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155 related items for PubMed ID: 9300654

  • 1. Transgenic models of Huntington's disease.
    Bates GP, Mangiarini L, Mahal A, Davies SW.
    Hum Mol Genet; 1997; 6(10):1633-7. PubMed ID: 9300654
    [Abstract] [Full Text] [Related]

  • 2. Instability of highly expanded CAG repeats in mice transgenic for the Huntington's disease mutation.
    Mangiarini L, Sathasivam K, Mahal A, Mott R, Seller M, Bates GP.
    Nat Genet; 1997 Feb; 15(2):197-200. PubMed ID: 9020849
    [Abstract] [Full Text] [Related]

  • 3. A CAG repeat threshold for therapeutics targeting somatic instability in Huntington's disease.
    Aldous SG, Smith EJ, Landles C, Osborne GF, Cañibano-Pico M, Nita IM, Phillips J, Zhang Y, Jin B, Hirst MB, Benn CL, Bond BC, Edelmann W, Greene JR, Bates GP.
    Brain; 2024 May 03; 147(5):1784-1798. PubMed ID: 38387080
    [Abstract] [Full Text] [Related]

  • 4. Transgenic mouse models of neurodegenerative disease caused by CAG/polyglutamine expansions.
    Bates GP, Davies SW.
    Mol Med Today; 1997 Nov 03; 3(11):508-15. PubMed ID: 9430787
    [Abstract] [Full Text] [Related]

  • 5. Transgenic mice expressing mutated full-length HD cDNA: a paradigm for locomotor changes and selective neuronal loss in Huntington's disease.
    Reddy PH, Charles V, Williams M, Miller G, Whetsell WO, Tagle DA.
    Philos Trans R Soc Lond B Biol Sci; 1999 Jun 29; 354(1386):1035-45. PubMed ID: 10434303
    [Abstract] [Full Text] [Related]

  • 6. Length-dependent gametic CAG repeat instability in the Huntington's disease knock-in mouse.
    Wheeler VC, Auerbach W, White JK, Srinidhi J, Auerbach A, Ryan A, Duyao MP, Vrbanac V, Weaver M, Gusella JF, Joyner AL, MacDonald ME.
    Hum Mol Genet; 1999 Jan 29; 8(1):115-22. PubMed ID: 9887339
    [Abstract] [Full Text] [Related]

  • 7. The use of transgenic and knock-in mice to study Huntington's disease.
    Hickey MA, Chesselet MF.
    Cytogenet Genome Res; 2003 Jan 29; 100(1-4):276-86. PubMed ID: 14526189
    [Abstract] [Full Text] [Related]

  • 8. Quantification of age-dependent somatic CAG repeat instability in Hdh CAG knock-in mice reveals different expansion dynamics in striatum and liver.
    Lee JM, Pinto RM, Gillis T, St Claire JC, Wheeler VC.
    PLoS One; 2011 Jan 29; 6(8):e23647. PubMed ID: 21897851
    [Abstract] [Full Text] [Related]

  • 9. Expanded CAG repeats in the murine Huntington's disease gene increases neuronal differentiation of embryonic and neural stem cells.
    Lorincz MT, Zawistowski VA.
    Mol Cell Neurosci; 2009 Jan 29; 40(1):1-13. PubMed ID: 18625318
    [Abstract] [Full Text] [Related]

  • 10. How does the Huntington's disease mutation damage cells?
    Rubinsztein DC.
    Sci Aging Knowledge Environ; 2003 Sep 17; 2003(37):PE26. PubMed ID: 13679594
    [Abstract] [Full Text] [Related]

  • 11. Partial resistance to malonate-induced striatal cell death in transgenic mouse models of Huntington's disease is dependent on age and CAG repeat length.
    Hansson O, Castilho RF, Korhonen L, Lindholm D, Bates GP, Brundin P.
    J Neurochem; 2001 Aug 17; 78(4):694-703. PubMed ID: 11520890
    [Abstract] [Full Text] [Related]

  • 12. Mouse models of Huntington's disease.
    Menalled LB, Chesselet MF.
    Trends Pharmacol Sci; 2002 Jan 17; 23(1):32-9. PubMed ID: 11804649
    [Abstract] [Full Text] [Related]

  • 13. Are neuronal intranuclear inclusions the common neuropathology of triplet-repeat disorders with polyglutamine-repeat expansions?
    Davies SW, Beardsall K, Turmaine M, DiFiglia M, Aronin N, Bates GP.
    Lancet; 1998 Jan 10; 351(9096):131-3. PubMed ID: 9439509
    [Abstract] [Full Text] [Related]

  • 14. Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice.
    Mangiarini L, Sathasivam K, Seller M, Cozens B, Harper A, Hetherington C, Lawton M, Trottier Y, Lehrach H, Davies SW, Bates GP.
    Cell; 1996 Nov 01; 87(3):493-506. PubMed ID: 8898202
    [Abstract] [Full Text] [Related]

  • 15. Dramatic mutation instability in HD mouse striatum: does polyglutamine load contribute to cell-specific vulnerability in Huntington's disease?
    Kennedy L, Shelbourne PF.
    Hum Mol Genet; 2000 Oct 12; 9(17):2539-44. PubMed ID: 11030759
    [Abstract] [Full Text] [Related]

  • 16. Parent-of-origin differences of mutant HTT CAG repeat instability in Huntington's disease.
    Aziz NA, van Belzen MJ, Coops ID, Belfroid RD, Roos RA.
    Eur J Med Genet; 2011 Oct 12; 54(4):e413-8. PubMed ID: 21540131
    [Abstract] [Full Text] [Related]

  • 17. Transgenic models of Huntington's disease.
    Sathasivam K, Hobbs C, Mangiarini L, Mahal A, Turmaine M, Doherty P, Davies SW, Bates GP.
    Philos Trans R Soc Lond B Biol Sci; 1999 Jun 29; 354(1386):963-9. PubMed ID: 10434294
    [Abstract] [Full Text] [Related]

  • 18. Loss of normal huntingtin function: new developments in Huntington's disease research.
    Cattaneo E, Rigamonti D, Goffredo D, Zuccato C, Squitieri F, Sipione S.
    Trends Neurosci; 2001 Mar 29; 24(3):182-8. PubMed ID: 11182459
    [Abstract] [Full Text] [Related]

  • 19. Lack of huntingtin promotes neural stem cells differentiation into glial cells while neurons expressing huntingtin with expanded polyglutamine tracts undergo cell death.
    Conforti P, Camnasio S, Mutti C, Valenza M, Thompson M, Fossale E, Zeitlin S, MacDonald ME, Zuccato C, Cattaneo E.
    Neurobiol Dis; 2013 Feb 29; 50():160-70. PubMed ID: 23089356
    [Abstract] [Full Text] [Related]

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