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226 related items for PubMed ID: 8895748

  • 1. Loss of retinoic acid receptors in mouse skin and skin tumors is associated with activation of the ras(Ha) oncogene and high risk for premalignant progression.
    Darwiche N, Scita G, Jones C, Rutberg S, Greenwald E, Tennenbaum T, Collins SJ, De Luca LM, Yuspa SH.
    Cancer Res; 1996 Nov 01; 56(21):4942-9. PubMed ID: 8895748
    [Abstract] [Full Text] [Related]

  • 2. Up-regulation of vascular endothelial growth factor/vascular permeability factor in mouse skin carcinogenesis correlates with malignant progression state and activated H-ras expression levels.
    Larcher F, Robles AI, Duran H, Murillas R, Quintanilla M, Cano A, Conti CJ, Jorcano JL.
    Cancer Res; 1996 Dec 01; 56(23):5391-6. PubMed ID: 8968091
    [Abstract] [Full Text] [Related]

  • 3. Topical retinoic acid reduces skin papilloma formation but resistant papillomas are at high risk for malignant conversion.
    Tennenbaum T, Lowry D, Darwiche N, Morgan DL, Gartsbein M, Hansen L, De Luca LM, Hennings H, Yuspa SH.
    Cancer Res; 1998 Apr 01; 58(7):1435-43. PubMed ID: 9537245
    [Abstract] [Full Text] [Related]

  • 4. PTEN loss promotes rasHa-mediated papillomatogenesis via dual up-regulation of AKT activity and cell cycle deregulation but malignant conversion proceeds via PTEN-associated pathways.
    Yao D, Alexander CL, Quinn JA, Porter MJ, Wu H, Greenhalgh DA.
    Cancer Res; 2006 Feb 01; 66(3):1302-12. PubMed ID: 16452183
    [Abstract] [Full Text] [Related]

  • 5. Genetic alterations cooperate with v-Ha-ras to accelerate multistage carcinogenesis in TG.AC transgenic mouse skin.
    Owens DM, Spalding JW, Tennant RW, Smart RC.
    Cancer Res; 1995 Jul 15; 55(14):3171-8. PubMed ID: 7606738
    [Abstract] [Full Text] [Related]

  • 6. Overexpression of retinoic acid receptors alpha and gamma into neoplastic epidermal cells causes retinoic acid-induced growth arrest and apoptosis.
    Hatoum A, El-Sabban ME, Khoury J, Yuspa SH, Darwiche N.
    Carcinogenesis; 2001 Dec 15; 22(12):1955-63. PubMed ID: 11751425
    [Abstract] [Full Text] [Related]

  • 7. Protection against malignant conversion in SENCAR mouse skin by all trans retinoic acid: inhibition of the ras p21-processing enzyme farnesyltransferase and Ha-ras p21 membrane localization.
    Agarwal R, Mohan RR, Ahmad N, Mukhtar H.
    Mol Carcinog; 1996 Sep 15; 17(1):13-22. PubMed ID: 8876671
    [Abstract] [Full Text] [Related]

  • 8. Smad7 but not Smad6 cooperates with oncogenic ras to cause malignant conversion in a mouse model for squamous cell carcinoma.
    Liu X, Lee J, Cooley M, Bhogte E, Hartley S, Glick A.
    Cancer Res; 2003 Nov 15; 63(22):7760-8. PubMed ID: 14633701
    [Abstract] [Full Text] [Related]

  • 9. Targeted disruption of the epidermal growth factor receptor impairs growth of squamous papillomas expressing the v-ras(Ha) oncogene but does not block in vitro keratinocyte responses to oncogenic ras.
    Dlugosz AA, Hansen L, Cheng C, Alexander N, Denning MF, Threadgill DW, Magnuson T, Coffey RJ, Yuspa SH.
    Cancer Res; 1997 Aug 01; 57(15):3180-8. PubMed ID: 9242447
    [Abstract] [Full Text] [Related]

  • 10. Development of an in vitro model to study carcinogen-induced neoplastic progression of initiated mouse epidermal cells.
    Morgan D, Welty D, Glick A, Greenhalgh D, Hennings H, Yuspa SH.
    Cancer Res; 1992 Jun 01; 52(11):3145-56. PubMed ID: 1375535
    [Abstract] [Full Text] [Related]

