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Journal Abstract Search


351 related items for PubMed ID: 9259311

  • 1. CDK2 is a target for retinoic acid-mediated growth inhibition in MCF-7 human breast cancer cells.
    Teixeira C, Pratt MA.
    Mol Endocrinol; 1997 Aug; 11(9):1191-202. PubMed ID: 9259311
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  • 2. Multifaceted regulation of cell cycle progression by estrogen: regulation of Cdk inhibitors and Cdc25A independent of cyclin D1-Cdk4 function.
    Foster JS, Henley DC, Bukovsky A, Seth P, Wimalasena J.
    Mol Cell Biol; 2001 Feb; 21(3):794-810. PubMed ID: 11154267
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  • 3. Inhibition of the melanoma cell cycle and regulation at the G1/S transition by 12-O-tetradecanoylphorbol-13-acetate (TPA) by modulation of CDK2 activity.
    Coppock DL, Buffolino P, Kopman C, Nathanson L.
    Exp Cell Res; 1995 Nov; 221(1):92-102. PubMed ID: 7589260
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  • 6. Induction of G1 phase arrest in MCF human breast cancer cells by pentagalloylglucose through the down-regulation of CDK4 and CDK2 activities and up-regulation of the CDK inhibitors p27(Kip) and p21(Cip).
    Chen WJ, Chang CY, Lin JK.
    Biochem Pharmacol; 2003 Jun 01; 65(11):1777-85. PubMed ID: 12781329
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  • 9. Retinoic acid-mediated G1 arrest is associated with induction of p27(Kip1) and inhibition of cyclin-dependent kinase 3 in human lung squamous carcinoma CH27 cells.
    Hsu SL, Hsu JW, Liu MC, Chen LY, Chang CD.
    Exp Cell Res; 2000 Aug 01; 258(2):322-31. PubMed ID: 10896783
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  • 10. Retinoic acid-mediated growth arrest of EBV-immortalized B lymphocytes is associated with multiple changes in G1 regulatory proteins: p27Kip1 up-regulation is a relevant early event.
    Zancai P, Cariati R, Rizzo S, Boiocchi M, Dolcetti R.
    Oncogene; 1998 Oct 08; 17(14):1827-36. PubMed ID: 9778049
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  • 11. Aryl hydrocarbon receptor-mediated antiestrogenicity in MCF-7 cells: modulation of hormone-induced cell cycle enzymes.
    Wang W, Smith R, Safe S.
    Arch Biochem Biophys; 1998 Aug 15; 356(2):239-48. PubMed ID: 9705214
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  • 13. Disruption of the actin cytoskeleton leads to inhibition of mitogen-induced cyclin E expression, Cdk2 phosphorylation, and nuclear accumulation of the retinoblastoma protein-related p107 protein.
    Reshetnikova G, Barkan R, Popov B, Nikolsky N, Chang LS.
    Exp Cell Res; 2000 Aug 25; 259(1):35-53. PubMed ID: 10942577
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  • 14. Cell cycle exit during terminal erythroid differentiation is associated with accumulation of p27(Kip1) and inactivation of cdk2 kinase.
    Hsieh FF, Barnett LA, Green WF, Freedman K, Matushansky I, Skoultchi AI, Kelley LL.
    Blood; 2000 Oct 15; 96(8):2746-54. PubMed ID: 11023508
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  • 15. Inhibition of cyclin-dependent kinases 2 and 4 activities as well as induction of Cdk inhibitors p21 and p27 during growth arrest of human breast carcinoma cells by (-)-epigallocatechin-3-gallate.
    Liang YC, Lin-Shiau SY, Chen CF, Lin JK.
    J Cell Biochem; 1999 Oct 01; 75(1):1-12. PubMed ID: 10462699
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  • 16. Involvement of p21 and p27 in the regulation of CDK activity and cell cycle progression in the regenerating liver.
    Albrecht JH, Poon RY, Ahonen CL, Rieland BM, Deng C, Crary GS.
    Oncogene; 1998 Apr 23; 16(16):2141-50. PubMed ID: 9572495
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  • 17. Antiestrogen inhibition of cell cycle progression in breast cancer cells in associated with inhibition of cyclin-dependent kinase activity and decreased retinoblastoma protein phosphorylation.
    Watts CK, Brady A, Sarcevic B, deFazio A, Musgrove EA, Sutherland RL.
    Mol Endocrinol; 1995 Dec 23; 9(12):1804-13. PubMed ID: 8614416
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