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Journal Abstract Search

366 related items for PubMed ID: 9115981

  • 1. Importance of two adjacent C-terminal sequences of SNAP-25 in exocytosis from intact and permeabilized chromaffin cells revealed by inhibition with botulinum neurotoxins A and E.
    Lawrence GW, Foran P, Mohammed N, DasGupta BR, Dolly JO.
    Biochemistry; 1997 Mar 18; 36(11):3061-7. PubMed ID: 9115981
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  • 2. Distinct exocytotic responses of intact and permeabilised chromaffin cells after cleavage of the 25-kDa synaptosomal-associated protein (SNAP-25) or synaptobrevin by botulinum toxin A or B.
    Lawrence GW, Foran P, Dolly JO.
    Eur J Biochem; 1996 Mar 15; 236(3):877-86. PubMed ID: 8665909
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  • 3. Botulinum neurotoxin C1 cleaves both syntaxin and SNAP-25 in intact and permeabilized chromaffin cells: correlation with its blockade of catecholamine release.
    Foran P, Lawrence GW, Shone CC, Foster KA, Dolly JO.
    Biochemistry; 1996 Feb 27; 35(8):2630-6. PubMed ID: 8611567
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  • 6. SNAP-25 is required for a late postdocking step in Ca2+-dependent exocytosis.
    Banerjee A, Kowalchyk JA, DasGupta BR, Martin TF.
    J Biol Chem; 1996 Aug 23; 271(34):20227-30. PubMed ID: 8702751
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  • 7. Dissociation of SNAP-25 and VAMP-2 by MgATP in permeabilized adrenal chromaffin cells.
    Misonou H, Nishiki T, Sekiguchi M, Takahashi M, Kamata Y, Kozaki S, Ohara-Imaizumi M, Kumakura K.
    Brain Res; 1996 Oct 21; 737(1-2):351-5. PubMed ID: 8930391
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  • 10. Novel therapeutics based on recombinant botulinum neurotoxins to normalize the release of transmitters and pain mediators.
    Dolly JO, Wang J, Zurawski TH, Meng J.
    FEBS J; 2011 Dec 21; 278(23):4454-66. PubMed ID: 21645262
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  • 11. Proteolysis of SNAP-25 isoforms by botulinum neurotoxin types A, C, and E: domains and amino acid residues controlling the formation of enzyme-substrate complexes and cleavage.
    Vaidyanathan VV, Yoshino K, Jahnz M, Dörries C, Bade S, Nauenburg S, Niemann H, Binz T.
    J Neurochem; 1999 Jan 21; 72(1):327-37. PubMed ID: 9886085
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  • 14. Fusion of Golgi-derived vesicles mediated by SNAP-25 is essential for sympathetic neuron outgrowth but relatively insensitive to botulinum neurotoxins in vitro.
    Lawrence GW, Wang J, Brin MF, Aoki KR, Wheeler L, Dolly JO.
    FEBS J; 2014 Jul 21; 281(14):3243-60. PubMed ID: 24863955
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  • 15. A peptide that mimics the C-terminal sequence of SNAP-25 inhibits secretory vesicle docking in chromaffin cells.
    Gutierrez LM, Viniegra S, Rueda J, Ferrer-Montiel AV, Canaves JM, Montal M.
    J Biol Chem; 1997 Jan 31; 272(5):2634-9. PubMed ID: 9006897
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  • 17. Presynaptic protein interactions in vivo: evidence from botulinum A, C, D and E action at frog neuromuscular junction.
    Raciborska DA, Trimble WS, Charlton MP.
    Eur J Neurosci; 1998 Aug 31; 10(8):2617-28. PubMed ID: 9767392
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  • 19. Enhancement of the endopeptidase activity of purified botulinum neurotoxins A and E by an isolated component of the native neurotoxin associated proteins.
    Sharma SK, Singh BR.
    Biochemistry; 2004 Apr 27; 43(16):4791-8. PubMed ID: 15096048
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  • 20. Persistence of botulinum neurotoxin action in cultured spinal cord cells.
    Keller JE, Neale EA, Oyler G, Adler M.
    FEBS Lett; 1999 Jul 30; 456(1):137-42. PubMed ID: 10452545
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