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Journal Abstract Search

380 related items for PubMed ID: 9202234

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  • 3. Fos and Jun inhibit estrogen-induced transcription of the human progesterone receptor gene through an activator protein-1 site.
    Petz LN, Ziegler YS, Schultz JR, Nardulli AM.
    Mol Endocrinol; 2004 Mar; 18(3):521-32. PubMed ID: 14684847
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  • 4. Occupancy and composition of proteins bound to the AP-1 sites in the glucocorticoid receptor and c-jun promoters after glucocorticoid treatment and in different cell types.
    Barrett TJ, Vedeckis WV.
    Recept Signal Transduct; 1996 Mar; 6(3-4):179-93. PubMed ID: 9259052
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  • 5. An estrogen receptor-alpha splicing variant mediates both positive and negative effects on gene transcription.
    Bollig A, Miksicek RJ.
    Mol Endocrinol; 2000 May; 14(5):634-49. PubMed ID: 10809228
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  • 6. Transrepression of c-jun gene expression by the glucocorticoid receptor requires both AP-1 sites in the c-jun promoter.
    Wei P, Inamdar N, Vedeckis WV.
    Mol Endocrinol; 1998 Sep; 12(9):1322-33. PubMed ID: 9731701
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  • 7. Estrogen receptor alpha and activating protein-1 mediate estrogen responsiveness of the progesterone receptor gene in MCF-7 breast cancer cells.
    Petz LN, Ziegler YS, Loven MA, Nardulli AM.
    Endocrinology; 2002 Dec; 143(12):4583-91. PubMed ID: 12446585
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  • 8. Estradiol increases and anti-estrogens antagonize the growth factor-induced activator protein-1 activity in MCF7 breast cancer cells without affecting c-fos and c-jun synthesis.
    Philips A, Chalbos D, Rochefort H.
    J Biol Chem; 1993 Jul 05; 268(19):14103-8. PubMed ID: 8314777
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  • 9. The inhibitory activity of a transdominant c-jun mutant fused to the ligand binding domain of the estrogen receptor.
    Kim S, Brown PH, Birrer MJ.
    Oncogene; 1996 Mar 07; 12(5):1043-53. PubMed ID: 8649795
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  • 12. Receptor-interacting protein 140 binds c-Jun and inhibits estradiol-induced activator protein-1 activity by reversing glucocorticoid receptor-interacting protein 1 effect.
    Teyssier C, Belguise K, Galtier F, Cavailles V, Chalbos D.
    Mol Endocrinol; 2003 Feb 07; 17(2):287-99. PubMed ID: 12554755
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  • 13. Estrogen receptor alpha, fos-related antigen-2, and c-Jun coordinately regulate human UDP glucuronosyltransferase 2B15 and 2B17 expression in response to 17beta-estradiol in MCF-7 cells.
    Hu DG, Mackenzie PI.
    Mol Pharmacol; 2009 Aug 07; 76(2):425-39. PubMed ID: 19487245
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  • 14. Synergistic activation of neurotensin/neuromedin N gene expression by c-Jun and glucocorticoids: novel effects of Fos family proteins.
    Harrison RJ, McNeil GP, Dobner PR.
    Mol Endocrinol; 1995 Aug 07; 9(8):981-93. PubMed ID: 7476995
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  • 16. Glucocorticoid regulation of c-fos, c-jun and transcription factor AP-1 in the AtT-20 corticotrope cell.
    Autelitano DJ.
    J Neuroendocrinol; 1994 Dec 07; 6(6):627-37. PubMed ID: 7894465
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  • 17. A c-fos/Estrogen receptor fusion protein promotes cell cycle progression and proliferation of human cancer cell lines.
    Crowe DL, Brown TN, Kim R, Smith SM, Lee MK.
    Mol Cell Biol Res Commun; 2000 Apr 07; 3(4):243-8. PubMed ID: 10891399
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  • 18. Cross talk between glucocorticoid and estrogen receptors occurs at a subset of proinflammatory genes.
    Cvoro A, Yuan C, Paruthiyil S, Miller OH, Yamamoto KR, Leitman DC.
    J Immunol; 2011 Apr 01; 186(7):4354-60. PubMed ID: 21357268
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  • 20. Glucocorticoid regulation of alkaline phosphatase, osteocalcin, and proto-oncogenes in normal human osteoblast-like cells.
    Subramaniam M, Colvard D, Keeting PE, Rasmussen K, Riggs BL, Spelsberg TC.
    J Cell Biochem; 1992 Dec 01; 50(4):411-24. PubMed ID: 1469072
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