granule secretion from electropermeabilized human platelets: Ca(2+)-independent actions of phorbol ester and GTP gamma S.
    Coorssen JR, Davidson MM, Haslam RJ.
    Cell Regul; 1990 Dec 15; 1(13):1027-41. PubMed ID: 1966891
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  • 12. Ca2+ mobilization primes protein kinase C in human platelets. Ca2+ and phorbol esters stimulate platelet aggregation and secretion synergistically through protein kinase C.
    Siess W, Lapetina EG.
    Biochem J; 1988 Oct 01; 255(1):309-18. PubMed ID: 3143357
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  • 13. Tyrosine dephosphorylation, but not phosphorylation, of p130Cas is dependent on integrin alpha IIb beta 3-mediated aggregation in platelets: implication of p130Cas involvement in pathways unrelated to cytoskeletal reorganization.
    Ohmori T, Yatomi Y, Inoue K, Satoh K, Ozaki Y.
    Biochemistry; 2000 May 16; 39(19):5797-807. PubMed ID: 10801330
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  • 15. Synthesis of intracellular histamine in platelets is associated with activation of protein kinase C, but not with mobilization of Ca2+.
    Saxena SP, Robertson C, Becker AB, Gerrard JM.
    Biochem J; 1991 Jan 15; 273(Pt 2)(Pt 2):405-8. PubMed ID: 1899334
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  • 16. Action of guanosine 5'-[beta-thio]diphosphate on thrombin-induced activation and Ca2+ mobilization in saponin-permeabilized and intact human platelets.
    Authi KS, Rao GH, Evenden BJ, Crawford N.
    Biochem J; 1988 Nov 01; 255(3):885-93. PubMed ID: 3063257
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  • 17. Involvement of Na+/Ca2+ exchanger in inside-out signaling through the platelet integrin IIbbeta3.
    Shiraga M, Tomiyama Y, Honda S, Suzuki H, Kosugi S, Tadokoro S, Kanakura Y, Tanoue K, Kurata Y, Matsuzawa Y.
    Blood; 1998 Nov 15; 92(10):3710-20. PubMed ID: 9808565
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  • 18. Ethanol inhibits thrombin-induced secretion by human platelets at a site distinct from phospholipase C or protein kinase C.
    Benistant C, Rubin R.
    Biochem J; 1990 Jul 15; 269(2):489-97. PubMed ID: 2117442
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  • 20. HDL3-mediated inhibition of thrombin-induced platelet aggregation and fibrinogen binding occurs via decreased production of phosphoinositide-derived second messengers 1,2-diacylglycerol and inositol 1,4,5-tris-phosphate.
    Nofer JR, Walter M, Kehrel B, Wierwille S, Tepel M, Seedorf U, Assmann G.
    Arterioscler Thromb Vasc Biol; 1998 Jun 15; 18(6):861-9. PubMed ID: 9633924
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