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180 related items for PubMed ID: 8890955

  • 1. Influence of the glucocorticoid receptor on c-fos inducibility in activated ras-containing mouse lung cells.
    Johnson TA, Li J, Beer DG.
    Mol Carcinog; 1996 Oct; 17(2):70-7. PubMed ID: 8890955
    [Abstract] [Full Text] [Related]

  • 2. Loss of glucocorticoid-dependent growth inhibition in transformed mouse lung cells.
    Li J, Johnson TA, Hanson LA, Beer DG.
    Mol Carcinog; 1996 Aug; 16(4):213-20. PubMed ID: 8784464
    [Abstract] [Full Text] [Related]

  • 3. Occupancy and composition of proteins bound to the AP-1 sites in the glucocorticoid receptor and c-jun promoters after glucocorticoid treatment and in different cell types.
    Barrett TJ, Vedeckis WV.
    Recept Signal Transduct; 1996 Aug; 6(3-4):179-93. PubMed ID: 9259052
    [Abstract] [Full Text] [Related]

  • 4. Regulation of epidermal growth factor receptor by activated H-ras and V-myc oncogenes in mouse Balb/3T3 cells: possible roles of AP-1.
    Okimoto T, Kohno K, Kuwano M, Gopas J, Kung HF, Ono M.
    Oncogene; 1996 Apr 18; 12(8):1625-33. PubMed ID: 8622882
    [Abstract] [Full Text] [Related]

  • 5. Human T-cell leukemia virus type 1 tax protein activates transcription through AP-1 site by inducing DNA binding activity in T cells.
    Iwai K, Mori N, Oie M, Yamamoto N, Fujii M.
    Virology; 2001 Jan 05; 279(1):38-46. PubMed ID: 11145887
    [Abstract] [Full Text] [Related]

  • 6. Suppression of cellular transformation by glucocorticoid receptor mutants.
    Kameda T, Iba H.
    Cancer Res; 1998 Mar 01; 58(5):867-70. PubMed ID: 9500440
    [Abstract] [Full Text] [Related]

  • 7. [Transcription of c-fos gene and DNA binding activity of transcription factor AP-1 increase upon differentiation of mouse F9 teratocarcinoma cells].
    Chuĭkin IA, Lianguzova MS, Pospelov VA.
    Tsitologiia; 2004 Mar 01; 46(12):1080-90. PubMed ID: 15747838
    [Abstract] [Full Text] [Related]

  • 8. Impaired induction of c-fos/c-jun genes and of transcriptional regulatory proteins binding distinct c-fos/c-jun promoter elements in activated human T cells during aging.
    Whisler RL, Chen M, Beiqing L, Carle KW.
    Cell Immunol; 1997 Jan 10; 175(1):41-50. PubMed ID: 9015187
    [Abstract] [Full Text] [Related]

  • 9. Downregulation of c-fos gene transcription in cells transformed by E1A and cHa-ras oncogenes: a role of sustained activation of MAP/ERK kinase cascade and of inactive chromatin structure at c-fos promoter.
    Kukushkin AN, Abramova MV, Svetlikova SB, Darieva ZA, Pospelova TV, Pospelov VA.
    Oncogene; 2002 Jan 24; 21(5):719-30. PubMed ID: 11850800
    [Abstract] [Full Text] [Related]

  • 10. AP-1 (Fos-Jun) regulation by IP-1: effect of signal transduction pathways and cell growth.
    Auwerx J, Sassone-Corsi P.
    Oncogene; 1992 Nov 24; 7(11):2271-80. PubMed ID: 1437149
    [Abstract] [Full Text] [Related]

  • 11. Rap1 reverses transcriptional repression of TGF-beta type II receptor by a mechanism involving AP-1 in the human pancreatic cancer cell line, UK Pan-1.
    Fralix KD, Zhao S, Venkatasubbarao K, Freeman JW.
    J Cell Physiol; 2003 Jan 24; 194(1):88-99. PubMed ID: 12447993
    [Abstract] [Full Text] [Related]

  • 12. Transcriptional induction of cyclooxygenase-2 gene by okadaic acid inhibition of phosphatase activity in human chondrocytes: co-stimulation of AP-1 and CRE nuclear binding proteins.
    Miller C, Zhang M, He Y, Zhao J, Pelletier JP, Martel-Pelletier J, Di Battista JA.
    J Cell Biochem; 1998 Jun 15; 69(4):392-413. PubMed ID: 9620167
    [Abstract] [Full Text] [Related]

  • 13. Signaling pathways in the induction of c-fos and c-jun proto-oncogenes by 3-methylcholanthrene.
    Ainbinder E, Bergelson S, Daniel V.
    Recept Signal Transduct; 1997 Jun 15; 7(4):279-89. PubMed ID: 9633828
    [Abstract] [Full Text] [Related]

  • 14. Antisense targeting of c-fos transcripts inhibits serum- and TGF-beta 1-stimulated PAI-1 gene expression and directed motility in renal epithelial cells.
    Kutz SM, Providence KM, Higgins PJ.
    Cell Motil Cytoskeleton; 2001 Mar 15; 48(3):163-74. PubMed ID: 11223948
    [Abstract] [Full Text] [Related]

  • 15. A peptide fragment of ependymin neurotrophic factor uses protein kinase C and the mitogen-activated protein kinase pathway to activate c-Jun N-terminal kinase and a functional AP-1 containing c-Jun and c-Fos proteins in mouse NB2a cells.
    Adams DS, Hasson B, Boyer-Boiteau A, El-Khishin A, Shashoua VE.
    J Neurosci Res; 2003 May 01; 72(3):405-16. PubMed ID: 12692907
    [Abstract] [Full Text] [Related]

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  • 18. Transformation blocks differentiation-induced inhibition of serum response factor interactions with serum response elements.
    Ding W, Witte MM, Scott RE.
    Cancer Res; 1999 Aug 01; 59(15):3795-802. PubMed ID: 10446998
    [Abstract] [Full Text] [Related]

  • 19. Endothelin-1-induced cardiac hypertrophy is inhibited by activation of peroxisome proliferator-activated receptor-alpha partly via blockade of c-Jun NH2-terminal kinase pathway.
    Irukayama-Tomobe Y, Miyauchi T, Sakai S, Kasuya Y, Ogata T, Takanashi M, Iemitsu M, Sudo T, Goto K, Yamaguchi I.
    Circulation; 2004 Feb 24; 109(7):904-10. PubMed ID: 14967736
    [Abstract] [Full Text] [Related]

  • 20. B-ATF functions as a negative regulator of AP-1 mediated transcription and blocks cellular transformation by Ras and Fos.
    Echlin DR, Tae HJ, Mitin N, Taparowsky EJ.
    Oncogene; 2000 Mar 30; 19(14):1752-63. PubMed ID: 10777209
    [Abstract] [Full Text] [Related]

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