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154 related items for PubMed ID: 9162025

  • 1. The Jun kinase/stress-activated protein kinase pathway functions to regulate DNA repair and inhibition of the pathway sensitizes tumor cells to cisplatin.
    Potapova O, Haghighi A, Bost F, Liu C, Birrer MJ, Gjerset R, Mercola D.
    J Biol Chem; 1997 May 30; 272(22):14041-4. PubMed ID: 9162025
    [Abstract] [Full Text] [Related]

  • 2. Late activation of stress-activated protein kinases/c-Jun N-terminal kinases triggered by cisplatin-induced DNA damage in repair-defective cells.
    Helbig L, Damrot J, Hülsenbeck J, Köberle B, Brozovic A, Osmak M, Fiket Z, Kaina B, Fritz G.
    J Biol Chem; 2011 Apr 15; 286(15):12991-3001. PubMed ID: 21324906
    [Abstract] [Full Text] [Related]

  • 3. The activation of c-Jun NH2-terminal kinase (JNK) by DNA-damaging agents serves to promote drug resistance via activating transcription factor 2 (ATF2)-dependent enhanced DNA repair.
    Hayakawa J, Depatie C, Ohmichi M, Mercola D.
    J Biol Chem; 2003 Jun 06; 278(23):20582-92. PubMed ID: 12663670
    [Abstract] [Full Text] [Related]

  • 4. Cisplatin-induced activation of mitogen-activated protein kinases in ovarian carcinoma cells: inhibition of extracellular signal-regulated kinase activity increases sensitivity to cisplatin.
    Persons DL, Yazlovitskaya EM, Cui W, Pelling JC.
    Clin Cancer Res; 1999 May 06; 5(5):1007-14. PubMed ID: 10353733
    [Abstract] [Full Text] [Related]

  • 5. Inhibition of basal JNK activity by small interfering RNAs enhances cisplatin sensitivity and decreases DNA repair in T98G glioblastoma cells.
    Parra E, Gutiérrez L, Ferreira J.
    Oncol Rep; 2015 Jan 06; 33(1):413-8. PubMed ID: 25351964
    [Abstract] [Full Text] [Related]

  • 6. Differential induction of c-Jun NH2-terminal kinase and c-Abl kinase in DNA mismatch repair-proficient and -deficient cells exposed to cisplatin.
    Nehmé A, Baskaran R, Aebi S, Fink D, Nebel S, Cenni B, Wang JY, Howell SB, Christen RD.
    Cancer Res; 1997 Aug 01; 57(15):3253-7. PubMed ID: 9242457
    [Abstract] [Full Text] [Related]

  • 7. Induction of JNK and c-Abl signalling by cisplatin and oxaliplatin in mismatch repair-proficient and -deficient cells.
    Nehmé A, Baskaran R, Nebel S, Fink D, Howell SB, Wang JY, Christen RD.
    Br J Cancer; 1999 Mar 01; 79(7-8):1104-10. PubMed ID: 10098743
    [Abstract] [Full Text] [Related]

  • 8. Inhibition of extracellular signal-regulated protein kinase or c-Jun N-terminal protein kinase cascade, differentially activated by cisplatin, sensitizes human ovarian cancer cell line.
    Hayakawa J, Ohmichi M, Kurachi H, Ikegami H, Kimura A, Matsuoka T, Jikihara H, Mercola D, Murata Y.
    J Biol Chem; 1999 Oct 29; 274(44):31648-54. PubMed ID: 10531373
    [Abstract] [Full Text] [Related]

  • 9. Long-term activation of SAPK/JNK, p38 kinase and fas-L expression by cisplatin is attenuated in human carcinoma cells that acquired drug resistance.
    Brozovic A, Fritz G, Christmann M, Zisowsky J, Jaehde U, Osmak M, Kaina B.
    Int J Cancer; 2004 Dec 20; 112(6):974-85. PubMed ID: 15386344
    [Abstract] [Full Text] [Related]

