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Journal Abstract Search

250 related items for PubMed ID: 9186478

  • 1. Hexokinase inactivation induced by ascorbic acid/Fe(II) in rabbit erythrocytes is independent of glutathione-reductive processes and appears to be mediated by dehydroascorbic acid.
    Fiorani M, De Sanctis R, Saltarelli R, Stocchi V.
    Arch Biochem Biophys; 1997 Jun 15; 342(2):191-6. PubMed ID: 9186478
    [Abstract] [Full Text] [Related]

  • 2. Role of dehydroascorbate in rabbit erythrocyte hexokinase inactivation induced by ascorbic acid/Fe(II).
    Fiorani M, Saltarelli R, De Sanctis R, Palma F, Ceccaroli P, Stocchi V.
    Arch Biochem Biophys; 1996 Oct 15; 334(2):357-61. PubMed ID: 8900411
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  • 3. Substrates of hexokinase, glucose-6-phosphate dehydrogenase, and glyceraldehyde-3-phosphate dehydrogenase prevent the inhibitory response induced by ascorbic acid/iron and dehydroascorbic acid in rabbit erythrocytes.
    Fiorani M, De Sanctis R, Scarlatti F, Stocchi V.
    Arch Biochem Biophys; 1998 Aug 15; 356(2):159-66. PubMed ID: 9705206
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  • 4. Inactivation of rabbit red blood cell hexokinase activity promoted in vitro by an oxygen-radical-generating system.
    Stocchi V, Biagiarelli B, Fiorani M, Palma F, Piccoli G, Cucchiarini L, Dachà M.
    Arch Biochem Biophys; 1994 May 15; 311(1):160-7. PubMed ID: 8185313
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  • 9. Free radicals promote "in vitro" a different intracellular decay of rabbit reticulocyte and erythrocyte glycolytic enzymes.
    Stocchi V, Biagiarelli B, Masat L, Palma F, Palma F, Piccoli G, Cucchiarini L, Magnani M.
    Adv Exp Med Biol; 1991 May 15; 307():217-23. PubMed ID: 1805588
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  • 10. Physiological role of dehydroascorbic acid.
    Banerjee S.
    Indian J Physiol Pharmacol; 1977 May 15; 21(2):85-93. PubMed ID: 407155
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  • 11. Loss of glutathione, ascorbate recycling, and free radical scavenging in human erythrocytes exposed to filtered cigarette smoke.
    Maranzana A, Mehlhorn RJ.
    Arch Biochem Biophys; 1998 Feb 15; 350(2):169-82. PubMed ID: 9473290
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  • 12. Antiviral effects of ascorbic and dehydroascorbic acids in vitro.
    Furuya A, Uozaki M, Yamasaki H, Arakawa T, Arita M, Koyama AH.
    Int J Mol Med; 2008 Oct 15; 22(4):541-5. PubMed ID: 18813862
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  • 13. Ascorbic acid oxidation by hydrogen peroxide.
    Deutsch JC.
    Anal Biochem; 1998 Jan 01; 255(1):1-7. PubMed ID: 9448835
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  • 14. UVA irradiation of human lens proteins produces residual oxidation of ascorbic acid even in the presence of high levels of glutathione.
    Ortwerth BJ, Coots A, James HL, Linetsky M.
    Arch Biochem Biophys; 1998 Mar 15; 351(2):189-96. PubMed ID: 9515056
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  • 15. Dehydroascorbic acid prevents oxidative cell death through a glutathione pathway in primary astrocytes.
    Kim EJ, Park YG, Baik EJ, Jung SJ, Won R, Nahm TS, Lee BH.
    J Neurosci Res; 2005 Mar 01; 79(5):670-9. PubMed ID: 15668957
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  • 16. Action of oxidized and reduced glutathione on rabbit red blood cell hexokinase.
    Magnani M, Stocchi V, Ninfali P, Dachà M, Fornaini G.
    Biochim Biophys Acta; 1980 Sep 09; 615(1):113-20. PubMed ID: 6968593
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  • 17. [On the mechanism of ascorbic acid induced methemoglobin reduction of human erythrocytes (author's transl)].
    Waller HD, Benöhr HC, Tigges FJ.
    Klin Wochenschr; 1977 Oct 01; 55(19):955-64. PubMed ID: 926709
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  • 19. Unequivocal evidence in support of the nonenzymatic redox coupling between glutathione/glutathione disulfide and ascorbic acid/dehydroascorbic acid.
    Winkler BS.
    Biochim Biophys Acta; 1992 Oct 27; 1117(3):287-90. PubMed ID: 1420278
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  • 20. Enzymatic basis for altered ascorbic acid and dehydroascorbic acid levels in diabetes.
    Bode AM, Yavarow CR, Fry DA, Vargas T.
    Biochem Biophys Res Commun; 1993 Mar 31; 191(3):1347-53. PubMed ID: 8466510
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