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Journal Abstract Search

209 related items for PubMed ID: 9302008

  • 1. Disruption of IcsP, the major Shigella protease that cleaves IcsA, accelerates actin-based motility.
    Shere KD, Sallustio S, Manessis A, D'Aversa TG, Goldberg MB.
    Mol Microbiol; 1997 Aug; 25(3):451-62. PubMed ID: 9302008
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  • 2. Actin-based motility is sufficient for bacterial membrane protrusion formation and host cell uptake.
    Monack DM, Theriot JA.
    Cell Microbiol; 2001 Sep; 3(9):633-47. PubMed ID: 11553015
    [Abstract] [Full Text] [Related]

  • 3. The unipolar Shigella surface protein IcsA is targeted directly to the bacterial old pole: IcsP cleavage of IcsA occurs over the entire bacterial surface.
    Steinhauer J, Agha R, Pham T, Varga AW, Goldberg MB.
    Mol Microbiol; 1999 Apr; 32(2):367-77. PubMed ID: 10231492
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  • 4. SopA, the outer membrane protease responsible for polar localization of IcsA in Shigella flexneri.
    Egile C, d'Hauteville H, Parsot C, Sansonetti PJ.
    Mol Microbiol; 1997 Mar; 23(5):1063-73. PubMed ID: 9076742
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  • 5. Functional analysis of a rickettsial OmpA homology domain of Shigella flexneri icsA.
    Charles M, Magdalena J, Theriot JA, Goldberg MB.
    J Bacteriol; 1999 Feb; 181(3):869-78. PubMed ID: 9922250
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  • 6. Genetic modulation of Shigella flexneri 2a lipopolysaccharide O antigen modal chain length reveals that it has been optimized for virulence.
    Morona R, Daniels C, Van Den Bosch L.
    Microbiology (Reading); 2003 Apr; 149(Pt 4):925-939. PubMed ID: 12686635
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  • 7. Outer membrane protein A (OmpA): a new player in shigella flexneri protrusion formation and inter-cellular spreading.
    Ambrosi C, Pompili M, Scribano D, Zagaglia C, Ripa S, Nicoletti M.
    PLoS One; 2012 Apr; 7(11):e49625. PubMed ID: 23166731
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  • 8. Effect of O side-chain length and composition on the virulence of Shigella flexneri 2a.
    Sandlin RC, Goldberg MB, Maurelli AT.
    Mol Microbiol; 1996 Oct; 22(1):63-73. PubMed ID: 8899709
    [Abstract] [Full Text] [Related]

  • 9. Shigella flexneri cell-to-cell spread, and growth and inflammation in mice, is limited by the outer membrane protease IcsP.
    Tran EN, Attridge SR, Teh MY, Morona R.
    FEMS Microbiol Lett; 2015 Jun; 362(12):fnv088. PubMed ID: 26025071
    [Abstract] [Full Text] [Related]

  • 10. LPS unmasking of Shigella flexneri reveals preferential localisation of tagged outer membrane protease IcsP to septa and new poles.
    Tran EN, Doyle MT, Morona R.
    PLoS One; 2013 Jun; 8(7):e70508. PubMed ID: 23936222
    [Abstract] [Full Text] [Related]

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  • 12. Activation of the CDC42 effector N-WASP by the Shigella flexneri IcsA protein promotes actin nucleation by Arp2/3 complex and bacterial actin-based motility.
    Egile C, Loisel TP, Laurent V, Li R, Pantaloni D, Sansonetti PJ, Carlier MF.
    J Cell Biol; 1999 Sep 20; 146(6):1319-32. PubMed ID: 10491394
    [Abstract] [Full Text] [Related]

  • 13. Cardiolipin Synthesis and Outer Membrane Localization Are Required for Shigella flexneri Virulence.
    Rossi RM, Yum L, Agaisse H, Payne SM.
    mBio; 2017 Aug 29; 8(4):. PubMed ID: 28851846
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  • 15. Avirulence of rough mutants of Shigella flexneri: requirement of O antigen for correct unipolar localization of IcsA in the bacterial outer membrane.
    Sandlin RC, Lampel KA, Keasler SP, Goldberg MB, Stolzer AL, Maurelli AT.
    Infect Immun; 1995 Jan 29; 63(1):229-37. PubMed ID: 7528731
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  • 18. The Shigella Virulence Factor IcsA Relieves N-WASP Autoinhibition by Displacing the Verprolin Homology/Cofilin/Acidic (VCA) Domain.
    Mauricio RP, Jeffries CM, Svergun DI, Deane JE.
    J Biol Chem; 2017 Jan 06; 292(1):134-145. PubMed ID: 27881679
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