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Journal Abstract Search

119 related items for PubMed ID: 8793292

  • 1. Disease mechanism: unravelling Wiskott-Aldrich syndrome.
    Kirchhausen T, Rosen FS.
    Curr Biol; 1996 Jun 01; 6(6):676-8. PubMed ID: 8793292
    [Abstract] [Full Text] [Related]

  • 2. Wiskott-Aldrich syndrome protein induces actin clustering without direct binding to Cdc42.
    Kato M, Miki H, Imai K, Nonoyama S, Suzuki T, Sasakawa C, Takenawa T.
    J Biol Chem; 1999 Sep 17; 274(38):27225-30. PubMed ID: 10480940
    [Abstract] [Full Text] [Related]

  • 3. The Cdc42/Rac interactive binding region motif of the Wiskott Aldrich syndrome protein (WASP) is necessary but not sufficient for tight binding to Cdc42 and structure formation.
    Rudolph MG, Bayer P, Abo A, Kuhlmann J, Vetter IR, Wittinghofer A.
    J Biol Chem; 1998 Jul 17; 273(29):18067-76. PubMed ID: 9660763
    [Abstract] [Full Text] [Related]

  • 4. Two GTPases, Cdc42 and Rac, bind directly to a protein implicated in the immunodeficiency disorder Wiskott-Aldrich syndrome.
    Aspenström P, Lindberg U, Hall A.
    Curr Biol; 1996 Jan 01; 6(1):70-5. PubMed ID: 8805223
    [Abstract] [Full Text] [Related]

  • 5. Structure of Cdc42 in complex with the GTPase-binding domain of the 'Wiskott-Aldrich syndrome' protein.
    Abdul-Manan N, Aghazadeh B, Liu GA, Majumdar A, Ouerfelli O, Siminovitch KA, Rosen MK.
    Nature; 1999 May 27; 399(6734):379-83. PubMed ID: 10360578
    [Abstract] [Full Text] [Related]

  • 6. SLP-76 coordinates Nck-dependent Wiskott-Aldrich syndrome protein recruitment with Vav-1/Cdc42-dependent Wiskott-Aldrich syndrome protein activation at the T cell-APC contact site.
    Zeng R, Cannon JL, Abraham RT, Way M, Billadeau DD, Bubeck-Wardenberg J, Burkhardt JK.
    J Immunol; 2003 Aug 01; 171(3):1360-8. PubMed ID: 12874226
    [Abstract] [Full Text] [Related]

  • 7. The many faces of WAS protein.
    Featherstone C.
    Science; 1997 Jan 03; 275(5296):27-8. PubMed ID: 8999530
    [No Abstract] [Full Text] [Related]

  • 8. Wiskott-Aldrich syndrome protein, a novel effector for the GTPase CDC42Hs, is implicated in actin polymerization.
    Symons M, Derry JM, Karlak B, Jiang S, Lemahieu V, Mccormick F, Francke U, Abo A.
    Cell; 1996 Mar 08; 84(5):723-34. PubMed ID: 8625410
    [Abstract] [Full Text] [Related]

  • 9. Waltzing with WASP.
    Ramesh N, Antón IM, Martínez-Quiles N, Geha RS.
    Trends Cell Biol; 1999 Jan 08; 9(1):15-9. PubMed ID: 10087612
    [Abstract] [Full Text] [Related]

  • 10. Direct interaction of the Wiskott-Aldrich syndrome protein with the GTPase Cdc42.
    Kolluri R, Tolias KF, Carpenter CL, Rosen FS, Kirchhausen T.
    Proc Natl Acad Sci U S A; 1996 May 28; 93(11):5615-8. PubMed ID: 8643625
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  • 15. The interaction between Cdc42 and WASP is required for SDF-1-induced T-lymphocyte chemotaxis.
    Haddad E, Zugaza JL, Louache F, Debili N, Crouin C, Schwarz K, Fischer A, Vainchenker W, Bertoglio J.
    Blood; 2001 Jan 01; 97(1):33-8. PubMed ID: 11133739
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  • 18. Global disruption of the WASP autoinhibited structure on Cdc42 binding. Ligand displacement as a novel method for monitoring amide hydrogen exchange.
    Buck M, Xu W, Rosen MK.
    Biochemistry; 2001 Nov 27; 40(47):14115-22. PubMed ID: 11714264
    [Abstract] [Full Text] [Related]

  • 19. WASP (Wiskott-Aldrich syndrome protein) gene mutations and phenotype.
    Imai K, Nonoyama S, Ochs HD.
    Curr Opin Allergy Clin Immunol; 2003 Dec 27; 3(6):427-36. PubMed ID: 14612666
    [Abstract] [Full Text] [Related]

  • 20. How does one gene cause Wiskott-Aldrich syndrome?
    Featherstone C.
    Lancet; 1996 Oct 05; 348(9032):950. PubMed ID: 8926834
    [No Abstract] [Full Text] [Related]

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