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Journal Abstract Search

209 related items for PubMed ID: 9000126

  • 1. PKC epsilon functions as an oncogene by enhancing activation of the Raf kinase.
    Cacace AM, Ueffing M, Philipp A, Han EK, Kolch W, Weinstein IB.
    Oncogene; 1996 Dec 19; 13(12):2517-26. PubMed ID: 9000126
    [Abstract] [Full Text] [Related]

  • 2. Overexpression of PKCepsilon in R6 fibroblasts causes increased production of active TGFbeta.
    Cacace AM, Ueffing M, Han EK, Marmè D, Weinstein IB.
    J Cell Physiol; 1998 Jun 19; 175(3):314-22. PubMed ID: 9572476
    [Abstract] [Full Text] [Related]

  • 3. Protein kinase Cepsilon is oncogenic in colon epithelial cells by interaction with the ras signal transduction pathway.
    Perletti GP, Concari P, Brusaferri S, Marras E, Piccinini F, Tashjian AH.
    Oncogene; 1998 Jun 25; 16(25):3345-8. PubMed ID: 9681835
    [Abstract] [Full Text] [Related]

  • 4. Protein kinase C-epsilon associates with the Raf-1 kinase and induces the production of growth factors that stimulate Raf-1 activity.
    Ueffing M, Lovrić J, Philipp A, Mischak H, Kolch W.
    Oncogene; 1997 Dec 11; 15(24):2921-7. PubMed ID: 9416835
    [Abstract] [Full Text] [Related]

  • 5. The epsilon isoform of protein kinase C is an oncogene when overexpressed in rat fibroblasts.
    Cacace AM, Guadagno SN, Krauss RS, Fabbro D, Weinstein IB.
    Oncogene; 1993 Aug 11; 8(8):2095-104. PubMed ID: 8336936
    [Abstract] [Full Text] [Related]

  • 6. Differential regulation of the p72-74 RAF-1 kinase in 3T3 fibroblasts expressing ras or src oncogenes.
    Reed JC, Yum S, Cuddy MP, Turner BC, Rapp UR.
    Cell Growth Differ; 1991 May 11; 2(5):235-43. PubMed ID: 1888699
    [Abstract] [Full Text] [Related]

  • 7. pim-1 proto-oncogene expression in anti-CD3-mediated T cell activation is associated with protein kinase C activation and is independent of Raf-1.
    Wingett D, Long A, Kelleher D, Magnuson NS.
    J Immunol; 1996 Jan 15; 156(2):549-57. PubMed ID: 8543805
    [Abstract] [Full Text] [Related]

  • 8. Endothelial exposure to hypoxia induces Egr-1 expression involving PKCalpha-mediated Ras/Raf-1/ERK1/2 pathway.
    Lo LW, Cheng JJ, Chiu JJ, Wung BS, Liu YC, Wang DL.
    J Cell Physiol; 2001 Sep 15; 188(3):304-12. PubMed ID: 11473356
    [Abstract] [Full Text] [Related]

  • 9. Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways.
    Rusyn EV, Reynolds ER, Shao H, Grana TM, Chan TO, Andres DA, Cox AD.
    Oncogene; 2000 Sep 28; 19(41):4685-94. PubMed ID: 11032018
    [Abstract] [Full Text] [Related]

  • 10. H-ras and raf-1 cooperate in transformation of NIH3T3 fibroblasts.
    Cuadrado A, Bruder JT, Heidaran MA, App H, Rapp UR, Aaronson SA.
    Oncogene; 1993 Sep 28; 8(9):2443-8. PubMed ID: 8361757
    [Abstract] [Full Text] [Related]

  • 11. Involvement of PI3K in PKCepsilon-mediated oncogenic signal in rat colonic epithelial cells.
    Marras E, Concari P, Cortellezzi L, Dondi D, De Eguileor M, Perletti G.
    Int J Oncol; 2001 Aug 28; 19(2):395-9. PubMed ID: 11445858
    [Abstract] [Full Text] [Related]

  • 12. Role of diacylglycerol-regulated protein kinase C isotypes in growth factor activation of the Raf-1 protein kinase.
    Cai H, Smola U, Wixler V, Eisenmann-Tappe I, Diaz-Meco MT, Moscat J, Rapp U, Cooper GM.
    Mol Cell Biol; 1997 Feb 28; 17(2):732-41. PubMed ID: 9001227
    [Abstract] [Full Text] [Related]

  • 13. Protein kinase C alpha activates RAF-1 by direct phosphorylation.
    Kolch W, Heidecker G, Kochs G, Hummel R, Vahidi H, Mischak H, Finkenzeller G, Marmé D, Rapp UR.
    Nature; 1993 Jul 15; 364(6434):249-52. PubMed ID: 8321321
    [Abstract] [Full Text] [Related]

  • 14. Transforming growth factor-beta1-induced activation of the Raf-MEK-MAPK signaling pathway in rat lung fibroblasts via a PKC-dependent mechanism.
    Axmann A, Seidel D, Reimann T, Hempel U, Wenzel KW.
    Biochem Biophys Res Commun; 1998 Aug 19; 249(2):456-60. PubMed ID: 9712718
    [Abstract] [Full Text] [Related]

  • 15. Carbon source-dependent regulation of cell growth by murine protein kinase C epsilon expression in Saccharomyces cerevisiae.
    Parissenti AM, Villeneuve D, Kirwan-Rhude A, Busch D.
    J Cell Physiol; 1999 Feb 19; 178(2):216-26. PubMed ID: 10048586
    [Abstract] [Full Text] [Related]

  • 16. Raf revertant cells resist transformation by non-nuclear oncogenes and are deficient in the induction of early response genes by TPA and serum.
    Kolch W, Heidecker G, Troppmair J, Yanagihara K, Bassin RH, Rapp UR.
    Oncogene; 1993 Feb 19; 8(2):361-70. PubMed ID: 8426742
    [Abstract] [Full Text] [Related]

  • 17. Src tyrosine kinase mediates stimulation of Raf-1 and mitogen-activated protein kinase by the tumor promoter thapsigargin.
    Chao TS, Abe M, Hershenson MB, Gomes I, Rosner MR.
    Cancer Res; 1997 Aug 01; 57(15):3168-73. PubMed ID: 9242445
    [Abstract] [Full Text] [Related]

  • 18. Ornithine decarboxylase induction in transformation by H-Ras and RhoA.
    Shantz LM, Pegg AE.
    Cancer Res; 1998 Jul 01; 58(13):2748-53. PubMed ID: 9661886
    [Abstract] [Full Text] [Related]

  • 19. Synergism between two growth regulatory pathways: cooperative transformation of NIH3T3 cells by G alpha 12 and c-raf-1.
    Zhang Y, Saez R, Leal MA, Chan AM.
    Oncogene; 1996 Jun 06; 12(11):2377-83. PubMed ID: 8649778
    [Abstract] [Full Text] [Related]

  • 20. Raf-1 protein kinase is required for growth of induced NIH/3T3 cells.
    Kolch W, Heidecker G, Lloyd P, Rapp UR.
    Nature; 1991 Jan 31; 349(6308):426-8. PubMed ID: 1992343
    [Abstract] [Full Text] [Related]

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