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Journal Abstract Search
180 related items for PubMed ID: 8853333
1. NO synthase inhibition modulates NMDA-induced changes in cerebral blood flow and EEG activity. Pelligrino DA, Gay RL, Baughman VL, Wang Q. Am J Physiol; 1996 Sep; 271(3 Pt 2):H990-5. PubMed ID: 8853333 [Abstract] [Full Text] [Related]
5. Permissive and obligatory roles of NO in cerebrovascular responses to hypercapnia and acetylcholine. Iadecola C, Zhang F. Am J Physiol; 1996 Oct; 271(4 Pt 2):R990-1001. PubMed ID: 8897992 [Abstract] [Full Text] [Related]
6. Inhibition of nitric oxide synthase induces a selective reduction in tumor blood flow that is reversible with L-arginine. Tozer GM, Prise VE, Chaplin DJ. Cancer Res; 1997 Mar 01; 57(5):948-55. PubMed ID: 9041200 [Abstract] [Full Text] [Related]
12. Nitric oxide (NO) is an endogenous anticonvulsant but not a mediator of the increase in cerebral blood flow accompanying bicuculline-induced seizures in rats. Wang Q, Theard MA, Pelligrino DA, Baughman VL, Hoffman WE, Albrecht RF, Cwik M, Paulson OB, Lassen NA. Brain Res; 1994 Sep 26; 658(1-2):192-8. PubMed ID: 7530579 [Abstract] [Full Text] [Related]
14. Nitric oxide synthase inhibition attenuates delayed vasodilation and increases injury after cerebral ischemia in fetal sheep. Marks KA, Mallard CE, Roberts I, Williams CE, Gluckman PD, Edwards AD. Pediatr Res; 1996 Aug 26; 40(2):185-91. PubMed ID: 8827765 [Abstract] [Full Text] [Related]