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606 related items for PubMed ID: 8806883

  • 1. Aryl-hydrocarbon receptor-deficient mice are resistant to 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced toxicity.
    Fernandez-Salguero PM, Hilbert DM, Rudikoff S, Ward JM, Gonzalez FJ.
    Toxicol Appl Pharmacol; 1996 Sep; 140(1):173-9. PubMed ID: 8806883
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  • 2. Female Sprague-Dawley rats exposed to a single oral dose of 2,3,7,8-tetrachlorodibenzo-p-dioxin exhibit sustained depletion of aryl hydrocarbon receptor protein in liver, spleen, thymus, and lung.
    Pollenz RS, Santostefano MJ, Klett E, Richardson VM, Necela B, Birnbaum LS.
    Toxicol Sci; 1998 Apr; 42(2):117-28. PubMed ID: 9579024
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  • 3. A chimeric aryl hydrocarbon receptor knockout mouse model indicates that aryl hydrocarbon receptor activation in hematopoietic cells contributes to the hepatic lesions induced by 2,3,7, 8-tetrachlorodibenzo-p-dioxin.
    Thurmond TS, Silverstone AE, Baggs RB, Quimby FW, Staples JE, Gasiewicz TA.
    Toxicol Appl Pharmacol; 1999 Jul 01; 158(1):33-40. PubMed ID: 10387930
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  • 7. Knockout of the aryl hydrocarbon receptor results in distinct hepatic and renal phenotypes in rats and mice.
    Harrill JA, Hukkanen RR, Lawson M, Martin G, Gilger B, Soldatow V, Lecluyse EL, Budinsky RA, Rowlands JC, Thomas RS.
    Toxicol Appl Pharmacol; 2013 Oct 15; 272(2):503-18. PubMed ID: 23859880
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  • 8. Thymic alterations induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin are strictly dependent on aryl hydrocarbon receptor activation in hemopoietic cells.
    Staples JE, Murante FG, Fiore NC, Gasiewicz TA, Silverstone AE.
    J Immunol; 1998 Apr 15; 160(8):3844-54. PubMed ID: 9558089
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  • 9. Role of aryl hydrocarbon receptor (AHR) in overall retinoid metabolism: Response comparisons to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure between wild-type and AHR knockout mice.
    Esteban J, Sánchez-Pérez I, Hamscher G, Miettinen HM, Korkalainen M, Viluksela M, Pohjanvirta R, Håkansson H.
    Reprod Toxicol; 2021 Apr 15; 101():33-49. PubMed ID: 33607186
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  • 11. Dioxin-induced adseverin expression in the mouse thymus is strictly regulated and dependent on the aryl hydrocarbon receptor.
    Svensson C, Silverstone AE, Lai ZW, Lundberg K.
    Biochem Biophys Res Commun; 2002 Mar 15; 291(5):1194-200. PubMed ID: 11883943
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  • 14. Gene expression profile by 2,3,7,8-tetrachlorodibenzo-p-dioxin in the liver of wild-type (AhR+/+) and aryl hydrocarbon receptor-deficient (AhR-/-) mice.
    Yoon CY, Park M, Kim BH, Park JY, Park MS, Jeong YK, Kwon H, Jung HK, Kang H, Lee YS, Lee BJ.
    J Vet Med Sci; 2006 Jul 15; 68(7):663-8. PubMed ID: 16891777
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  • 17. Cell proliferation arrest within intrathymic lymphocyte progenitor cells causes thymic atrophy mediated by the aryl hydrocarbon receptor.
    Laiosa MD, Wyman A, Murante FG, Fiore NC, Staples JE, Gasiewicz TA, Silverstone AE.
    J Immunol; 2003 Nov 01; 171(9):4582-91. PubMed ID: 14568932
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  • 18. Aryl hydrocarbon receptor-deficient mice generate normal immune responses to model antigens and are resistant to TCDD-induced immune suppression.
    Vorderstrasse BA, Steppan LB, Silverstone AE, Kerkvliet NI.
    Toxicol Appl Pharmacol; 2001 Mar 15; 171(3):157-64. PubMed ID: 11243915
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  • 19. Aryl hydrocarbon receptor activation in genital tubercle, palate, and other embryonic tissues in 2,3,7, 8-tetrachlorodibenzo-p-dioxin-responsive lacZ mice.
    Willey JJ, Stripp BR, Baggs RB, Gasiewicz TA.
    Toxicol Appl Pharmacol; 1998 Jul 15; 151(1):33-44. PubMed ID: 9705885
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  • 20. T cell-specific disruption of arylhydrocarbon receptor nuclear translocator (Arnt) gene causes resistance to 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced thymic involution.
    Tomita S, Jiang HB, Ueno T, Takagi S, Tohi K, Maekawa S, Miyatake A, Furukawa A, Gonzalez FJ, Takeda J, Ichikawa Y, Takahama Y.
    J Immunol; 2003 Oct 15; 171(8):4113-20. PubMed ID: 14530333
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