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131 related items for PubMed ID: 9284348

  • 1. Radiation-induced apoptosis in developing rats and kainic acid-induced excitotoxicity in adult rats are associated with distinctive morphological and biochemical c-Jun/AP-1 (N) expression.
    Ferrer I, Planas AM, Pozas E.
    Neuroscience; 1997 Sep; 80(2):449-58. PubMed ID: 9284348
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  • 2. Kainic acid-induced excitotoxicity is associated with a complex c-Fos and c-Jun response which does not preclude either cell death or survival.
    Pozas E, Ballabriga J, Planas AM, Ferrer I.
    J Neurobiol; 1997 Sep; 33(3):232-46. PubMed ID: 9298762
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  • 3. Strong c-Jun/AP-1 immunoreactivity is restricted to apoptotic cells following intracerebral ibotenic acid injection in developing rats.
    Ferrer I, Pozas E, Ballabriga J, Planas AM.
    Neurosci Res; 1997 May; 28(1):21-31. PubMed ID: 9179877
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  • 4. Localization and expression of Jun-like immunoreactivity in apoptotic neurons induced by colchicine administration in vivo and in vitro depends on the antisera used.
    Pozas E, Aguado F, Ferrer I.
    Acta Neuropathol; 1999 Aug; 98(2):119-28. PubMed ID: 10442550
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  • 7. Increase in Bcl-2 phosphorylation and reduced levels of BH3-only Bcl-2 family proteins in kainic acid-mediated neuronal death in the rat brain.
    Korhonen L, Belluardo N, Mudo G, Lindholm D.
    Eur J Neurosci; 2003 Sep; 18(5):1121-34. PubMed ID: 12956712
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  • 11. Cell death in the normal developing brain, and following ionizing radiation, methyl-azoxymethanol acetate, and hypoxia-ischaemia in the rat.
    Ferrer I.
    Neuropathol Appl Neurobiol; 1996 Dec; 22(6):489-94. PubMed ID: 9004234
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  • 12. Differential c-Fos and caspase expression following kainic acid excitotoxicity.
    Ferrer I, López E, Blanco R, Rivera R, Krupinski J, Martí E.
    Acta Neuropathol; 2000 Mar; 99(3):245-56. PubMed ID: 10663966
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  • 13. Specific association of c-Jun-like immunoreactivity but not c-Jun p39 with normal and induced programmed cell death in the chick embryo.
    Ayala V, Casas C, Ribera J, Calderó J, Oppenhiem RW, Esquerda JE.
    J Neurobiol; 1999 Feb 05; 38(2):171-90. PubMed ID: 10022565
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  • 14. Naturally occurring (programmed) and radiation-induced apoptosis are associated with selective c-Jun expression in the developing rat brain.
    Ferrer I, Olive M, Ribera J, Planas AM.
    Eur J Neurosci; 1996 Jun 05; 8(6):1286-98. PubMed ID: 8752598
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  • 15. Kainic acid-induced seizures produce necrotic, not apoptotic, neurons with internucleosomal DNA cleavage: implications for programmed cell death mechanisms.
    Fujikawa DG, Shinmei SS, Cai B.
    Neuroscience; 2000 Jun 05; 98(1):41-53. PubMed ID: 10858610
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  • 16. Expression of activating transcription factor-2, serum response factor and cAMP/Ca response element binding protein in the adult rat brain following generalized seizures, nerve fibre lesion and ultraviolet irradiation.
    Herdegen T, Blume A, Buschmann T, Georgakopoulos E, Winter C, Schmid W, Hsieh TF, Zimmermann M, Gass P.
    Neuroscience; 1997 Nov 05; 81(1):199-212. PubMed ID: 9300412
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  • 18. Dexamethasone attenuates kainate-induced AP-1 activation in rat brain.
    Unlap T, Jope RS.
    Brain Res Mol Brain Res; 1994 Jul 05; 24(1-4):275-82. PubMed ID: 7968367
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  • 19. Role of caspases in ionizing radiation-induced apoptosis in the developing cerebellum.
    Ferrer I.
    J Neurobiol; 1999 Dec 05; 41(4):549-58. PubMed ID: 10590178
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  • 20. Kainate-induced apoptosis correlates with c-Jun activation in cultured cerebellar granule cells.
    Cheung NS, Carroll FY, Larm JA, Beart PM, Giardina SF.
    J Neurosci Res; 1998 Apr 01; 52(1):69-82. PubMed ID: 9556030
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