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166 related items for PubMed ID: 8971365

  • 1. Biliary excretion of copper, manganese, and horseradish peroxidase in Eisai hyperbilirubinemic mutant rats (EHBRs) with defective biliary excretion of glutathione.
    Sugawara N, Lai Y, Yuasa M, Dhar SK, Arizono K.
    Biol Trace Elem Res; 1996; 55(1-2):181-9. PubMed ID: 8971365
    [Abstract] [Full Text] [Related]

  • 2. Primary active transport of pravastatin across the liver canalicular membrane in normal and mutant Eisai hyperbilirubinemic rats.
    Yamazaki M, Kobayashi K, Sugiyama Y.
    Biopharm Drug Dispos; 1996 Oct; 17(7):607-21. PubMed ID: 8894118
    [Abstract] [Full Text] [Related]

  • 3. Biliary excretion of exogenous cadmium, and endogenous copper and zinc in the Eisai hyperbilirubinuric (EHB) rat with a near absence of biliary glutathione.
    Sugawara N, Lai YR, Arizono K, Ariyoshi T.
    Toxicology; 1996 Aug 01; 112(1):87-94. PubMed ID: 8792851
    [Abstract] [Full Text] [Related]

  • 4. Primary active transport of pravastatin across the liver canalicular membrane in normal and mutant Eisai hyperbilirubinaemic rats.
    Yamazaki M, Kobayashi K, Sugiyama Y.
    Biopharm Drug Dispos; 1996 Nov 01; 17(8):645-59. PubMed ID: 8950045
    [Abstract] [Full Text] [Related]

  • 5. Biliary and renal excretions of cefpiramide in Eisai hyperbilirubinemic rats.
    Muraoka I, Hasegawa T, Nadai M, Wang L, Haghgoo S, Tagaya O, Nabeshima T.
    Antimicrob Agents Chemother; 1995 Jan 01; 39(1):70-4. PubMed ID: 7695332
    [Abstract] [Full Text] [Related]

  • 6. Lack of biliary excretion of Cd linked to an inherent defect of the canalicular isoform of multidrug resistance protein (cMrp) does not abnormally stimulate accumulation of Cd in the Eisai hyperbilirubinemic (EHB) rat liver.
    Sugawara N, Lai YR, Arizono K, Kitajima T, Inoue H.
    Arch Toxicol; 1997 Jan 01; 71(5):336-9. PubMed ID: 9137813
    [Abstract] [Full Text] [Related]

  • 7. Biliary excretion of copper, metallothionein, and glutathione into Long-Evans Cinnamon rats: a convincing animal model for Wilson disease.
    Sugawara N, Sato M, Yuasa M, Sugawara C.
    Biochem Mol Med; 1995 Jun 01; 55(1):38-42. PubMed ID: 7551824
    [Abstract] [Full Text] [Related]

  • 8. The mutant Eisai hyperbilirubinemic rat is resistant to bile acid-induced cholestasis and cytotoxicity.
    Hoshino M, Hayakawa T, Hirano A, Kamiya Y, Ohiwa T, Tanaka A, Kumai T, Inagaki T, Miyaji M, Takeuchi T.
    Hepatology; 1994 Oct 01; 20(4 Pt 1):932-9. PubMed ID: 7927235
    [Abstract] [Full Text] [Related]

  • 9. Alterations in the pharmacokinetics and protein binding of enprofylline in Eisai hyperbilirubinemic rats.
    Nadai M, Hasegawa T, Wang L, Tagaya O, Nabeshima T.
    Drug Metab Dispos; 1994 Oct 01; 22(4):561-5. PubMed ID: 7956730
    [Abstract] [Full Text] [Related]

  • 10. Mechanism of hepatobiliary transport of a novel thromboxane A2 receptor antagonist, [2-(4-chlorophenylsulfonylaminomethyl)indan-5-yl]acetate (Z-335), and its xenobiotic taurine conjugate (Z-335-Tau) in rats.
    Kawabata Y, Nakamura H, Kamada E, Furuta S, Shinozaki Y, Kurimoto T, Nishigaki R.
    J Pharm Sci; 2003 Jan 01; 92(1):67-76. PubMed ID: 12486683
    [Abstract] [Full Text] [Related]