  • 11. 12-O-tetradecanoylphorbol-13-acetate promotion of transgenic mouse epidermis coexpressing transforming growth factor-alpha and v-fos: acceleration of autonomous papilloma formation and malignant conversion via c-Ha-ras activation.
    Wang XJ, Liefer KM, Greenhalgh DA, Roop DR.
    Mol Carcinog; 1999 Dec 01; 26(4):305-11. PubMed ID: 10569807
    [Abstract] [Full Text] [Related]

  • 12. RARgamma acts as a tumor suppressor in mouse keratinocytes.
    Chen CF, Goyette P, Lohnes D.
    Oncogene; 2004 Jul 08; 23(31):5350-9. PubMed ID: 15094780
    [Abstract] [Full Text] [Related]

  • 13. Autocrine transforming growth factor alpha is dispensible for v-rasHa-induced epidermal neoplasia: potential involvement of alternate epidermal growth factor receptor ligands.
    Dlugosz AA, Cheng C, Williams EK, Darwiche N, Dempsey PJ, Mann B, Dunn AR, Coffey RJ, Yuspa SH.
    Cancer Res; 1995 May 01; 55(9):1883-93. PubMed ID: 7728756
    [Abstract] [Full Text] [Related]

  • 14. Loss of p21CIP1/WAF1 does not recapitulate accelerated malignant conversion caused by p53 loss in experimental skin carcinogenesis.
    Weinberg WC, Montano NE, Deng C.
    Oncogene; 1997 Aug 07; 15(6):685-90. PubMed ID: 9264409
    [Abstract] [Full Text] [Related]

  • 15. Targeted expression of bcl-2 to murine basal epidermal keratinocytes results in paradoxical retardation of ultraviolet- and chemical-induced tumorigenesis.
    Rossiter H, Beissert S, Mayer C, Schön MP, Wienrich BG, Tschachler E, Kupper TS.
    Cancer Res; 2001 May 01; 61(9):3619-26. PubMed ID: 11325830
    [Abstract] [Full Text] [Related]

  • 16. Activator protein 1 transcription factors are fundamental to v-rasHa-induced changes in gene expression in neoplastic keratinocytes.
    Rutberg SE, Adams TL, Glick A, Bonovich MT, Vinson C, Yuspa SH.
    Cancer Res; 2000 Nov 15; 60(22):6332-8. PubMed ID: 11103794
    [Abstract] [Full Text] [Related]

  • 17. Frequent codon 12 Ki-ras mutations in mouse skin tumors initiated by N-methyl-N'-nitro-N-nitrosoguanidine and promoted by mezerein.
    Rehman I, Lowry DT, Adams C, Abdel-Fattah R, Holly A, Yuspa SH, Hennings H.
    Mol Carcinog; 2000 Apr 15; 27(4):298-307. PubMed ID: 10747294
    [Abstract] [Full Text] [Related]

  • 18. Oncogene activation and tumor suppressor gene inactivation during multistage mouse skin carcinogenesis.
    Bowden GT, Schneider B, Domann R, Kulesz-Martin M.
    Cancer Res; 1994 Apr 01; 54(7 Suppl):1882s-1885s. PubMed ID: 8137304
    [Abstract] [Full Text] [Related]

  • 19. Transgenic coexpression of v-Ha-ras and transforming growth factor alpha increases epidermal hyperproliferation and tumorigenesis and predisposes to malignant conversion via endogenous c-Ha-ras activation.
    Wang XJ, Greenhalgh DA, Roop DR.
    Mol Carcinog; 2000 Mar 01; 27(3):200-9. PubMed ID: 10708482
    [Abstract] [Full Text] [Related]

  • 20. Definition by specific antisense oligonucleotides of a role for protein kinase C alpha in expression of differentiation markers in normal and neoplastic mouse epidermal keratinocytes.
    Lee YS, Dlugosz AA, McKay R, Dean NM, Yuspa SH.
    Mol Carcinog; 1997 Jan 01; 18(1):44-53. PubMed ID: 9022812
    [Abstract] [Full Text] [Related]

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