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Journal Abstract Search

74 related items for PubMed ID: 9105763

  • 1. Expression of thromboxane synthase in kidney tissues from patients with IgA nephropathy.
    Endoh M, Kashem A, Yamauchi F, Yano N, Nomoto Y, Sakai H, Kurokawa K.
    Clin Nephrol; 1997 Mar; 47(3):168-75. PubMed ID: 9105763
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  • 3. Decreased synthesis of glomerular adrenomedullin in patients with IgA nephropathy.
    Kuo MC, Kuo HT, Chiu YW, Chang JM, Guh JY, Lai YH, Chen HC.
    J Lab Clin Med; 2005 May; 145(5):233-8. PubMed ID: 15902095
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  • 4. Anti-macrophage migration inhibitory factor reduces transforming growth factor-beta 1 expression in experimental IgA nephropathy.
    Leung JC, Chan LY, Tsang AW, Liu EW, Lam MF, Tang SC, Lai KN.
    Nephrol Dial Transplant; 2004 Aug; 19(8):1976-85. PubMed ID: 15187193
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  • 5. External suppression causes the low expression of the Cosmc gene in IgA nephropathy.
    Qin W, Zhong X, Fan JM, Zhang YJ, Liu XR, Ma XY.
    Nephrol Dial Transplant; 2008 May; 23(5):1608-14. PubMed ID: 18202089
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  • 6. CC chemokines and chemokine receptors in IgA nephropathy (IgAN) and in non-IgA mesangial proliferative glomerulonephritis (MesProGN). the immunohistochemical comparative study.
    Wagrowska-Danilewicz M, Danilewicz M, Stasikowska O.
    Pol J Pathol; 2005 May; 56(3):121-6. PubMed ID: 16334979
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  • 7. Upregulation of thromboxane synthase in human colorectal carcinoma and the cancer cell proliferation by thromboxane A2.
    Sakai H, Suzuki T, Takahashi Y, Ukai M, Tauchi K, Fujii T, Horikawa N, Minamimura T, Tabuchi Y, Morii M, Tsukada K, Takeguchi N.
    FEBS Lett; 2006 Jun 12; 580(14):3368-74. PubMed ID: 16709411
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  • 9. Glomerular angiotensin-converting enzyme 2 in pediatric IgA nephropathy.
    Urushihara M, Seki Y, Tayama T, Nagai T, Kinoshita Y, Jamba A, Kondo S, Kagami S.
    Am J Nephrol; 2013 Jun 12; 38(5):355-67. PubMed ID: 24158104
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  • 11. Alternate splicing of human thromboxane synthase mRNA.
    Wang LH, Tazawa R, Lang AQ, Wu KK.
    Arch Biochem Biophys; 1994 Dec 12; 315(2):273-8. PubMed ID: 7986068
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  • 12. Latent IgA deposition from donor kidney is the major risk factor for recurrent IgA nephropathy in renal transplantation.
    Moriyama T, Nitta K, Suzuki K, Honda K, Horita S, Uchida K, Yumura W, Tanabe K, Toma H, Nihei H, Yamaguchi Y.
    Clin Transplant; 2005 Dec 12; 19 Suppl 14():41-8. PubMed ID: 15955168
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  • 15. Immunoexpression of podocyte-associated proteins in acquired human glomerulopathies with nephrotic syndrome.
    Wagrowska-Danilewicz M, Stasikowska O, Danilewicz M.
    Pol J Pathol; 2006 Dec 12; 57(1):17-21. PubMed ID: 16739878
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  • 16. The role of alpha-smooth muscle actin and platelet-derived growth factor-beta receptor in the progression of renal damage in human IgA nephropathy.
    Ranieri E, Gesualdo L, Grandaliano G, Maiorano E, Schena FP.
    J Nephrol; 2001 Dec 12; 14(4):253-62. PubMed ID: 11506247
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  • 17. Pattern of renal beta 1(alpha 1-alpha 6) integrins distribution in IgA nephropathy and Henoch-Schoenlein nephritis.
    Wagrowska-Danilewicz M, Danilewicz M.
    Pol J Pathol; 2002 Dec 12; 53(2):51-7. PubMed ID: 12140867
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  • 18. Altered mRNA expression in renal biopsy tissue from patients with IgA nephropathy.
    Waga I, Yamamoto J, Sasai H, Munger WE, Hogan SL, Preston GA, Sun HW, Jennette JC, Falk RJ, Alcorta DA.
    Kidney Int; 2003 Oct 12; 64(4):1253-64. PubMed ID: 12969143
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  • 19. The level of serum secretory IgA of patients with IgA nephropathy is elevated and associated with pathological phenotypes.
    Zhang JJ, Xu LX, Liu G, Zhao MH, Wang HY.
    Nephrol Dial Transplant; 2008 Jan 12; 23(1):207-12. PubMed ID: 17938148
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