  • 11. Hepatobiliary transport of YM466, a novel factor Xa inhibitor, in rats.
    Mano Y, Usui T, Kamimura H.
    Eur J Drug Metab Pharmacokinet; 2006 Jan 01; 31(2):117-21. PubMed ID: 16898080
    [Abstract] [Full Text] [Related]

  • 12. Impaired biliary excretion and whole body elimination of methylmercury in rats with congenital defect in biliary glutathione excretion.
    Ballatori N, Gatmaitan Z, Truong AT.
    Hepatology; 1995 Nov 01; 22(5):1469-73. PubMed ID: 7590665
    [Abstract] [Full Text] [Related]

  • 13. Decreased pancreatic exocrine function in the mutant Eisai hyperbilirubinemic rat (EHBR).
    Inagaki T, Hoshino M, Ohara H, Yamada T, Ogasawara T, Shimizu H, Itoh M.
    Pancreas; 1999 Mar 01; 18(2):172-9. PubMed ID: 10090415
    [Abstract] [Full Text] [Related]

  • 14. Recombinant human hepatocyte growth factor facilitates biliary transport after hepatocyte transplantation in Eisai hyperbilirubinemic rats.
    Yamaguchi Y, Hamaguchi H, Yamada S, Fujiwara K, Higashio K, Miyanari N, Ichiguchi O, Goto M, Mori K, Ogawa M.
    Dig Dis Sci; 1997 Mar 01; 42(3):522-8. PubMed ID: 9073133
    [Abstract] [Full Text] [Related]

  • 15. Biliary excretion of exogenous cadmium and manganese in Long-Evans Cinnamon (LEC) rats characterized by an inherently gross amount of copper-metallothionein in the liver.
    Sugawara N, Li D, Sugawara C.
    Arch Toxicol; 1994 Mar 01; 68(8):520-3. PubMed ID: 7802594
    [Abstract] [Full Text] [Related]

  • 16. Both cMOAT/MRP2 and another unknown transporter(s) are responsible for the biliary excretion of glucuronide conjugate of the nonpeptide angiotensin II antagonist, telmisaltan.
    Nishino A, Kato Y, Igarashi T, Sugiyama Y.
    Drug Metab Dispos; 2000 Oct 01; 28(10):1146-8. PubMed ID: 10997931
    [Abstract] [Full Text] [Related]

  • 17. Increase in bile flow and biliary excretion of glutathione-derived sulfhydryls in rats by drug-metabolizing enzyme inducers is mediated by multidrug resistance protein 2.
    Johnson DR, Habeebu SS, Klaassen CD.
    Toxicol Sci; 2002 Mar 01; 66(1):16-26. PubMed ID: 11861969
    [Abstract] [Full Text] [Related]

  • 18. Bile flow in a mutant Sprague-Dawley rat with defective biliary excretion of glutathione.
    Yoshida H, Okano K, Tamano T, Kuronuma Y, Iijima M, Harada T.
    J Gastroenterol; 1994 Aug 01; 29(4):439-42. PubMed ID: 7951853
    [Abstract] [Full Text] [Related]

  • 19. Alterations in glutathione homeostasis in mutant Eisai hyperbilirubinemic rats.
    Lu SC, Cai J, Kuhlenkamp J, Sun WM, Takikawa H, Takenaka O, Horie T, Yi J, Kaplowitz N.
    Hepatology; 1996 Jul 01; 24(1):253-8. PubMed ID: 8707271
    [Abstract] [Full Text] [Related]

  • 20. Biliary excretion of bile acid conjugates in a hyperbilirubinemic mutant Sprague-Dawley rat.
    Takikawa H, Sano N, Narita T, Uchida Y, Yamanaka M, Horie T, Mikami T, Tagaya O.
    Hepatology; 1991 Aug 01; 14(2):352-60. PubMed ID: 1860692
    [Abstract] [Full Text] [Related]

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