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Journal Abstract Search

103 related items for PubMed ID: 9201808

  • 21. Activation of 5-HT1B receptors in the nucleus accumbens reduces amphetamine-induced enhancement of responding for conditioned reward.
    Fletcher PJ, Korth KM.
    Psychopharmacology (Berl); 1999 Feb; 142(2):165-74. PubMed ID: 10102769
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  • 22. Dissociable hippocampal and amygdalar D1-like receptor contribution to discriminated Pavlovian conditioned approach learning.
    Andrzejewski ME, Ryals C.
    Behav Brain Res; 2016 Feb 15; 299():111-21. PubMed ID: 26632336
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  • 25. The D1 agonist SKF 38393 attenuates amphetamine-produced enhancement of responding for conditioned reward in rats.
    Ranaldi R, Pantalony D, Beninger RJ.
    Pharmacol Biochem Behav; 1995 Sep 15; 52(1):131-7. PubMed ID: 7501655
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  • 26. 6-Hydroxydopamine lesions of the nucleus accumbens, but not of the caudate nucleus, attenuate enhanced responding with reward-related stimuli produced by intra-accumbens d-amphetamine.
    Taylor JR, Robbins TW.
    Psychopharmacology (Berl); 1986 Sep 15; 90(3):390-7. PubMed ID: 3097729
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  • 27. Oxytocin injected into the ventral subiculum or the posteromedial cortical nucleus of the amygdala induces penile erection and increases extracellular dopamine levels in the nucleus accumbens of male rats.
    Melis MR, Succu S, Sanna F, Boi A, Argiolas A.
    Eur J Neurosci; 2009 Oct 15; 30(7):1349-57. PubMed ID: 19769589
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  • 28. Enhanced appetitive conditioning following repeated pretreatment with d-amphetamine.
    Harmer CJ, Phillips GD.
    Behav Pharmacol; 1998 Jul 15; 9(4):299-308. PubMed ID: 10065918
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  • 29. Post-training intra-amygdala amphetamine injections given during acquisition of a stimulus-response (S-R) habit task enhance the expression of stimulus-reward learning: further evidence for incidental amygdala learning.
    Holahan MR, Hong NS, Chan C, McDonald RJ.
    Brain Res Bull; 2005 Aug 15; 66(3):222-8. PubMed ID: 16023919
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  • 30. Dopamine D1 or D2 receptor antagonism within the basolateral amygdala differentially alters the acquisition of cocaine-cue associations necessary for cue-induced reinstatement of cocaine-seeking.
    Berglind WJ, Case JM, Parker MP, Fuchs RA, See RE.
    Neuroscience; 2006 Aug 15; 137(2):699-706. PubMed ID: 16289883
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  • 31. The role of different subregions of the basolateral amygdala in cue-induced reinstatement and extinction of food-seeking behavior.
    McLaughlin RJ, Floresco SB.
    Neuroscience; 2007 Jun 08; 146(4):1484-94. PubMed ID: 17449185
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  • 34. Enhanced dopamine efflux in the amygdala by a predictive, but not a non-predictive, stimulus: facilitation by prior repeated D-amphetamine.
    Harmer CJ, Phillips GD.
    Neuroscience; 1999 Apr 08; 90(1):119-30. PubMed ID: 10188939
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  • 35. The ventral subiculum modulation of prepulse inhibition is not mediated via dopamine D2 or nucleus accumbens non-NMDA glutamate receptor activity.
    Wan FJ, Caine SB, Swerdlow NR.
    Eur J Pharmacol; 1996 Oct 24; 314(1-2):9-18. PubMed ID: 8957213
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  • 37. The effects of amphetamine, apomorphine, SKF 38393, quinpirole and bromocriptine on responding for conditioned reward in rats.
    Beninger RJ, Ranaldi R.
    Behav Pharmacol; 1992 Apr 24; 3(2):155-163. PubMed ID: 11224114
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  • 40. Involvement of the nucleus accumbens and medial prefrontal cortex in the expression of conditioned hyperactivity to a cocaine-associated environment in rats.
    Franklin TR, Druhan JP.
    Neuropsychopharmacology; 2000 Dec 24; 23(6):633-44. PubMed ID: 11063919
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