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180 related items for PubMed ID: 8890955

  • 21. Opposing activities of c-Fos and Fra-2 on AP-1 regulated transcriptional activity in mouse keratinocytes induced to differentiate by calcium and phorbol esters.
    Rutberg SE, Saez E, Lo S, Jang SI, Markova N, Spiegelman BM, Yuspa SH.
    Oncogene; 1997 Sep; 15(11):1337-46. PubMed ID: 9315102
    [Abstract] [Full Text] [Related]

  • 22. Coordinate regulation of glucocorticoid receptor and c-jun gene expression is cell type-specific and exhibits differential hormonal sensitivity for down- and up-regulation.
    Barrett TJ, Vig E, Vedeckis WV.
    Biochemistry; 1996 Jul 30; 35(30):9746-53. PubMed ID: 8703946
    [Abstract] [Full Text] [Related]

  • 23. Effects of mutationally activated Ha-ras on c-fos expression kinetics in rat tracheal epithelial cells.
    Hubbard FC, Cosma G, Garte SJ.
    Mol Carcinog; 1996 Jun 30; 16(2):77-82. PubMed ID: 8645429
    [Abstract] [Full Text] [Related]

  • 24. Transcriptional regulation of intercellular adhesion molecule 1 by phorbol ester in human neuroblastoma cell line SK-N-SH involves jun- and fos-containing activator protein 1 site binding complex(es).
    Farina AR, Cappabianca L, Mackay AR, Tiberio A, Tacconelli A, Tessitore A, Frati L, Martinotti S, Gulino A.
    Cell Growth Differ; 1997 Jul 30; 8(7):789-800. PubMed ID: 9218873
    [Abstract] [Full Text] [Related]

  • 25. Transition from SCLC to NSCLC phenotype is accompanied by an increased TRE-binding activity and recruitment of specific AP-1 proteins.
    Risse-Hackl G, Adamkiewicz J, Wimmel A, Schuermann M.
    Oncogene; 1998 Jun 11; 16(23):3057-68. PubMed ID: 9662339
    [Abstract] [Full Text] [Related]

  • 26. Hormonal regulation of the c-fms proto-oncogene in breast cancer cells is mediated by a composite glucocorticoid response element.
    Flick MB, Sapi E, Kacinski BM.
    J Cell Biochem; 2002 Jun 11; 85(1):10-23. PubMed ID: 11891846
    [Abstract] [Full Text] [Related]

  • 27. Inhibition of activator protein 1 activity and cell growth by purified green tea and black tea polyphenols in H-ras-transformed cells: structure-activity relationship and mechanisms involved.
    Chung JY, Huang C, Meng X, Dong Z, Yang CS.
    Cancer Res; 1999 Sep 15; 59(18):4610-7. PubMed ID: 10493515
    [Abstract] [Full Text] [Related]

  • 28. Oncogenic transformation by ras and fos is mediated by c-Jun N-terminal phosphorylation.
    Behrens A, Jochum W, Sibilia M, Wagner EF.
    Oncogene; 2000 May 18; 19(22):2657-63. PubMed ID: 10851065
    [Abstract] [Full Text] [Related]

  • 29. Transcriptional activation of the hepatocyte growth factor receptor (c-met) gene by its ligand (hepatocyte growth factor) is mediated through AP-1.
    Seol DW, Chen Q, Zarnegar R.
    Oncogene; 2000 Feb 24; 19(9):1132-7. PubMed ID: 10713700
    [Abstract] [Full Text] [Related]

  • 30. A dominant role for the c-Jun NH2-terminal kinase in oncogenic ras-induced morphologic transformation of human lung carcinoma cells.
    Xiao L, Lang W.
    Cancer Res; 2000 Jan 15; 60(2):400-8. PubMed ID: 10667594
    [Abstract] [Full Text] [Related]

  • 31. Induction of c-fos, c-jun, junB and junD mRNA and AP-1 by alkylating mutagens in cells deficient and proficient for the DNA repair protein O6-methylguanine-DNA methyltransferase (MGMT) and its relationship to cell death, mutation induction and chromosomal instability.
    Dosch J, Kaina B.
    Oncogene; 1996 Nov 07; 13(9):1927-35. PubMed ID: 8934539
    [Abstract] [Full Text] [Related]

  • 32. Ursolic acid-induced down-regulation of MMP-9 gene is mediated through the nuclear translocation of glucocorticoid receptor in HT1080 human fibrosarcoma cells.
    Cha HJ, Park MT, Chung HY, Kim ND, Sato H, Seiki M, Kim KW.
    Oncogene; 1998 Feb 12; 16(6):771-8. PubMed ID: 9488041
    [Abstract] [Full Text] [Related]

  • 33. Expression of dominant negative Erk2 inhibits AP-1 transactivation and neoplastic transformation.
    Watts RG, Huang C, Young MR, Li JJ, Dong Z, Pennie WD, Colburn NH.
    Oncogene; 1998 Dec 31; 17(26):3493-8. PubMed ID: 10030673
    [Abstract] [Full Text] [Related]

  • 34. mos-induced inhibition of glucocorticoid receptor function is mediated by Fos.
    Touray M, Ryan F, Saurer S, Martin F, Jaggi R.
    Oncogene; 1991 Feb 31; 6(2):211-7. PubMed ID: 1847998
    [Abstract] [Full Text] [Related]

  • 35. AP-1 transrepressing retinoic acid does not deplete coactivators or AP-1 monomers but may target specific Jun or Fos containing dimers.
    Suzukawa K, Colburn NH.
    Oncogene; 2002 Mar 28; 21(14):2181-90. PubMed ID: 11948401
    [Abstract] [Full Text] [Related]

  • 36. Presence of distinct AP-1 dimers in normal and transformed rat hepatocytes under basal conditions and after epidermal growth factor stimulation.
    Nadori F, Lardeux B, Rahmani M, Bringuier A, Durand-Schneider AM, Bernuau D.
    Hepatology; 1997 Dec 28; 26(6):1477-83. PubMed ID: 9397987
    [Abstract] [Full Text] [Related]

  • 37. Tumorigenic and metastatic properties of two ras-oncogene transfected rat fibrosarcoma cell lines defective in c-jun.
    Marshall-Heyman H, Engel G, Ljungdahl S, Shoshan MC, Svensson C, Wasylyk B, Linder S.
    Oncogene; 1994 Dec 28; 9(12):3655-63. PubMed ID: 7970724
    [Abstract] [Full Text] [Related]

  • 38. Binding of the 97 kDa glucocorticoid receptor to the 5' upstream flanking region of the mouse c-Ha-ras oncogene.
    Strawhecker JM, Betz NA, Neades RY, Houser W, Pelling JC.
    Oncogene; 1989 Nov 28; 4(11):1317-22. PubMed ID: 2682459
    [Abstract] [Full Text] [Related]

  • 39. Inhibition of Fos- and Ras-induced transformation by mutant Fos proteins with structural alterations in functionally different domains.
    Wick M, Lucibello FC, Müller R.
    Oncogene; 1992 May 28; 7(5):859-67. PubMed ID: 1570155
    [Abstract] [Full Text] [Related]

  • 40. Transformation suppressor activity of a Jun transcription factor lacking its activation domain.
    Lloyd A, Yancheva N, Wasylyk B.
    Nature; 1991 Aug 15; 352(6336):635-8. PubMed ID: 1907719
    [Abstract] [Full Text] [Related]

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