  • 10. Regulation of platinum-compound cytotoxicity by the c-Jun N-terminal kinase and c-Jun signaling pathway in small-cell lung cancer cells.
    Levresse V, Marek L, Blumberg D, Heasley LE.
    Mol Pharmacol; 2002 Sep 20; 62(3):689-97. PubMed ID: 12181446
    [Abstract] [Full Text] [Related]

  • 11. The JUN kinase/stress-activated protein kinase pathway is required for epidermal growth factor stimulation of growth of human A549 lung carcinoma cells.
    Bost F, McKay R, Dean N, Mercola D.
    J Biol Chem; 1997 Dec 26; 272(52):33422-9. PubMed ID: 9407138
    [Abstract] [Full Text] [Related]

  • 12. The stress-activated protein kinase pathway mediates cell death following injury induced by cis-platinum, UV irradiation or heat.
    Zanke BW, Boudreau K, Rubie E, Winnett E, Tibbles LA, Zon L, Kyriakis J, Liu FF, Woodgett JR.
    Curr Biol; 1996 May 01; 6(5):606-13. PubMed ID: 8805279
    [Abstract] [Full Text] [Related]

  • 13. Distinct regulation of osmoprotective genes in yeast and mammals. Aldose reductase osmotic response element is induced independent of p38 and stress-activated protein kinase/Jun N-terminal kinase in rabbit kidney cells.
    Kültz D, Garcia-Perez A, Ferraris JD, Burg MB.
    J Biol Chem; 1997 May 16; 272(20):13165-70. PubMed ID: 9148932
    [Abstract] [Full Text] [Related]

  • 14. Microtubule-interfering agents activate c-Jun N-terminal kinase/stress-activated protein kinase through both Ras and apoptosis signal-regulating kinase pathways.
    Wang TH, Wang HS, Ichijo H, Giannakakou P, Foster JS, Fojo T, Wimalasena J.
    J Biol Chem; 1998 Feb 27; 273(9):4928-36. PubMed ID: 9478937
    [Abstract] [Full Text] [Related]

  • 15. Microtubule dysfunction induced by paclitaxel initiates apoptosis through both c-Jun N-terminal kinase (JNK)-dependent and -independent pathways in ovarian cancer cells.
    Wang TH, Popp DM, Wang HS, Saitoh M, Mural JG, Henley DC, Ichijo H, Wimalasena J.
    J Biol Chem; 1999 Mar 19; 274(12):8208-16. PubMed ID: 10075725
    [Abstract] [Full Text] [Related]

  • 16. Ligation of membrane IgM stimulates a novel c-Jun amino-terminal domain kinase activity in Daudi human B cells.
    Li X, Franklin CC, Kraft AS, Carter RH.
    Mol Immunol; 1997 Apr 19; 34(5):409-18. PubMed ID: 9293774
    [Abstract] [Full Text] [Related]

  • 17. Mutational status of the p53 gene modulates the basal level of jun N-terminal kinase and its inducibility by ultraviolet irradiation in A1-5 rat fibroblasts.
    Ramaswamy NT, Pelling JC.
    Mol Carcinog; 1999 Aug 19; 25(4):262-72. PubMed ID: 10449033
    [Abstract] [Full Text] [Related]

  • 18. Activation of stress-activated protein kinase/c-Jun NH2-terminal kinase and p38 kinase in calphostin C-induced apoptosis requires caspase-3-like proteases but is dispensable for cell death.
    Ozaki I, Tani E, Ikemoto H, Kitagawa H, Fujikawa H.
    J Biol Chem; 1999 Feb 26; 274(9):5310-7. PubMed ID: 10026138
    [Abstract] [Full Text] [Related]

  • 19. Lack of c-Jun activity increases survival to cisplatin.
    Sánchez-Pérez I, Perona R.
    FEBS Lett; 1999 Jun 18; 453(1-2):151-8. PubMed ID: 10403393
    [Abstract] [Full Text] [Related]

  • 20. c-Jun N-terminal kinase is essential for growth of human T98G glioblastoma cells.
    Potapova O, Gorospe M, Bost F, Dean NM, Gaarde WA, Mercola D, Holbrook NJ.
    J Biol Chem; 2000 Aug 11; 275(32):24767-75. PubMed ID: 10825181
    [Abstract] [Full Text] [Related]

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