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Journal Abstract Search

347 related items for PubMed ID: 8931405

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  • 5. Prospective screening for pediatric mitochondrial trifunctional protein defects in pregnancies complicated by liver disease.
    Yang Z, Yamada J, Zhao Y, Strauss AW, Ibdah JA.
    JAMA; 2002 Nov 06; 288(17):2163-6. PubMed ID: 12413376
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  • 6. Heterozygosity for the common LCHAD mutation (1528g>C) is not a major cause of HELLP syndrome and the prevalence of the mutation in the Dutch population is low.
    den Boer ME, Ijlst L, Wijburg FA, Oostheim W, van Werkhoven MA, van Pampus MG, Heymans HS, Wanders RJ.
    Pediatr Res; 2000 Aug 06; 48(2):151-4. PubMed ID: 10926288
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  • 7. Acute fatty liver of pregnancy and long-chain 3-hydroxyacyl-coenzyme A dehydrogenase deficiency.
    Treem WR, Rinaldo P, Hale DE, Stanley CA, Millington DS, Hyams JS, Jackson S, Turnbull DM.
    Hepatology; 1994 Feb 06; 19(2):339-45. PubMed ID: 8294091
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  • 8. Highly active antiretroviral therapy does not affect mitochondrial beta-oxidation of fatty acids: an in vitro study in fibroblasts.
    Butt YM, Kamrudin SA, Rakheja D.
    Pediatr Dev Pathol; 2008 Feb 06; 11(1):35-8. PubMed ID: 18240871
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  • 10. Neither maternal nor fetal mutation (E474Q) in the alpha-subunit of the trifunctional protein is frequent in pregnancies complicated by HELLP syndrome.
    Mütze S, Ahillen I, Rudnik-Schoeneborn S, Eggermann T, Leeners B, Neumaier-Wagner PM, Kuse S, Rath W, Zerres K.
    J Perinat Med; 2007 Feb 06; 35(1):76-8. PubMed ID: 17313315
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  • 11. Evidence for fatty acid oxidation in human placenta, and the relationship of fatty acid oxidation enzyme activities with gestational age.
    Rakheja D, Bennett MJ, Foster BM, Domiati-Saad R, Rogers BB.
    Placenta; 2002 May 06; 23(5):447-50. PubMed ID: 12061861
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  • 12. Long-chain fatty acid oxidation during early human development.
    Oey NA, den Boer ME, Wijburg FA, Vekemans M, Augé J, Steiner C, Wanders RJ, Waterham HR, Ruiter JP, Attié-Bitach T.
    Pediatr Res; 2005 Jun 06; 57(6):755-9. PubMed ID: 15845636
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  • 13. No mutation was found in the alpha-subunit of the mitochondrial tri-functional protein in one patient with severe acute fatty liver of pregnancy and her relatives.
    Kong XF, Zhang XX, Yu YY, Shi Q, La DD, Zhu-Ge CD, Deng L, Gong QM, Shen BY, Peng CH, Li HW.
    J Gastroenterol Hepatol; 2007 Dec 06; 22(12):2107-11. PubMed ID: 18031367
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  • 14. Elevated hydroxyacylcarnitines in a carrier of LCHAD deficiency during acute liver disease of pregnancy - a common feature of the pregnancy complication?
    Eskelin PM, Laitinen KA, Tyni TA.
    Mol Genet Metab; 2010 Jun 06; 100(2):204-6. PubMed ID: 20363656
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  • 15. Fetal fatty acid oxidation defects and maternal liver disease in pregnancy.
    Browning MF, Levy HL, Wilkins-Haug LE, Larson C, Shih VE.
    Obstet Gynecol; 2006 Jan 06; 107(1):115-20. PubMed ID: 16394048
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  • 16. The molecular basis of pediatric long chain 3-hydroxyacyl-CoA dehydrogenase deficiency associated with maternal acute fatty liver of pregnancy.
    Sims HF, Brackett JC, Powell CK, Treem WR, Hale DE, Bennett MJ, Gibson B, Shapiro S, Strauss AW.
    Proc Natl Acad Sci U S A; 1995 Jan 31; 92(3):841-5. PubMed ID: 7846063
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  • 19. Mitochondrial fatty acid oxidation and acute fatty liver of pregnancy.
    Treem WR.
    Semin Gastrointest Dis; 2002 Jan 31; 13(1):55-66. PubMed ID: 11944635
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