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Journal Abstract Search

244 related items for PubMed ID: 9220023

  • 1. Specificity of transcription enhancement via the STAT responsive element in the serine protease inhibitor 2.1 promoter.
    Wood TJ, Sliva D, Lobie PE, Goullieux F, Mui AL, Groner B, Norstedt G, Haldosén LA.
    Mol Cell Endocrinol; 1997 Jun 20; 130(1-2):69-81. PubMed ID: 9220023
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  • 4. Stimulation of endogenous GH and interleukin-6 receptors selectively activates different Jaks and Stats, with a Stat5 specific synergistic effect of dexamethasone.
    von Laue S, Finidori J, Maamra M, Shen XY, Justice S, Dobson PR, Ross RJ.
    J Endocrinol; 2000 May 20; 165(2):301-11. PubMed ID: 10810294
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  • 5. Prolactin, growth hormone, erythropoietin and granulocyte-macrophage colony stimulating factor induce MGF-Stat5 DNA binding activity.
    Gouilleux F, Pallard C, Dusanter-Fourt I, Wakao H, Haldosen LA, Norstedt G, Levy D, Groner B.
    EMBO J; 1995 May 01; 14(9):2005-13. PubMed ID: 7744007
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  • 6. Prolactin induces phosphorylation of Tyr694 of Stat5 (MGF), a prerequisite for DNA binding and induction of transcription.
    Gouilleux F, Wakao H, Mundt M, Groner B.
    EMBO J; 1994 Sep 15; 13(18):4361-9. PubMed ID: 7925280
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  • 7. The last proline of Box 1 is essential for association with JAK2 and functional activation of the prolactin receptor.
    Pezet A, Buteau H, Kelly PA, Edery M.
    Mol Cell Endocrinol; 1997 May 16; 129(2):199-208. PubMed ID: 9202403
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  • 8. Interactions among Janus kinases and the prolactin (PRL) receptor in the regulation of a PRL response element.
    Gao J, Hughes JP, Auperin B, Buteau H, Edery M, Zhuang H, Wojchowski DM, Horseman ND.
    Mol Endocrinol; 1996 Jul 16; 10(7):847-56. PubMed ID: 8813725
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  • 9. The role of the growth hormone (GH) receptor and JAK1 and JAK2 kinases in the activation of Stats 1, 3, and 5 by GH.
    Smit LS, Meyer DJ, Billestrup N, Norstedt G, Schwartz J, Carter-Su C.
    Mol Endocrinol; 1996 May 16; 10(5):519-33. PubMed ID: 8732683
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  • 10. Dominant negative variants of the SHP-2 tyrosine phosphatase inhibit prolactin activation of Jak2 (janus kinase 2) and induction of Stat5 (signal transducer and activator of transcription 5)-dependent transcription.
    Berchtold S, Volarevic S, Moriggl R, Mercep M, Groner B.
    Mol Endocrinol; 1998 Apr 16; 12(4):556-67. PubMed ID: 9544991
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  • 17. Desensitization of the growth hormone-induced Janus kinase 2 (Jak 2)/signal transducer and activator of transcription 5 (Stat5)-signaling pathway requires protein synthesis and phospholipase C.
    Fernández L, Flores-Morales A, Lahuna O, Sliva D, Norstedt G, Haldosén LA, Mode A, Gustafsson JA.
    Endocrinology; 1998 Apr 16; 139(4):1815-24. PubMed ID: 9528967
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  • 18. Epo-induced hemoglobinization of SKT6 cells is mediated by minimal cytoplasmic domains of the Epo or prolactin receptors without modulation of GATA-1 or EKLF.
    Reese TT, Gregory RC, Sharlow ER, Pacifici RE, Crouse JA, Todokoro K, Wojchowski DM.
    Growth Factors; 1997 Apr 16; 14(2-3):161-76. PubMed ID: 9255607
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  • 19. Dissociation of Janus kinase 2 and signal transducer and activator of transcription 5 activation after treatment of Nb2 cells with a molecular mimic of phosphorylated prolactin.
    Coss D, Kuo CB, Yang L, Ingleton P, Luben R, Walker AM.
    Endocrinology; 1999 Nov 16; 140(11):5087-94. PubMed ID: 10537136
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  • 20. Activation of the Jak/Stat signal transduction pathway in GH-treated rat osteoblast-like cells in culture.
    Gerland K, Bataillé-Simoneau N, Baslé M, Fourcin M, Gascan H, Mercier L.
    Mol Cell Endocrinol; 2000 Oct 25; 168(1-2):1-9. PubMed ID: 11064147
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