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Journal Abstract Search
709 related items for PubMed ID: 8918887
1. BID: a novel BH3 domain-only death agonist. Wang K, Yin XM, Chao DT, Milliman CL, Korsmeyer SJ. Genes Dev; 1996 Nov 15; 10(22):2859-69. PubMed ID: 8918887 [Abstract] [Full Text] [Related]
2. MAP-1, a novel proapoptotic protein containing a BH3-like motif that associates with Bax through its Bcl-2 homology domains. Tan KO, Tan KM, Chan SL, Yee KS, Bevort M, Ang KC, Yu VC. J Biol Chem; 2001 Jan 26; 276(4):2802-7. PubMed ID: 11060313 [Abstract] [Full Text] [Related]
3. Proapoptotic protein Bax heterodimerizes with Bcl-2 and homodimerizes with Bax via a novel domain (BH3) distinct from BH1 and BH2. Zha H, Aimé-Sempé C, Sato T, Reed JC. J Biol Chem; 1996 Mar 29; 271(13):7440-4. PubMed ID: 8631771 [Abstract] [Full Text] [Related]
4. Characterization of the anti-apoptotic mechanism of Bcl-B. Zhai D, Ke N, Zhang H, Ladror U, Joseph M, Eichinger A, Godzik A, Ng SC, Reed JC. Biochem J; 2003 Nov 15; 376(Pt 1):229-36. PubMed ID: 12921534 [Abstract] [Full Text] [Related]
5. The C. elegans orthologue ceBNIP3 interacts with CED-9 and CED-3 but kills through a BH3- and caspase-independent mechanism. Cizeau J, Ray R, Chen G, Gietz RD, Greenberg AH. Oncogene; 2000 Nov 16; 19(48):5453-63. PubMed ID: 11114722 [Abstract] [Full Text] [Related]
6. Bcl-X(L) and calyculin A prevent translocation of Bax to mitochondria during apoptosis. Ganju N, Eastman A. Biochem Biophys Res Commun; 2002 Mar 15; 291(5):1258-64. PubMed ID: 11883953 [Abstract] [Full Text] [Related]
7. Direct addition of BimL to mitochondria does not lead to cytochrome c release. Terradillos O, Montessuit S, Huang DC, Martinou JC. FEBS Lett; 2002 Jul 03; 522(1-3):29-34. PubMed ID: 12095614 [Abstract] [Full Text] [Related]
8. Identification of novel isoforms of the BH3 domain protein Bim which directly activate Bax to trigger apoptosis. Marani M, Tenev T, Hancock D, Downward J, Lemoine NR. Mol Cell Biol; 2002 Jun 03; 22(11):3577-89. PubMed ID: 11997495 [Abstract] [Full Text] [Related]
9. BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly. Kuwana T, Bouchier-Hayes L, Chipuk JE, Bonzon C, Sullivan BA, Green DR, Newmeyer DD. Mol Cell; 2005 Feb 18; 17(4):525-35. PubMed ID: 15721256 [Abstract] [Full Text] [Related]
10. Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis. Desagher S, Osen-Sand A, Nichols A, Eskes R, Montessuit S, Lauper S, Maundrell K, Antonsson B, Martinou JC. J Cell Biol; 1999 Mar 08; 144(5):891-901. PubMed ID: 10085289 [Abstract] [Full Text] [Related]
11. Bid chimeras indicate that most BH3-only proteins can directly activate Bak and Bax, and show no preference for Bak versus Bax. Hockings C, Anwari K, Ninnis RL, Brouwer J, O'Hely M, Evangelista M, Hinds MG, Czabotar PE, Lee EF, Fairlie WD, Dewson G, Kluck RM. Cell Death Dis; 2015 Apr 23; 6(4):e1735. PubMed ID: 25906158 [Abstract] [Full Text] [Related]
12. Induction of apoptosis in prostate carcinoma cells by BH3 peptides which inhibit Bak/Bcl-2 interactions. Finnegan NM, Curtin JF, Prevost G, Morgan B, Cotter TG. Br J Cancer; 2001 Jul 06; 85(1):115-21. PubMed ID: 11437412 [Abstract] [Full Text] [Related]
13. Membrane-insertion fragments of Bcl-xL, Bax, and Bid. García-Sáez AJ, Mingarro I, Pérez-Payá E, Salgado J. Biochemistry; 2004 Aug 31; 43(34):10930-43. PubMed ID: 15323553 [Abstract] [Full Text] [Related]
14. harakiri, a novel regulator of cell death, encodes a protein that activates apoptosis and interacts selectively with survival-promoting proteins Bcl-2 and Bcl-X(L). Inohara N, Ding L, Chen S, Núñez G. EMBO J; 1997 Apr 01; 16(7):1686-94. PubMed ID: 9130713 [Abstract] [Full Text] [Related]
15. Functional identification of the apoptosis effector BH3 domain in cellular protein BNIP1. Yasuda M, Chinnadurai G. Oncogene; 2000 May 04; 19(19):2363-7. PubMed ID: 10822388 [Abstract] [Full Text] [Related]
16. Bid-induced cytochrome c release is mediated by a pathway independent of mitochondrial permeability transition pore and Bax. Kim TH, Zhao Y, Barber MJ, Kuharsky DK, Yin XM. J Biol Chem; 2000 Dec 15; 275(50):39474-81. PubMed ID: 10982793 [Abstract] [Full Text] [Related]
17. The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4. Huang DC, Adams JM, Cory S. EMBO J; 1998 Feb 16; 17(4):1029-39. PubMed ID: 9463381 [Abstract] [Full Text] [Related]
18. Trail-induced apoptosis in Type I leukemic cells is not enhanced by overexpression of bax. Jia L, Patwari Y, Kelsey SM, Newland AC. Biochem Biophys Res Commun; 2001 May 25; 283(5):1037-45. PubMed ID: 11355877 [Abstract] [Full Text] [Related]
19. The first alpha helix of Bax plays a necessary role in its ligand-induced activation by the BH3-only proteins Bid and PUMA. Cartron PF, Gallenne T, Bougras G, Gautier F, Manero F, Vusio P, Meflah K, Vallette FM, Juin P. Mol Cell; 2004 Dec 03; 16(5):807-18. PubMed ID: 15574335 [Abstract] [Full Text] [Related]