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Journal Abstract Search

294 related items for PubMed ID: 8786300

  • 1. H-2Dp transgene alters natural killer cell specificity at the target and effector cell levels. Comparison with an H-2Dd transgene.
    Sentman CL, Olsson-Alheim MY, Lendahl U, Kärre K.
    J Immunol; 1996 Apr 01; 156(7):2423-9. PubMed ID: 8786300
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  • 4. H-2 allele-specific protection from NK cell lysis in vitro for lymphoblasts but not tumor targets. Protection mediated by alpha 1/alpha 2 domains.
    Sentman CL, Olsson MY, Salcedo M, Höglund P, Lendahl U, Käre K.
    J Immunol; 1994 Dec 15; 153(12):5482-90. PubMed ID: 7989750
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  • 5. NK cell receptor calibration: effects of MHC class I induction on killing by Ly49Ahigh and Ly49Alow NK cells.
    Olsson-Alheim MY, Salcedo M, Ljunggren HG, Kärre K, Sentman CL.
    J Immunol; 1997 Oct 01; 159(7):3189-94. PubMed ID: 9317116
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  • 6. Role of major histocompatibility complex class I alpha 1/alpha 2 domain polymorphism and in vivo expression pattern in tumor resistance: studies with transgenic mice and lymphoma cell transfectants.
    Höglund P, Waldenström M, Kärre K.
    J Immunother Emphasis Tumor Immunol; 1993 Oct 01; 14(3):175-81. PubMed ID: 8297899
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  • 7. Inhibition of natural killer cell-mediated bone marrow graft rejection by allogeneic major histocompatibility complex class I, but not class II molecules.
    Ohlén C, Höglund P, Sentman CL, Carbone E, Ljunggren HG, Koller B, Kärre K.
    Eur J Immunol; 1995 May 01; 25(5):1286-91. PubMed ID: 7774631
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  • 8. MHC class I alloantigen specificity of Ly-49+ IL-2-activated natural killer cells.
    Karlhofer FM, Ribaudo RK, Yokoyama WM.
    Nature; 1992 Jul 02; 358(6381):66-70. PubMed ID: 1614533
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  • 13. Rejection of tumors in mice with severe combined immunodeficiency syndrome determined by the major histocompatibility complex. Class I expression on the graft.
    Glas R, Waldenström M, Höglund P, Klein G, Kärre K, Ljunggren HG.
    Cancer Res; 1995 May 01; 55(9):1911-6. PubMed ID: 7728758
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  • 14. Altered expression of Ly49 inhibitory receptors on natural killer cells from MHC class I-deficient mice.
    Salcedo M, Diehl AD, Olsson-Alheim MY, Sundbäck J, Van Kaer L, Kärre K, Ljunggren HG.
    J Immunol; 1997 Apr 01; 158(7):3174-80. PubMed ID: 9120271
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  • 15. Positive recognition of MHC class I molecules by the Ly49D receptor of murine NK cells.
    George TC, Mason LH, Ortaldo JR, Kumar V, Bennett M.
    J Immunol; 1999 Feb 15; 162(4):2035-43. PubMed ID: 9973475
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  • 16. The ability of H-2Dd molecule to affect natural resistance to hemopoietic allografts is an intrinsic property shared by Ddm1 but not Ld.
    Milisauskas VK, Nakamura I.
    Eur J Immunol; 1994 Feb 15; 24(2):336-42. PubMed ID: 8299682
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  • 17. Differential effects of the rejection of bone marrow allografts by the depletion of activating versus inhibiting Ly-49 natural killer cell subsets.
    Raziuddin A, Longo DL, Mason L, Ortaldo JR, Bennett M, Murphy WJ.
    J Immunol; 1998 Jan 01; 160(1):87-94. PubMed ID: 9551959
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  • 18. The alpha2 domain of H-2Dd restricts the allelic specificity of the murine NK cell inhibitory receptor Ly-49A.
    Sundbäck J, Nakamura MC, Waldenström M, Niemi EC, Seaman WE, Ryan JC, Kärre K.
    J Immunol; 1998 Jun 15; 160(12):5971-8. PubMed ID: 9637511
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  • 19. H-2 antigen expression and sensitivity of BL6 melanoma cells to natural killer cell cytotoxicity.
    Gorelik E, Gunji Y, Herberman RB.
    J Immunol; 1988 Mar 15; 140(6):2096-102. PubMed ID: 3126240
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  • 20. Evidence for a role for T cell receptors (TCR) in the effector phase of acute bone marrow graft rejection. TCR V beta 5 transgenic mice lack effector cells able to cause graft rejection.
    Kikly K, Dennert G.
    J Immunol; 1992 Dec 01; 149(11):3489-94. PubMed ID: 1431119
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