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127 related items for PubMed ID: 8798665

  • 21. The E1B 19K protein associates with lamins in vivo and its proper localization is required for inhibition of apoptosis.
    Rao L, Modha D, White E.
    Oncogene; 1997 Sep 25; 15(13):1587-97. PubMed ID: 9380411
    [Abstract] [Full Text] [Related]

  • 22. Bcl-xL and E1B-19K proteins inhibit p53-induced irreversible growth arrest and senescence by preventing reactive oxygen species-dependent p38 activation.
    Jung MS, Jin DH, Chae HD, Kang S, Kim SC, Bang YJ, Choi TS, Choi KS, Shin DY.
    J Biol Chem; 2004 Apr 23; 279(17):17765-71. PubMed ID: 14764594
    [Abstract] [Full Text] [Related]

  • 23. Survival activity of Bcl-2 homologs Bcl-w and A1 only partially correlates with their ability to bind pro-apoptotic family members.
    Holmgreen SP, Huang DC, Adams JM, Cory S.
    Cell Death Differ; 1999 Jun 23; 6(6):525-32. PubMed ID: 10381646
    [Abstract] [Full Text] [Related]

  • 24. Cloning of a bcl-2 homologue by interaction with adenovirus E1B 19K.
    Farrow SN, White JH, Martinou I, Raven T, Pun KT, Grinham CJ, Martinou JC, Brown R.
    Nature; 1995 Apr 20; 374(6524):731-3. PubMed ID: 7715729
    [Abstract] [Full Text] [Related]

  • 25. Proapoptotic protein Bax heterodimerizes with Bcl-2 and homodimerizes with Bax via a novel domain (BH3) distinct from BH1 and BH2.
    Zha H, Aimé-Sempé C, Sato T, Reed JC.
    J Biol Chem; 1996 Mar 29; 271(13):7440-4. PubMed ID: 8631771
    [Abstract] [Full Text] [Related]

  • 26. Bik, a novel death-inducing protein shares a distinct sequence motif with Bcl-2 family proteins and interacts with viral and cellular survival-promoting proteins.
    Boyd JM, Gallo GJ, Elangovan B, Houghton AB, Malstrom S, Avery BJ, Ebb RG, Subramanian T, Chittenden T, Lutz RJ.
    Oncogene; 1995 Nov 02; 11(9):1921-8. PubMed ID: 7478623
    [Abstract] [Full Text] [Related]

  • 27. Distinct domains of Bcl-XL are involved in Bax and Bad antagonism and in apoptosis inhibition.
    Zhou H, Hou Q, Chai Y, Hsu YT.
    Exp Cell Res; 2005 Oct 01; 309(2):316-28. PubMed ID: 16061221
    [Abstract] [Full Text] [Related]

  • 28. Role of membrane anchor domain of Bcl-2 in suppression of apoptosis caused by E1B-defective adenovirus.
    Nguyen M, Branton PE, Walton PA, Oltvai ZN, Korsmeyer SJ, Shore GC.
    J Biol Chem; 1994 Jun 17; 269(24):16521-4. PubMed ID: 8206964
    [Abstract] [Full Text] [Related]

  • 29. Bax can antagonize Bcl-XL during etoposide and cisplatin-induced cell death independently of its heterodimerization with Bcl-XL.
    Simonian PL, Grillot DA, Merino R, Nuñez G.
    J Biol Chem; 1996 Sep 13; 271(37):22764-72. PubMed ID: 8798452
    [Abstract] [Full Text] [Related]

  • 30. Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death.
    Yang E, Zha J, Jockel J, Boise LH, Thompson CB, Korsmeyer SJ.
    Cell; 1995 Jan 27; 80(2):285-91. PubMed ID: 7834748
    [Abstract] [Full Text] [Related]

  • 31. MAP-1, a novel proapoptotic protein containing a BH3-like motif that associates with Bax through its Bcl-2 homology domains.
    Tan KO, Tan KM, Chan SL, Yee KS, Bevort M, Ang KC, Yu VC.
    J Biol Chem; 2001 Jan 26; 276(4):2802-7. PubMed ID: 11060313
    [Abstract] [Full Text] [Related]

  • 32. Bcl-2/E1B 19 kDa-interacting protein 3-like protein (Bnip3L) interacts with bcl-2/Bcl-xL and induces apoptosis by altering mitochondrial membrane permeability.
    Imazu T, Shimizu S, Tagami S, Matsushima M, Nakamura Y, Miki T, Okuyama A, Tsujimoto Y.
    Oncogene; 1999 Aug 12; 18(32):4523-9. PubMed ID: 10467396
    [Abstract] [Full Text] [Related]

  • 33. The E1B 19K/Bcl-2-binding protein Nip3 is a dimeric mitochondrial protein that activates apoptosis.
    Chen G, Ray R, Dubik D, Shi L, Cizeau J, Bleackley RC, Saxena S, Gietz RD, Greenberg AH.
    J Exp Med; 1997 Dec 15; 186(12):1975-83. PubMed ID: 9396766
    [Abstract] [Full Text] [Related]

  • 34. Modulation of p53-mediated transcriptional repression and apoptosis by the adenovirus E1B 19K protein.
    Sabbatini P, Chiou SK, Rao L, White E.
    Mol Cell Biol; 1995 Feb 15; 15(2):1060-70. PubMed ID: 7823921
    [Abstract] [Full Text] [Related]

  • 35. Structure-function analysis of Bcl-2 family proteins. Regulators of programmed cell death.
    Reed JC, Zha H, Aime-Sempe C, Takayama S, Wang HG.
    Adv Exp Med Biol; 1996 Feb 15; 406():99-112. PubMed ID: 8910675
    [Abstract] [Full Text] [Related]

  • 36. Characterization of the anti-apoptotic mechanism of Bcl-B.
    Zhai D, Ke N, Zhang H, Ladror U, Joseph M, Eichinger A, Godzik A, Ng SC, Reed JC.
    Biochem J; 2003 Nov 15; 376(Pt 1):229-36. PubMed ID: 12921534
    [Abstract] [Full Text] [Related]

  • 37. Bad is a BH3 domain-containing protein that forms an inactivating dimer with Bcl-XL.
    Kelekar A, Chang BS, Harlan JE, Fesik SW, Thompson CB.
    Mol Cell Biol; 1997 Dec 15; 17(12):7040-6. PubMed ID: 9372935
    [Abstract] [Full Text] [Related]

  • 38. Functional identification of the apoptosis effector BH3 domain in cellular protein BNIP1.
    Yasuda M, Chinnadurai G.
    Oncogene; 2000 May 04; 19(19):2363-7. PubMed ID: 10822388
    [Abstract] [Full Text] [Related]

  • 39. Multiple Bcl-2 family members demonstrate selective dimerizations with Bax.
    Sedlak TW, Oltvai ZN, Yang E, Wang K, Boise LH, Thompson CB, Korsmeyer SJ.
    Proc Natl Acad Sci U S A; 1995 Aug 15; 92(17):7834-8. PubMed ID: 7644501
    [Abstract] [Full Text] [Related]

  • 40. A splicing variant of the Bcl-2 member Bok with a truncated BH3 domain induces apoptosis but does not dimerize with antiapoptotic Bcl-2 proteins in vitro.
    Hsu SY, Hsueh AJ.
    J Biol Chem; 1998 Nov 13; 273(46):30139-46. PubMed ID: 9804769
    [Abstract] [Full Text] [Related]

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